A quantitative comparison of plaque types in Alzheimer's disease and senile dementia of the Lewy body type

1996 ◽  
Vol 91 (5) ◽  
pp. 526-529 ◽  
Author(s):  
Jeanette E. McKenzie ◽  
Richard J. Edwards ◽  
Stephen M. Gentleman ◽  
Paul G. Ince ◽  
Robert H. Perry ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Lewy Body ◽  
Senile Dementia ◽  
Quantitative Comparison ◽  
Body Type ◽  
Senile Dementia Of The

Dementia with Lewy bodies

1998 ◽  
Vol 4 (6) ◽  
pp. 360-363 ◽  
Author(s):  
E. Jane Byrne
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Lewy Body ◽  
Senile Dementia ◽  
Lewy Bodies ◽  
Lewy Body Dementia ◽  
Body Type ◽  
Clinical Criteria ◽  
Dementia Syndrome ◽  
Distinct Disease

Dementia with cortical Lewy bodies (LBD) was first described by Okazakiet alin 1961 and is now recognised as a relatively common cause of the dementia syndrome. The true prevalence of LBD is unknown. In post-mortem studies of patients diagnosed as having dementia in life, the mean frequency of Lewy body dementia is 12.5% (Byrne, 1997). Clinically diagnosed LBD (using operational clinical criteria) is found in 10–23% of patients presenting to, or in the care of, psychogeriatric services (Collertonet al, 1996). What is not yet certain is its nosological status; opinion is divided between regarding it as a variety of Alzheimer's disease (the Lewy body variant), a distinct disease (senile dementia of the Lewy body type) or a spectrum disorder related to both Parkinson's disease and to Alzheimer's disease (Byrne, 1992).

Understanding Senile Dementia: A Challenge for the Future

1983 ◽  
Vol 16 (1) ◽  
pp. 1-6
Author(s):  
Martha Storandt
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Differential Diagnosis ◽  
Structural Changes ◽  
Senile Dementia ◽  
Accelerated Aging ◽  
Later Life ◽  
The Future ◽  
Supportive Environments ◽  
Senile Dementia Of The

The causes of dementia in later life are reviewed. Special attention is paid to senile dementia of the Alzheimer's type, its prevelance, the characteristic structural changes in the brains of persons suffering from this disorder, and the hypothesized causes of the disease. The hypothesis that Alzheimer's disease represents accelerated aging is described. Treatment issues are discussed, primarily in terms of supportive environments, differential diagnosis, and assistance to the caregiver.

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