Langerhans' cell histiocytosis as a cause of periportal abnormal signal intensity on MRI

1999 ◽  
Vol 24 (4) ◽  
pp. 373-377 ◽  
Author(s):  
M. Kim ◽  
C. Lyu ◽  
Y. Jin ◽  
H. Yoo
Keyword(s):  
Signal Intensity ◽  
Langerhans Cell Histiocytosis ◽  
Langerhans Cell ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity

Donor Site Evaluation After Osteochondral Autograft Transplantation for Capitellar Osteochondritis Dissecans

2019 ◽  
Vol 47 (12) ◽  
pp. 2836-2843 ◽  
Author(s):  
Takeshi Matsuura ◽  
Yusuke Hashimoto ◽  
Takuya Kinoshita ◽  
Kazuya Nishino ◽  
Yohei Nishida ◽  
...  
Keyword(s):  
Magnetic Resonance ◽  
Osteochondritis Dissecans ◽  
Signal Intensity ◽  
Donor Site ◽  
International Knee Documentation Committee ◽  
Mri Findings ◽  
Abnormal Signal ◽  
Osteochondral Autograft ◽  
Abnormal Signal Intensity ◽  
Usual Criterion

Background: Osteochondral autograft transplantation (OAT) for capitellar osteochondritis dissecans (OCD) requires harvesting of tissue from the asymptomatic knee joint. However, donor site morbidity (DSM) in such cases remains unclear. Purpose: To evaluate DSM and postoperative radiographic findings in patients undergoing OAT for advanced capitellar OCD. Study Design: Case series; Level of evidence, 4. Methods: Eighty-six juvenile athletes underwent OAT for advanced capitellar OCD. The 2000 International Knee Documentation Committee subjective score, rate of return to sports, and symptoms in the donor knee were assessed at a mean follow-up of 86 months (range, 28-185 months). Additionally, 49 of the 86 patients were assessed by radiographic and magnetic resonance imaging (MRI) findings of the knee donor site. DSM was defined according to the usual criterion (persistent symptoms for >1 year or the need for subsequent intervention) and a stricter criterion (the presence of any symptoms, such as effusion, patellofemoral complaints, crepitation, unspecified disturbance, stiffness, pain/instability during activities, and osteoarthritic change). The stricter criterion was also used to allocate patients into a DSM group and a no-DSM group. Results: Two and 11 patients were determined to have DSM according to the usual and stricter criteria, respectively. All patients returned to the competitive level of their sports. The mean International Knee Documentation Committee score was 99.0. MRI findings showed abnormal signal intensity in 17 patients (35%) and hypertrophic changes in 11 patients (22%) at the donor harvest sites. The MOCART (magnetic resonance observation of cartilage repair tissue) score was higher in the no-DSM group (mean, 68.2) than in the DSM group (mean, 52.9) ( P = .027). Conclusion: OAT for juvenile athletes with advanced capitellar OCD provided good clinical outcomes. The DSM rate was relatively low (2.3%) with the usual criterion but high (12.8%) with the stricter criterion. MRI showed an abnormal signal intensity and hypertrophy in some cases.

Longitudinal Assessment of Enhancing Foci of Abnormal Signal Intensity in Neurofibromatosis Type 1

2021 ◽  
Vol 42 (4) ◽  
pp. 766-773
Author(s):  
N. Hainc ◽  
M.W. Wagner ◽  
S. Laughlin ◽  
J. Rutka ◽  
C. Hawkins ◽  
...  
Keyword(s):  
Neurofibromatosis Type 1 ◽  
Signal Intensity ◽  
Neurofibromatosis Type ◽  
Longitudinal Assessment ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity

scholarly journals Carotid-Cavernous Fistula Associated with an Intracranial Lesion Caused by Cortical Venous Reflux

2006 ◽  
Vol 12 (1_suppl) ◽  
pp. 167-173 ◽  
Author(s):  
S. Takahashi ◽  
I. Sakuma ◽  
T. Otani ◽  
K. Yasuda ◽  
N. Tomura ◽  
...  
Keyword(s):  
Signal Intensity ◽  
Venous Drainage ◽  
Brain Parenchyma ◽  
Venous Congestion ◽  
Venous Reflux ◽  
Carotid Cavernous Fistula ◽  
Abnormal Signal ◽  
Cortical Venous Reflux ◽  
Abnormal Signal Intensity ◽  
The Brain

Digital subtraction angiography (DSA) and magnetic resonance imaging (MRI) findings in 20 patients with carotid-cavernous fistula (CCF; 3 direct CCFs and 17 indirect CCFs) were retrospectively reviewed to evaluate venous drainage patterns that may cause intracerebral haemorrhage or venous congestion of the brain parenchyma. We evaluated the relationship between cortical venous reflux and abnormal signal intensity of the brain parenchyma on MRI. Cortical venous reflux was identified on DSA in 12 of 20 patients (60.0%) into the superficial middle cerebral vein (SMCV; n=4), the uncal vein (n=2), the petrosal vein (n=2), the lateral mesencephalic vein (LMCV; n=1), the anterior pontomesencephalic vein (APMV; n=1), both the APMV and the petrosal vein (n=1) and both the uncal vein and the SMCV (n=1). Features of venous congestion, such as tortuous and engorged veins, focal staining and delayed appearance of the veins, were demonstrated along the region of cortical venous reflux in the venous phase of internal carotid or vertebral arteriography in six of 20 patients (30.0%). These findings were not observed in the eight CCF patients who did not demonstrate cortical venous reflux. MRI revealed abnormal signal intensity of the brain parenchyma along the region with cortical venous reflux in four of 20 indirect CCF patients (20%). Of these four patients, one presented with putaminal haemorrhage, while the other three presented with hyperintensity of the pons, the middle cerebellar peduncle or both on T2-weighted images, reflecting venous congestion. The venous drainage routes were obliterated except for cortical venous reflux in these four patients and the patients without abnormal signal intensity on MRI had other patent venous outlets in addition to cortical venous reflux. CCF is commonly associated with cortical venous reflux. The obliteration or stenosis of venous drainage routes causes a converging venous outflow that develops into cortical venous reflux and results in venous congestion of the brain parenchyma or intracerebral haemorrhage. Hyperintensity of brain parenchyma along the region of cortical venous reflux on T2-weighted images reflects venous congestion and is the crucial finding that indicates concentration of venous drainage into cortical venous reflux.

Neonatal methionine adenosyltransferase I/III deficiency with abnormal signal intensity in the central tegmental tract

2019 ◽  
Vol 41 (4) ◽  
pp. 382-388 ◽  
Author(s):  
Jun Kido ◽  
Takaaki Sawada ◽  
Ken Momosaki ◽  
Yosuke Suzuki ◽  
Hiroyuki Uetani ◽  
...  
Keyword(s):  
Signal Intensity ◽  
Methionine Adenosyltransferase ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity ◽  
Central Tegmental Tract

Abnormal signal intensity of corpus callosum in schizophrenia

1997 ◽  
Vol 24 (1-2) ◽  
pp. 143
Author(s):  
E.L. Dick ◽  
R. Kotwal ◽  
D.R. Rosenberg ◽  
W.W. Bagwell ◽  
T. Kisler ◽  
...  
Keyword(s):  
Corpus Callosum ◽  
Signal Intensity ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity

Differentiating Lumbar Disc Protrusions, Disc Bulges, and Discs With Normal Contour but Abnormal Signal Intensity

Spine ◽  
1999 ◽  
Vol 24 (1) ◽  
pp. 44-53 ◽  
Author(s):  
Pierre C. Milette ◽  
Suzanne Fontaine ◽  
Luigi Lepanto ◽  
Étienne Cardinal ◽  
Guy Breton
Keyword(s):  
Signal Intensity ◽  
Lumbar Disc ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity

Carotid-Cavernous Fistula Associated with an Intracranial Lesion Caused by Cortical Venous Reflux

1995 ◽  
Vol 8 (6) ◽  
pp. 167-173
Author(s):  
S. Takahashi ◽  
I. Sakuma ◽  
T. Otani ◽  
K. Yasuda ◽  
N. Tomura ◽  
...  
Keyword(s):  
Signal Intensity ◽  
Venous Drainage ◽  
Brain Parenchyma ◽  
Venous Congestion ◽  
Venous Reflux ◽  
Carotid Cavernous Fistula ◽  
Abnormal Signal ◽  
Cortical Venous Reflux ◽  
Abnormal Signal Intensity ◽  
The Brain

Digital subtraction angiography (DSA) and magnetic resonance imaging (MRI) findings in 20 patients with carotid-cavernous fistula (CCF; 3 direct CCFs and 17 indirect CCFs) were retrospectively reviewed to evaluate venous drainage patterns that may cause intracerebral haemorrhage or venous congestion of the brain parenchyma. We evaluated the relationship between cortical venous reflux and abnormal signal intensity of the brain parenchyma on MRI. Cortical venous reflux was identified on DSA in 12 of 20 patients (60.0%) into the superficial middle cerebral vein (SMCV; n=4), the uncal vein (n=2), the petrosal vein (n=2), the lateral mesencephalic vein (LMCV; n=1), the anterior pontomesencephalic vein (APMV; n=1), both the APMV and the petrosal vein (n=1) and both the uncal vein and the SMCV (n=1). Features of venous congestion, such as tortuous and engorged veins, focal staining and delayed appearance of the veins, were demonstrated along the region of cortical venous reflux in the venous phase of internal carotid or vertebral arteriography in six of 20 patients (30.0%). These findings were not observed in the eight CCF patients who did not demonstrate cortical venous reflux. MRI revealed abnormal signal intensity of the brain parenchyma along the region with cortical venous reflux in four of 20 indirect CCF patients (20%). Of these four patients, one presented with putaminal haemorrhage, while the other three presented with hyperintensity of the pons, the middle cerebellar peduncle or both on T2-weighted images, reflecting venous congestion. The venous drainage routes were obliterated except for cortical venous reflux in these four patients and the patients without abnormal signal intensity on MRI had other patent venous outlets in addition to cortical venous reflux. CCF is commonly associated with cortical venous reflux. The obliteration or stenosis of venous drainage routes causes a converging venous outflow that develops into cortical venous reflux and results in venous congestion of the brain parenchyma or intracerebral haemorrhage. Hyperintensity of brain parenchyma along the region of cortical venous reflux on T2-weighted images reflects venous congestion and is the crucial finding that indicates concentration of venous drainage into cortical venous reflux.

Diagnostic performance of T2-weighted imaging and intravoxel incoherent motion diffusion-weighted MRI for predicting metastatic axillary lymph nodes in T1 and T2 stage breast cancer

2021 ◽  
pp. 028418512110028
Author(s):  
Yuanyuan Liu ◽  
Hongbing Luo ◽  
Chunhua Wang ◽  
Xiaoyu Chen ◽  
Min Wang ◽  
...  
Keyword(s):  
Breast Cancer ◽  
Logistic Regression ◽  
Regression Analysis ◽  
Sensitivity And Specificity ◽  
Logistic Regression Analysis ◽  
Signal Intensity ◽  
Intravoxel Incoherent Motion ◽  
Axillary Lymph ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity

Background Non-invasive modalities for assessing axillary lymph node (ALN) are needed in clinical practice. Purpose To investigate the suspicious ALN on unenhanced T2-weighted (T2W) imaging and intravoxel incoherent motion diffusion-weighted imaging (IVIM DWI) for predicting ALN metastases (ALNM) in patients with T1–T2 stage breast cancer and clinically negative ALN. Material and Methods Two radiologists identified the most suspicious ALN or the largest ALN in negative axilla by T2W imaging features, including short axis (Size-S), long axis (Size-L)/S ratio, fatty hilum, margin, and signal intensity on T2W imaging. The IVIM parameters of these selected ALNs were also obtained. The Mann–Whitney U test or t-test was used to compare the metastatic and non-metastatic ALN groups. Finally, logistic regression analysis with T2W imaging and IVIM features for predicting ALNM was conducted. Results This study included 49 patients with metastatic ALNs and 50 patients with non-metastatic ALNs. Using the above conventional features on T2W imaging, the sensitivity and specificity in predicting ALNM were not high. Compared with non-metastatic ALNs, metastatic ALNs had lower pseudo-diffusion coefficient (D*) ( P = 0.043). Logistic regression analysis showed that the most useful features for predicting ALNM were signal intensity and D*. The sensitivity and specificity predicting ALNM that satisfied abnormal signal intensity and lower D* were 73.5% and 84%, respectively. Conclusions The abnormal signal intensity on T2W imaging and one IVIM feature (D*) were significantly associated with ALNM, with sensitivity of 73.5% and specificity of 84%.

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