Heat shock protein 60 from Klebsiella pneumoniae protects mononuclear cells from apoptotic cell death induced by dexamethasone

2011 ◽  
Vol 51 (5) ◽  
pp. 352-359 ◽  
Author(s):  
Yolanda Ortega-Ortega ◽  
Ethel Awilda García-Latorre ◽  
Ruth Angélica Lezama ◽  
Julieta Luna-Herrera ◽  
Armando Vega-López ◽  
...  
Keyword(s):  
Cell Death ◽  
Heat Shock ◽  
Klebsiella Pneumoniae ◽  
Heat Shock Protein ◽  
Mononuclear Cells ◽  
Apoptotic Cell ◽  
Apoptotic Cell Death ◽  
Heat Shock Protein 60

The Role of Inducible 70-kDa Heat Shock Protein in Cell Cycle Control, Differentiation, and Apoptotic Cell Death of the Human Myeloid Leukemic HL-60 Cells

1998 ◽  
Vol 187 (1) ◽  
pp. 1-12 ◽  
Author(s):  
Hyun-Jeong Kwak ◽  
Chang-Duk Jun ◽  
Hyun-Ock Pae ◽  
Ji-Chang Yoo ◽  
Young-Chul Park ◽  
...  
Keyword(s):  
Cell Cycle ◽  
Cell Death ◽  
Heat Shock ◽  
Heat Shock Protein ◽  
Apoptotic Cell ◽  
Cell Cycle Control ◽  
Apoptotic Cell Death

Preventing apoptotic cell death by a novel small heat shock protein

2007 ◽  
Vol 86 (3) ◽  
pp. 161-171 ◽  
Author(s):  
Szabolcs Bellyei ◽  
Andras Szigeti ◽  
Eva Pozsgai ◽  
Arpad Boronkai ◽  
Eva Gomori ◽  
...  
Keyword(s):  
Cell Death ◽  
Heat Shock ◽  
Heat Shock Protein ◽  
Apoptotic Cell ◽  
Small Heat Shock Protein ◽  
Apoptotic Cell Death

Heat Shock Protein 27 Protects Lung Epithelial Cells From Hyperoxia-Induced Apoptotic Cell Death

2009 ◽  
Vol 65 (3) ◽  
pp. 328-333 ◽  
Author(s):  
Lei Shao ◽  
Ricardo E Perez ◽  
William T Gerthoffer ◽  
William E Truog ◽  
Dong Xu
Keyword(s):  
Cell Death ◽  
Heat Shock ◽  
Epithelial Cells ◽  
Heat Shock Protein ◽  
Apoptotic Cell ◽  
Heat Shock Protein 27 ◽  
Apoptotic Cell Death ◽  
Lung Epithelial Cells ◽  
Lung Epithelial

scholarly journals Heat shock protein 60: an endogenous inducer of dopaminergic cell death in Parkinson disease

2014 ◽  
Vol 11 (1) ◽  
pp. 86 ◽  
Author(s):  
Carmen Noelker ◽  
Lydie Morel ◽  
Anke Osterloh ◽  
Daniel Alvarez-Fischer ◽  
Thomas Lescot ◽  
...  
Keyword(s):  
Cell Death ◽  
Heat Shock ◽  
Heat Shock Protein ◽  
Parkinson Disease ◽  
Heat Shock Protein 60 ◽  
Dopaminergic Cell

scholarly journals Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-κB pathway

2000 ◽  
Vol 12 (11) ◽  
pp. 1539-1546 ◽  
Author(s):  
Sreyashi Basu ◽  
Robert J. Binder ◽  
Ryuichiro Suto ◽  
Kirstin M. Anderson ◽  
Pramod K. Srivastava
Keyword(s):  
Cell Death ◽  
Dendritic Cells ◽  
Heat Shock ◽  
Heat Shock Proteins ◽  
Apoptotic Cell ◽  
Apoptotic Cell Death ◽  
Shock Proteins

Heat Shock Proteins: Endogenous Modulators of Apoptotic Cell Death

2001 ◽  
Vol 286 (3) ◽  
pp. 433-442 ◽  
Author(s):  
Carmen Garrido ◽  
Sandeep Gurbuxani ◽  
Luigi Ravagnan ◽  
Guido Kroemer
Keyword(s):  
Cell Death ◽  
Heat Shock ◽  
Heat Shock Proteins ◽  
Apoptotic Cell ◽  
Apoptotic Cell Death ◽  
Shock Proteins ◽  
Endogenous Modulators

scholarly journals Tissue Microenvironments within Functional Cortical Subdivisions Adjacent to Focal Stroke

2003 ◽  
Vol 23 (9) ◽  
pp. 997-1009 ◽  
Author(s):  
Diana Katsman ◽  
Jian Zheng ◽  
Kateri Spinelli ◽  
S. Thomas Carmichael
Keyword(s):  
Dna Damage ◽  
Cell Death ◽  
Heat Shock ◽  
Heat Shock Protein ◽  
Somatosensory Cortex ◽  
Oxidative Dna Damage ◽  
Apoptotic Cell ◽  
Reactive Gliosis ◽  
Ischemic Damage ◽  
Infarct Core

Stroke produces a region of complete cell death and areas of partial damage, injury, and gliosis. The spatial relationship of these regions of damage to the infarct core and within spared neuronal circuits has not been identified. A model of cortical stroke was developed within functional subsets of the somatosensory cortex. Infarct size, regions of apoptosis, oxidative DNA damage, heat shock protein induction, and subtypes of reactive gliosis were precisely mapped with the somatosensory body map, quantified, and interrelated. Three tissue microenvironments were recognized: zones of partial ischemic damage, heat shock protein induction, and distributed gliosis. These three zones involved progressively more distant cortical regions, each larger than the infarct core. The zone of partial ischemic damage represents an overlap region of apoptotic cell death, oxidative DNA damage, loss of synaptic connections, and local reactive gliosis. The zone of distributed gliosis occupies distinct functional areas of the somatosensory cortex. The tissue reorganization induced by stroke is much larger than the stroke site itself. Adjacent tissue microenvironments are sites of distinct reactive cellular signaling and may serve as a link between the processes of acute cell death and delayed neuronal plasticity after focal stroke.

scholarly journals Clusterin Gene Expression Mediates Resistance to Apoptotic Cell Death Induced by Heat Shock and Oxidative Stress

1999 ◽  
Vol 112 (3) ◽  
pp. 290-296 ◽  
Author(s):  
Isabelle Viard ◽  
Philippe Wehrli ◽  
Lan Jornot ◽  
Roberto Bullani ◽  
Jean-Luc Vechietti ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Gene Expression ◽  
Cell Death ◽  
Heat Shock ◽  
Apoptotic Cell ◽  
Apoptotic Cell Death ◽  
And Oxidative Stress
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