Tracer-diffusions in Liquids. IV. Selfdiffusion of Calcium Ion and Chloride Ion in Aqueous Calcium Chloride Solutions - Corrections

1953 ◽  
Vol 75 (24) ◽  
pp. 6361-6361
Author(s):  
Jui Wang
2003 ◽  
Vol 27 (2) ◽  
pp. 75-85 ◽  
Author(s):  
A. QUINTERO-RAMOS ◽  
M. C. BOURNE ◽  
J. BARNARD ◽  
A. ANZALDÚA-MORALES ◽  
R. GONZÁLEZ-LAREDO ◽  
...  

2021 ◽  
Vol 11 (1) ◽  
pp. 204589402098794
Author(s):  
Guogu Liu ◽  
Daiyan Fu ◽  
Heshen Tian ◽  
Aiguo Dai

Pulmonary hypertension(PH)is a kind of hemodynamic and pathophysiological state, in which the pulmonary artery pressure (PAP) rises above a certain threshold. The main pathological manifestation is pulmonary vasoconstriction and remodelling progressively. More and more studies have found that ions play a major role in the pathogenesis of PH. Many vasoactive substances, inflammatory mediators, transcription-inducing factors, apoptosis mediators, redox substances and translation modifiers can control the concentration of ions inside and outside the cell by regulating the activity of ion channels, which can regulate vascular contraction, cell proliferation, migration, apoptosis, inflammation and other functions. We all know that there are no effective drugs to treat PH. Ions are involved in the occurrence and development of PH, so it is necessary to clarify the mechanism of ions in PH as a therapeutic target for PH. The main ions involved in PH are calcium ion (Ca2+), potassium ion (K+), sodium ion (Na+) and chloride ion (Cl–). Here, we mainly discuss the distribution of these ions and their channels in pulmonary arteries and their role in the pathogenesis of PH.


1997 ◽  
Vol 4 (1) ◽  
pp. 13-20
Author(s):  
Luke J Janssen

Electrophysiological and pharmacological techniques were used to characterize the membrane conductance changes underlying spasmogen-evoked depolarization in airway smooth muscle (ASM). Changes included a transient activation of chloride ion channels and prolonged suppression of potassium ion channels; both changes are triggered by release of internally sequestered calcium ion and in turn cause opening of voltage-dependent calcium channels. The resultant influx of calcium ions contributes to contraction as well as to refilling of the internal calcium ion pool. Bronchodilators, on the other hand, act in part through activation of potassium channels, with consequent closure of calcium channels. The tools used to study ion channels in ASM are described, and the investigations of the roles of ion channels in ASM physiology (autacoid-evoked depolarization and hyperpolarization) and pathophysiology (airway hyperresponsiveness) are summarized. Finally, how the relationship between ion channels and ASM function/dysfunction may relate to the treatment of asthma and related breathing disorders is discussed.


Author(s):  
V. V. Shevchuk ◽  
T. N. Potkina ◽  
A. I. Vaitenka ◽  
O. V. Smetanina

The excess of magnesium chloride brines is formed during the polymineral ores processing in order to obtain potassium sulfate. One way to regenerate such brines is to produce artificial carnallite. It is necessary to purify these brines from sulfates for their further use as raw materials for the artificial carnallite production. In this work, the process of desulfurization of magnesium chloride brines with a solution of calcium chloride is studied. The temperature and the processing time, the magnesium chloride solutions concentration and the consumption of desulfurizing agent (calcium chloride) influence on the degree of magnesium chloride solutions purification from sulfate ions was determined. It has been established that almost all sulfate ions interact with calcium ion in 15 minutes and the desulfurization degree reaches 98,08 %. The increase in duration of the suspension mixing is necessary in order to establish equilibrium in the system and relieve the supersaturation in the solution. It has been shown that with increasing solutions saturation with MgCl2, the degree of the magnesium chloride brines purification from SO4 2– ion increases. Complete precipitation of calcium sulfate requires a certain excess concentration of calcium chloride.


1997 ◽  
Vol 47 (1) ◽  
pp. 137-147 ◽  
Author(s):  
Dragan Sinadinović ◽  
Željko Kamberović ◽  
Aleksandar Šutić

1921 ◽  
Vol 34 (4) ◽  
pp. 375-396 ◽  
Author(s):  
Elizabeth Pauline Wolf

1. Wright's method for the study of chemotaxis of leucocytes in vitro, slightly modified, has been found to be most satisfactory in the estimation of the degree of chemotaxis of various substances, because it is possible to make an exact quantitative determination of the leucocytes that have migrated from the blood clot and adhere to the surfaces containing the tested substance. 2. The calcium ion is the only inorganic ion per se which is found to be positively chemotactic under the conditions of these experiments. It is markedly chemotactic in all concentrations and in all combinations, except the citrate. Here the negative chemotaxis of the citrate ion neutralizes the positive chemotaxis of the calcium ion, and neutrality of chemotactic effect results. 3. The sodium and magnesium ions themselves are neutral. Magnesium and sodium salts are dependent upon the negative ion with which the magnesium or sodium is combined for such positive or negative chemotaxis as is exhibited. All the phosphates of sodium, whether tri-, di-, or monobasic salts, are markedly positively chemotactic, and when combined with other reagents which are themselves neutral or negatively chemotactic, produce marked positive chemotaxis. The blood of a person who has taken phosphates either by mouth or intravenously shows a great increase in chemotaxis with sodium phosphate, with calcium chloride, and even with sodium chloride which is ordinarily neutral. 4. All potassium salts are negatively chemotactic. 5. Many substances act synergistically as regards chemotaxis; e.g., when strontium and magnesium salts are mixed there is a marked increase in chemotaxis. Sodium phosphate acts synergistically with calcium chloride. 6. Mercury salts fix the leucocytes in this method so that their influence on chemotaxis cannot be determined. 7. Morphine and morphine salts are positively chemotactic; this is contrary to the results obtained by others with different methods. 8. Substances which produce a very acute inflammation, such as cantharidin, histamine, or turpentine, are found to be positively chemotactic by this method, but substances, such as mustard gas, which produce a marked necrotizing effect are found to be negatively chemotactic, or neutral, though physiologically they would appear to be positively chemotactic. 9. All amino-acids and amines are positively chemotactic to a certain extent. It seems that the longer the carbon chain, the greater the degree of chemotaxis, though this is not absolute. Tyramine is one exception to this, for it causes a peculiar clumping of the cells, so that it is impossible to count the number adhering, and thus determine whether or not tyramine is positively chemotactic. 10. The time that the blood of animals is examined after eating makes a marked difference in the number of cells adhering, for shortly after eating, within 30 minutes, very many more cells will adhere to the agar than at a later time. 11. The blood of different species of animals reacts differently towards different reagents. The chemical composition of these agents seems to have nothing to do with this difference in reaction as far as we could determine. 12. With frozen serial sections it has been found that the depth of penetration of the leucocytes into the agar is proportional to the positive chemotaxis produced by the substance combined with the agar, as demonstrated by the number of leucocytes adherent to the walls of the test chambers.


2007 ◽  
Vol 80 (1) ◽  
pp. 152-154
Author(s):  
V. G. Maiorov ◽  
A. I. Nikolaev ◽  
B. Ya. Zilberman

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