Free hydrogen in hydrogen sulfide generated from ferrous sulfide.

1949 ◽  
Vol 26 (4) ◽  
pp. 199 ◽  
Author(s):  
Jermain D. Porter
2012 ◽  
Vol 53 ◽  
pp. S174
Author(s):  
Elvis Peter ◽  
Xinggui Shen ◽  
Shivang Shah ◽  
Sibile Pardue ◽  
John D. Glawe ◽  
...  

2014 ◽  
Vol 986-987 ◽  
pp. 76-79
Author(s):  
Li Qun Xiu ◽  
Shuai Liu ◽  
Wei Dong Yuan ◽  
Chao Mu ◽  
Di Xu

Now steam stimulation gradually become one of the major thermal recovery methods, and hydrogen sulfide appears in the well casing. With the increase of the thermal recovery time and the application of SAGD technology, the number of wells containing hydrogen sulfide increased, and concentration of hydrogen sulfide are also rising,Producing corrosion products - ferrous sulfide. Ferrous sulfide has pyrophoric. When ferrous sulfide in contact with oxygen in the air can cause spontaneous combustion at room temperature. In order to avoid the occurrence of spontaneous combustion of ferrous sulfide we had an in-depth study of the spontaneous combustion of ferrous sulfide. Through laboratory tests we found the following points. At room temperature,sulfurization reaction of H2S gas and iron oxides occurs more easily in a dry environment. Temperature, H2S humidity, various iron oxides,have important effects on the generation of iron sulfide.


Nitric Oxide ◽  
2015 ◽  
Vol 47 ◽  
pp. S56
Author(s):  
Elise R. Hedegaard ◽  
Anja Gouliaev ◽  
Mathilde Aalling ◽  
Nirthika Sivasubramaniam ◽  
Nini Skovgaard ◽  
...  

1993 ◽  
Author(s):  
R. G. Bending ◽  
M. R. Albert ◽  
M. J. D'Aniello ◽  
S. E. Golunski ◽  
A. F. Diwell ◽  
...  

2013 ◽  
Vol 85 (7) ◽  
pp. 3638-3643 ◽  
Author(s):  
Artur P. Jarosz ◽  
Terence Yep ◽  
Bulent Mutus

2021 ◽  
pp. catalyst.2021.21003
Author(s):  
Rachel B. Allison ◽  
Austin Montgomery ◽  
Gavin L. Sacks

2008 ◽  
Vol 295 (5) ◽  
pp. R1479-R1485 ◽  
Author(s):  
Julie Furne ◽  
Aalia Saeed ◽  
Michael D. Levitt

Hydrogen sulfide is gaining acceptance as an endogenously produced modulator of tissue function. The present paradigm of H2S (diprotonated, gaseous form of hydrogen sulfide) as a tissue messenger consists of H2S being released from the desulfhydration of l-cysteine at a rate sufficient to maintain whole tissue hydrogen sulfide concentrations of 30 μM to >100 μM, and these tissue concentrations serve a messenger function. Utilizing physiological concentrations of l-cysteine and aerobic conditions, we found that catabolism of hydrogen sulfide by mouse liver and brain homogenates exceeded the rate of enzymatic release of this compound such that measureable hydrogen sulfide release was less with tissue-containing vs. tissue-free buffers. Analyses of the gas space over rapidly homogenized mouse brain and liver indicated that in situ tissue hydrogen sulfide concentrations were only about 15 nM. Human alveolar air measurements indicated negligible free H2S concentrations in blood. We conclude rapid tissue catabolism of hydrogen sulfide maintains whole tissue brain and liver concentrations of free hydrogen sulfide that are three orders of magnitude less than conventionally accepted values and only 1/5,000 of the hydrogen sulfide concentration (100 μM) required to alter cellular function in vitro. For hydrogen sulfide to serve as an endogenously produced messenger, tissue production and catabolism must result in intracellular microenvironments with a sufficiently high hydrogen sulfide concentration to activate a local signaling mechanism, while whole tissue concentrations remain very low.


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