Association of posterior tibial tendon abnormalities with abnormal signal intensity in the sinus tarsi on MR imaging

2000 ◽  
Vol 29 (9) ◽  
pp. 514-519 ◽  
Author(s):  
M. W. Anderson ◽  
P. A. Kaplan ◽  
R. G. Dussault ◽  
S. Hurwitz
Keyword(s):  
Mr Imaging ◽  
Signal Intensity ◽  
Posterior Tibial Tendon ◽  
Abnormal Signal ◽  
Posterior Tibial ◽  
Sinus Tarsi ◽  
Abnormal Signal Intensity

Abnormal Signal Intensity in Skeletal Muscle at MR Imaging: Patterns, Pearls, and Pitfalls

Radiographics ◽  
2000 ◽  
Vol 20 (suppl_1) ◽  
pp. S295-S315 ◽  
Author(s):  
David A. May ◽  
David G. Disler ◽  
Elizabeth A. Jones ◽  
Avinash A. Balkissoon ◽  
B. J. Manaster
Keyword(s):  
Skeletal Muscle ◽  
Mr Imaging ◽  
Signal Intensity ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity

Donor Site Evaluation After Osteochondral Autograft Transplantation for Capitellar Osteochondritis Dissecans

2019 ◽  
Vol 47 (12) ◽  
pp. 2836-2843 ◽  
Author(s):  
Takeshi Matsuura ◽  
Yusuke Hashimoto ◽  
Takuya Kinoshita ◽  
Kazuya Nishino ◽  
Yohei Nishida ◽  
...  
Keyword(s):  
Magnetic Resonance ◽  
Osteochondritis Dissecans ◽  
Signal Intensity ◽  
Donor Site ◽  
International Knee Documentation Committee ◽  
Mri Findings ◽  
Abnormal Signal ◽  
Osteochondral Autograft ◽  
Abnormal Signal Intensity ◽  
Usual Criterion

Background: Osteochondral autograft transplantation (OAT) for capitellar osteochondritis dissecans (OCD) requires harvesting of tissue from the asymptomatic knee joint. However, donor site morbidity (DSM) in such cases remains unclear. Purpose: To evaluate DSM and postoperative radiographic findings in patients undergoing OAT for advanced capitellar OCD. Study Design: Case series; Level of evidence, 4. Methods: Eighty-six juvenile athletes underwent OAT for advanced capitellar OCD. The 2000 International Knee Documentation Committee subjective score, rate of return to sports, and symptoms in the donor knee were assessed at a mean follow-up of 86 months (range, 28-185 months). Additionally, 49 of the 86 patients were assessed by radiographic and magnetic resonance imaging (MRI) findings of the knee donor site. DSM was defined according to the usual criterion (persistent symptoms for >1 year or the need for subsequent intervention) and a stricter criterion (the presence of any symptoms, such as effusion, patellofemoral complaints, crepitation, unspecified disturbance, stiffness, pain/instability during activities, and osteoarthritic change). The stricter criterion was also used to allocate patients into a DSM group and a no-DSM group. Results: Two and 11 patients were determined to have DSM according to the usual and stricter criteria, respectively. All patients returned to the competitive level of their sports. The mean International Knee Documentation Committee score was 99.0. MRI findings showed abnormal signal intensity in 17 patients (35%) and hypertrophic changes in 11 patients (22%) at the donor harvest sites. The MOCART (magnetic resonance observation of cartilage repair tissue) score was higher in the no-DSM group (mean, 68.2) than in the DSM group (mean, 52.9) ( P = .027). Conclusion: OAT for juvenile athletes with advanced capitellar OCD provided good clinical outcomes. The DSM rate was relatively low (2.3%) with the usual criterion but high (12.8%) with the stricter criterion. MRI showed an abnormal signal intensity and hypertrophy in some cases.

Longitudinal Assessment of Enhancing Foci of Abnormal Signal Intensity in Neurofibromatosis Type 1

2021 ◽  
Vol 42 (4) ◽  
pp. 766-773
Author(s):  
N. Hainc ◽  
M.W. Wagner ◽  
S. Laughlin ◽  
J. Rutka ◽  
C. Hawkins ◽  
...  
Keyword(s):  
Neurofibromatosis Type 1 ◽  
Signal Intensity ◽  
Neurofibromatosis Type ◽  
Longitudinal Assessment ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity

scholarly journals Carotid-Cavernous Fistula Associated with an Intracranial Lesion Caused by Cortical Venous Reflux

2006 ◽  
Vol 12 (1_suppl) ◽  
pp. 167-173 ◽  
Author(s):  
S. Takahashi ◽  
I. Sakuma ◽  
T. Otani ◽  
K. Yasuda ◽  
N. Tomura ◽  
...  
Keyword(s):  
Signal Intensity ◽  
Venous Drainage ◽  
Brain Parenchyma ◽  
Venous Congestion ◽  
Venous Reflux ◽  
Carotid Cavernous Fistula ◽  
Abnormal Signal ◽  
Cortical Venous Reflux ◽  
Abnormal Signal Intensity ◽  
The Brain

Digital subtraction angiography (DSA) and magnetic resonance imaging (MRI) findings in 20 patients with carotid-cavernous fistula (CCF; 3 direct CCFs and 17 indirect CCFs) were retrospectively reviewed to evaluate venous drainage patterns that may cause intracerebral haemorrhage or venous congestion of the brain parenchyma. We evaluated the relationship between cortical venous reflux and abnormal signal intensity of the brain parenchyma on MRI. Cortical venous reflux was identified on DSA in 12 of 20 patients (60.0%) into the superficial middle cerebral vein (SMCV; n=4), the uncal vein (n=2), the petrosal vein (n=2), the lateral mesencephalic vein (LMCV; n=1), the anterior pontomesencephalic vein (APMV; n=1), both the APMV and the petrosal vein (n=1) and both the uncal vein and the SMCV (n=1). Features of venous congestion, such as tortuous and engorged veins, focal staining and delayed appearance of the veins, were demonstrated along the region of cortical venous reflux in the venous phase of internal carotid or vertebral arteriography in six of 20 patients (30.0%). These findings were not observed in the eight CCF patients who did not demonstrate cortical venous reflux. MRI revealed abnormal signal intensity of the brain parenchyma along the region with cortical venous reflux in four of 20 indirect CCF patients (20%). Of these four patients, one presented with putaminal haemorrhage, while the other three presented with hyperintensity of the pons, the middle cerebellar peduncle or both on T2-weighted images, reflecting venous congestion. The venous drainage routes were obliterated except for cortical venous reflux in these four patients and the patients without abnormal signal intensity on MRI had other patent venous outlets in addition to cortical venous reflux. CCF is commonly associated with cortical venous reflux. The obliteration or stenosis of venous drainage routes causes a converging venous outflow that develops into cortical venous reflux and results in venous congestion of the brain parenchyma or intracerebral haemorrhage. Hyperintensity of brain parenchyma along the region of cortical venous reflux on T2-weighted images reflects venous congestion and is the crucial finding that indicates concentration of venous drainage into cortical venous reflux.

Neonatal methionine adenosyltransferase I/III deficiency with abnormal signal intensity in the central tegmental tract

2019 ◽  
Vol 41 (4) ◽  
pp. 382-388 ◽  
Author(s):  
Jun Kido ◽  
Takaaki Sawada ◽  
Ken Momosaki ◽  
Yosuke Suzuki ◽  
Hiroyuki Uetani ◽  
...  
Keyword(s):  
Signal Intensity ◽  
Methionine Adenosyltransferase ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity ◽  
Central Tegmental Tract

MR imaging of posterior tibial tendon dysfunction.

1996 ◽  
Vol 167 (3) ◽  
pp. 675-682 ◽  
Author(s):  
N J Khoury ◽  
G Y el-Khoury ◽  
C L Saltzman ◽  
E A Brandser
Keyword(s):  
Mr Imaging ◽  
Posterior Tibial Tendon ◽  
Posterior Tibial Tendon Dysfunction ◽  
Posterior Tibial

Abnormal signal intensity of corpus callosum in schizophrenia

1997 ◽  
Vol 24 (1-2) ◽  
pp. 143
Author(s):  
E.L. Dick ◽  
R. Kotwal ◽  
D.R. Rosenberg ◽  
W.W. Bagwell ◽  
T. Kisler ◽  
...  
Keyword(s):  
Corpus Callosum ◽  
Signal Intensity ◽  
Abnormal Signal ◽  
Abnormal Signal Intensity
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