Changes of urinary kallikrein excretion and plasma natriuretic factor in a patient with primary aldosteronism with special reference to adrenal histology

1994 ◽  
Vol 26 (4) ◽  
pp. 375-381 ◽  
Author(s):  
S. Hayami ◽  
T. Nakada ◽  
H. Kakizaki ◽  
T. Yagisawa ◽  
H. Kaneko ◽  
...  
1989 ◽  
Vol 7 ◽  
pp. S232-233 ◽  
Author(s):  
Paolo Dessì-Fulgheri ◽  
Giorgio Di Noto ◽  
Roberto Palermo ◽  
Roberto Catalini ◽  
Paolo Russo ◽  
...  

1991 ◽  
Vol 4 (3 Pt 1) ◽  
pp. 214-218
Author(s):  
P. Dessi-Fulgheri ◽  
G. Di Noto ◽  
R. Palermo ◽  
R. Catalini ◽  
P. Russo ◽  
...  

1977 ◽  
Vol 121 (2) ◽  
pp. 111-119 ◽  
Author(s):  
MASAHIDE SEINO ◽  
KEISHI ABE ◽  
YUTAKA SAKURAI ◽  
NOBUO IROKAWA ◽  
MINORU YASUJIMA ◽  
...  

1978 ◽  
Vol 55 (1) ◽  
pp. 51-55 ◽  
Author(s):  
A. Lechi ◽  
G. Covi ◽  
C. Lechi ◽  
A. Corgnati ◽  
E. Arosio ◽  
...  

1. The 24 h urinary excretion of kallikrein has been studied in 40 normotensive control subjects and in 74 age-matched patients with essential hypertension under similar conditions. By use of the renin-sodium index, hypertensive patients were divided into two subgroups: low-renin hypertension and normal-renin hypertension patients. Urinary kallikrein determinations were also obtained from six hypertensive patients with primary aldosteronism. 2. Urinary kallikrein was significantly lower both in patients with normal-renin and low-renin essential hypertension. Urinary kallikrein excretion was very high in the patients with primary aldosteronism. 3. In nine hypertensive patients β-adrenoreceptor-blocking therapy caused a significant decrease of plasma renin activity, but had no significant effect on urinary kallikrein excretion. 4. The results support the concept that low urinary kallikrein is likely to be a marker of essential hypertension. Under certain conditions its excretion is positively related to mineralocorticoid hormone concentrations but it is not primarily related to the renin-angiotensin system.


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