Concentrated Salt Solutions. III. Electrical Conductance of Solutions of Sodium Thiocyanate, Sodium Iodide and Sodium Perchlorate.

1956 ◽  
Vol 60 (2) ◽  
pp. 189-192 ◽  
Author(s):  
M. L. Miller
Keyword(s):  
Sodium Iodide ◽  
Sodium Perchlorate ◽  
Electrical Conductance ◽  
Sodium Thiocyanate ◽  
Salt Solutions

Effect of sodium thiocyanate and sodium perchlorate on poly(N-isopropylacrylamide) collapse

2020 ◽  
Vol 22 (1) ◽  
pp. 189-195
Author(s):  
Andrea Pica ◽  
Giuseppe Graziano
Keyword(s):  
Sodium Perchlorate ◽  
Sodium Thiocyanate ◽  
Conformational States

The two conformational states of PNIPAM have a similar accessibility to perchlorate ions.

Conductivity Study of Some Thiocyanate Salts in Aqueous Binary Mixtures of Tetrahydrofuran and 1,3-Dioxolane at 298.15 K

2010 ◽  
Vol 224 (06) ◽  
pp. 827-841 ◽  
Author(s):  
Riju Chanda ◽  
Mahendra Nath Roy
Keyword(s):  
Binary Mixtures ◽  
Structural Changes ◽  
Ion Pairs ◽  
Mixed Solvents ◽  
Tetrabutylammonium Bromide ◽  
Electrical Conductance ◽  
Ammonium Thiocyanate ◽  
Potassium Thiocyanate ◽  
Sodium Thiocyanate ◽  
Solvent Mixtures

AbstractElectrical conductance measurements are reported for different thiocyanate salts, viz. ammonium thiocyanate (NH4CNS), sodium thiocyanate (NaCNS) and potassium thiocyanate (KCNS), sodium bromide (NaBr), sodium tetraphenylborate (NaBPh4) and tetrabutylammonium bromide (Bu4NBr) in different mole fraction of water-tetrahydrofuran (THF) and water-1,3-dioxolane (1,3-DXL) binary mixtures at 298.15 K. The conductance data have been analyzed by the Fuoss conductance equation in terms of the limiting molar conductance (Λ0), the association constant (KA) and the association diameter (R). The limiting ionic conductances have been estimated from the appropriate division of the limiting molar conductivity value of the “reference electrolyte” Bu4NBPh4. Slight ionic association was found for all these electrolytes in these solvent mixtures. The results have been interpreted in terms of the formation of ion-pairs of ion–solvent interactions and structural changes in the mixed solvents.

Sodium-Iodide Symporter Mediates Iodide Secretion in Rat Gastric Mucosa In Vitro

2006 ◽  
Vol 231 (3) ◽  
pp. 277-281 ◽  
Author(s):  
Malin Josefsson ◽  
Lena Evilevitch ◽  
Björn Weström ◽  
Torsten Grunditz ◽  
Eva Ekblad
Keyword(s):  
Gastric Mucosa ◽  
Sodium Iodide ◽  
Sodium Perchlorate ◽  
Nitric Oxide Donor ◽  
Sodium Iodide Symporter ◽  
Iodide Transport ◽  
Gastric Lumen ◽  
Rat Gastric Mucosa ◽  
Mucosal Side

In vivo studies on rats have demonstrated that considerable amounts of iodide are transported from the bloodstream into the gastric lumen. The mechanisms for and functional significance of this transport are poorly understood. Active (driven by Na+/K+-ATPase) iodide transport into thyroid follicular cells is mediated by the sodium-iodide symporter (NIS), which is also abundantly expressed in gastric mucosa. We aimed to further investigate the iodide transport in gastric mucosa and the Possible role of NIS in this transport process. Iodide transport in rat gastric mucosa was studied in vitro in an Ussing chamber system using 125I as a marker. The system allows measurements in both directions over a mucosal specimen. A considerable transport of iodide (from the serosal to the mucosal side) was established across the gastric mucosa, whereas in the opposite direction (mucosa to serosa), iodide transport was negligible. Sodium Perchlorate (NaClO4), a competitive inhibitor of NIS, and ouabain, an inhibitor of the Na+/K+-ATPase, both attenuated gastric iodide transport from the serosal to the mucosal side. To investigate a possible neuroendocrine regulation of the iodide transport identified to occur from the serosal to the mucosal side of the stomach, thyroid-stimulating hormone (TSH), thyrotropin-releasing hormone (TRH), vasoactive intestinal peptide (VIP), histamine, or nitric oxide donor S-nitroso-N-acetyl-D,L-penicillamine (SNAP) was added. None of these substances influenced the iodide transport. We conclude that iodide is actively transported into the gastric lumen and that this transport is at least partly mediated by NIS. Additional investigations are needed to understand the regulation and significance of this transport.

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