Involvement of Oxygen-Derived Free Radicals in Chemical and Biochemical Degradation of Lignin

2001 ◽  
pp. 255-271 ◽  
Author(s):  
Torbjörn Reitberger ◽  
Josef Gierer ◽  
Erquan Yang ◽  
Byung-Ho Yoon
Keyword(s):  
Free Radicals ◽  
Oxygen Derived Free Radicals ◽  
Biochemical Degradation

In vivo measurement of oxygen-derived free radicals during reperfusion injury

Microsurgery ◽  
2002 ◽  
Vol 22 (3) ◽  
pp. 108-113 ◽  
Author(s):  
Rolf Büttemeyer ◽  
Andreas W. Philipp ◽  
Julian W. Mall ◽  
Bixia Ge ◽  
Frieder W. Scheller ◽  
...  
Keyword(s):  
Free Radicals ◽  
Reperfusion Injury ◽  
In Vivo Measurement ◽  
Oxygen Derived Free Radicals

Defective Ag-In-S/ZnS Quantum Dots: An Oxygen-derived Free Radicals Scavenger for Mitigating the Macrophage Inflammation

2021 ◽  
Author(s):  
Na Gao ◽  
Jing Jing ◽  
Hengzhi Zhao ◽  
Yazhou Liu ◽  
Chunlei Yang ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Quantum Dots ◽  
Heart Disease ◽  
Free Radicals ◽  
Inflammatory Diseases ◽  
Antioxidant Therapy ◽  
Oxygen Derived Free Radicals ◽  
Promising Strategy

Oxidative stress plays an important role in the development of inflammatory diseases including allergy, heart disease, diabetes and cancer. Nanomaterials-mediated antioxidant therapy is regarded as a promising strategy to treat...

Oxygen-Derived Free Radicals Mediate Isoflurane-Induced Vasoconstriction of Rabbit Coronary Resistance Arteries

1995 ◽  
Vol 80 (6) ◽  
pp. 1163-1167
Author(s):  
Kyung W. Park ◽  
Hai B. Dai ◽  
Edward Lowenstein ◽  
Amir Darvish ◽  
Frank W. Sellke
Keyword(s):  
Free Radicals ◽  
Coronary Resistance ◽  
Resistance Arteries ◽  
Oxygen Derived Free Radicals

Oxidative Stress in Critically Ill Patients

2002 ◽  
Vol 11 (6) ◽  
pp. 543-551 ◽  
Author(s):  
Caryl Goodyear-Bruch ◽  
Janet D. Pierce
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Superoxide Dismutase ◽  
Free Radicals ◽  
Critically Ill ◽  
Critically Ill Patients ◽  
Oxygen Free Radicals ◽  
Oxygen Species ◽  
Defense System ◽  
Oxygen Derived Free Radicals

Oxygen-derived free radicals play an important role in the development of disease in critically ill patients. Normally, oxygen free radicals are neutralized by antioxidants such as vitamin E or enzymes such as superoxide dismutase. However, in patients who require intensive care, oxygen free radicals become a problem when either a decrease in the removal or an overproduction of the radicals occurs. This oxidative stress and the damage due to it have been implicated in many diseases in critically ill patients. Many drugs and treatments now being investigated are directed toward preventing the damage from oxidative stress. The formation of reactive oxygen species, the damage caused by them, and the body’s defense system against them are reviewed. New interventions are described that may be used in critically ill patients to prevent or treat oxidative damage.

Role of oxygen-derived free radicals in free growth retardation induced by ischemia-reperfusion in rats

1997 ◽  
Vol 272 (2) ◽  
pp. H701-H705 ◽  
Author(s):  
H. Ishimoto ◽  
M. Natori ◽  
M. Tanaka ◽  
T. Miyazaki ◽  
T. Kobayashi ◽  
...  
Keyword(s):  
Blood Flow ◽  
Free Radicals ◽  
Growth Retardation ◽  
Ischemia Reperfusion ◽  
Lipid Peroxides ◽  
Treated Group ◽  
Sprague Dawley ◽  
Doppler Flowmetry ◽  
Oxygen Derived Free Radicals ◽  
The Right

We investigated the involvement of oxygen-derived free radicals in the pathogenesis of the intrauterine growth retardation (IUGR) induced in Sprague-Dawley rats by ischemia-reperfusion. On day 17 of gestation, rats received saline, superoxide dismutase (SOD, 50,000 U/kg), catalase (CAT, 50,000 U/kg), or SOD + CAT subcutaneously 1 h before induction of 30 min of ischemia of the right uterine horn. On day 21 the placental level of lipid peroxides was significantly increased (P < 0.001 vs. sham-operated group) and IUGR was induced (P < 0.001 vs. left horn) in the saline-treated group n = 6). Pretreatment with SOD + CAT (n = 6) significantly inhibited the increase in placental lipid peroxides and prevented IUGR. The effect of ischemia-reperfusion on uterine blood flow, with or without pretreatment with radical scavengers, was investigated in separate experiments by laser-Doppler flowmetry. The induction of hypoperfusion 3 h after ischemia (blood flow -40 +/- 5%, n = 6, P < 0.05) was blocked by pretreatment with SOD + CAT (n = 6). Results indicate that oxygen-derived free radicals may be important in the development of postischemic uteroplacental hypoperfusion and of ischemia-reperfusion-induced IUGR in the rat.

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