Candida albicans biofilm development, modeling a host–pathogen interaction

2006 ◽  
Vol 9 (4) ◽  
pp. 340-345 ◽  
Author(s):  
Jeniel Nett ◽  
David Andes
Keyword(s):  
Candida Albicans ◽  
Host Pathogen Interaction ◽  
Host Pathogen ◽  
Biofilm Development

scholarly journals Candida albicans cell shaving uncovers new proteins involved in cell wall integrity, yeast to hypha transition, stress response and host–pathogen interaction

2015 ◽  
Vol 127 ◽  
pp. 340-351 ◽  
Author(s):  
Ana Gil-Bona ◽  
Claudia Marcela Parra-Giraldo ◽  
María Luisa Hernáez ◽  
Jose Antonio Reales-Calderon ◽  
Norma V. Solis ◽  
...  
Keyword(s):  
Cell Wall ◽  
Candida Albicans ◽  
Stress Response ◽  
Cell Wall Integrity ◽  
Transition Stress ◽  
Host Pathogen Interaction ◽  
Host Pathogen

Phosphatidylinositol-dependent phospholipases C Plc2 and Plc3 of Candida albicans are dispensable for morphogenesis and host–pathogen interaction

2005 ◽  
Vol 156 (7) ◽  
pp. 822-829 ◽  
Author(s):  
Philipp Knechtle ◽  
Sophie Goyard ◽  
Sophie Brachat ◽  
Oumaïma Ibrahim-Granet ◽  
Christophe d'Enfert
Keyword(s):  
Candida Albicans ◽  
Host Pathogen Interaction ◽  
Host Pathogen

scholarly journals The Role of Macrophages in the Host’s Defense against Sporothrix schenckii

Pathogens ◽  
2021 ◽  
Vol 10 (7) ◽  
pp. 905
Author(s):  
Estela Ruiz-Baca ◽  
Armando Pérez-Torres ◽  
Yolanda Romo-Lozano ◽  
Daniel Cervantes-García ◽  
Carlos A. Alba-Fierro ◽  
...  
Keyword(s):  
Immune Responses ◽  
Macrophage Polarization ◽  
Sporothrix Schenckii ◽  
Macrophage Cell ◽  
Cell Recruitment ◽  
Host Pathogen Interaction ◽  
Interaction Processes ◽  
Host Pathogen ◽  
Molecular Patterns

The role of immune cells associated with sporotrichosis caused by Sporothrix schenckii is not yet fully clarified. Macrophages through pattern recognition receptors (PRRs) can recognize pathogen-associated molecular patterns (PAMPs) of Sporothrix, engulf it, activate respiratory burst, and secrete pro-inflammatory or anti-inflammatory biological mediators to control infection. It is important to consider that the characteristics associated with S. schenckii and/or the host may influence macrophage polarization (M1/M2), cell recruitment, and the type of immune response (1, 2, and 17). Currently, with the use of new monocyte-macrophage cell lines, it is possible to evaluate different host–pathogen interaction processes, which allows for the proposal of new mechanisms in human sporotrichosis. Therefore, in order to contribute to the understanding of these host–pathogen interactions, the aim of this review is to summarize and discuss the immune responses induced by macrophage-S. schenckii interactions, as well as the PRRs and PAMPs involved during the recognition of S. schenckii that favor the immune evasion by the fungus.

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