Effects of cytochalasin D on the distribution of actin and ACTH-induced steroidogenesis in cultured embryonic adrenal gland cells from the Pekin duck (Anas platyrhynchos)

1994 ◽  
Vol 276 (3) ◽  
pp. 587-592
Author(s):  
B.K. Reese ◽  
W.N. Holmes ◽  
J. Cronshaw
Keyword(s):  
Adrenal Gland ◽  
Anas Platyrhynchos ◽  
Cytochalasin D ◽  
Pekin Duck ◽  
Gland Cells

Cytoskeletal changes accompanying ACTH-induced steroidogenesis in cultured embryonic adrenal gland cells from the Pekin duck

1992 ◽  
Vol 268 (1) ◽  
pp. 157-165 ◽  
Author(s):  
James Cronshaw ◽  
B. K. Reese ◽  
M. A. Collie ◽  
W. N. Holmes
Keyword(s):  
Adrenal Gland ◽  
Pekin Duck ◽  
Gland Cells

scholarly journals Structural basis of antigen recognition: crystal structure of duck egg lysozyme

2017 ◽  
Vol 73 (11) ◽  
pp. 910-920 ◽  
Author(s):  
David Brent Langley ◽  
Ben Crossett ◽  
Peter Schofield ◽  
Jenny Jackson ◽  
Mahdi Zeraati ◽  
...  
Keyword(s):  
Crystal Structure ◽  
Mass Spectrometry ◽  
Binding Affinity ◽  
Anas Platyrhynchos ◽  
Antigen Recognition ◽  
Pekin Duck ◽  
Structural Basis ◽  
Primary Sequence ◽  
Duck Egg ◽  
Differential Affinity

Duck egg lysozyme (DEL) is a widely used model antigen owing to its capacity to bind with differential affinity to anti-chicken egg lysozyme antibodies. However, no structures of DEL have so far been reported, and the situation had been complicated by the presence of multiple isoforms and conflicting reports of primary sequence. Here, the structures of two DEL isoforms from the eggs of the commonly used Pekin duck (Anas platyrhynchos) are reported. Using structural analyses in combination with mass spectrometry, non-ambiguous DEL primary sequences are reported. Furthermore, the structures and sequences determined here enable rationalization of the binding affinity of DEL for well documented landmark anti-lysozyme antibodies.

Unraveling molecular mechanistic differences in liver metabolism between lean and fat lines of Pekin duck (Anas platyrhynchos domestica): A proteomic study

2014 ◽  
Vol 98 ◽  
pp. 271-288 ◽  
Author(s):  
Aijuan Zheng ◽  
Wenhuan Chang ◽  
Shuisheng Hou ◽  
Shu Zhang ◽  
Huiyi Cai ◽  
...  
Keyword(s):  
Anas Platyrhynchos ◽  
Liver Metabolism ◽  
Pekin Duck ◽  
Proteomic Study

scholarly journals MHC/class-II-positive cells inhibit corticosterone of adrenal gland cells in experimental arthritis: a role for IL-1β, IL-18, and the inflammasome

2020 ◽  
Vol 10 (1) ◽  
Author(s):  
Hubert Stangl ◽  
Anita Krammetsvogl ◽  
Martin Lesiak ◽  
Christine Wolff ◽  
Rainer H. Straub
Keyword(s):  
Dendritic Cells ◽  
Adrenal Gland ◽  
Mhc Class Ii ◽  
Adrenal Glands ◽  
Collagen Type ◽  
Experimental Arthritis ◽  
Activated Macrophages ◽  
Adrenal Cells ◽  
Gland Cells ◽  
Key Factor

Abstract In experimental arthritis, glucocorticoid secretion is inadequate relative to inflammation. We hypothesized that IL-1 is a key factor for inadequate glucocorticoid secretion in arthritic rats. Collagen type II—induced arthritis (CIA) in DA rats was the model to study effects of IL-1 on adrenal function. In the CIA model, an increase of intraadrenal MHCII-positive cells was observed. MHCII-positive cells or bone marrow-derived dendritic cells inhibited glucocorticoid secretion of adrenal gland cells. IL-1, but also IL-18 and the inflammasome were critical in glucocorticoid inhibition. Arthritic compared to control adrenal gland cells produced higher amounts of CXC chemokines from MHCII+ adrenal cells, particularly CINC-2, which is strongly dependent on presence of IL-1. In CIA, macrophages and/or dendritic cells inhibit glucocorticoid secretion via IL-1 in adrenal glands. These findings show that activated macrophages and/or dendritic cells inhibit glucocorticoid secretion in experimental arthritis and that IL-1β is a decisive factor.

Impaired arginine–vasopressin-induced aldosterone release from adrenal gland cells in mice lacking the vasopressin V1A receptor

2007 ◽  
Vol 566 (1-3) ◽  
pp. 226-230 ◽  
Author(s):  
Jun-ichi Birumachi ◽  
Masami Hiroyama ◽  
Yoko Fujiwara ◽  
Toshinori Aoyagi ◽  
Atsushi Sanbe ◽  
...  
Keyword(s):  
Adrenal Gland ◽  
Arginine Vasopressin ◽  
V1a Receptor ◽  
Gland Cells ◽  
Vasopressin V1a Receptor
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