Influence du glucose sur le transport intestinal de la phénylalanine en présence et en absence de dinitrophénol

1972 ◽  
Vol 28 (11) ◽  
pp. 1313-1315 ◽  
Author(s):  
A. -L. Luisier ◽  
J. W. L. Robinson
Keyword(s):  
Transport Intestinal

scholarly journals Avancées dans la dissection fonctionnelle du transport intestinal des lipides

2007 ◽  
Vol 23 (11) ◽  
pp. 1014-1020
Author(s):  
Emile Levy ◽  
Marc-André Lavoie ◽  
Edgard Delvin ◽  
Ernest Seidman ◽  
Marie Lambert ◽  
...  
Keyword(s):  
Transport Intestinal

Transport intestinal des cations et fonctionnement des phosphatases alcalines

1991 ◽  
Vol 5 (4) ◽  
pp. 215-225
Author(s):  
Yvonne Dupuis ◽  
Zofia Porembska ◽  
Bernard Lacour ◽  
Sylviane Tardivel ◽  
Gabriel Crouzoulon ◽  
...  
Keyword(s):  
Transport Intestinal

scholarly journals Inositols Depletion and Resistance: Principal Mechanisms and Therapeutic Strategies

2021 ◽  
Vol 22 (13) ◽  
pp. 6796
Author(s):  
Elisa Lepore ◽  
Rosa Lauretta ◽  
Marta Bianchini ◽  
Marilda Mormando ◽  
Cherubino Di Lorenzo ◽  
...  
Keyword(s):  
Renal Excretion ◽  
Polycystic Ovary ◽  
Signal Pathways ◽  
Therapeutic Approaches ◽  
Ovary Syndrome ◽  
Physiological Processes ◽  
Metabolic Functions ◽  
Pathological Conditions ◽  
Endogenous Synthesis ◽  
Transport Intestinal

Inositols are natural molecules involved in several biochemical and metabolic functions in different organs and tissues. The term “inositols” refers to five natural stereoisomers, among which myo-Inositol (myo-Ins) is the most abundant one. Several mechanisms contribute to regulate cellular and tissue homeostasis of myo-Ins levels, including its endogenous synthesis and catabolism, transmembrane transport, intestinal adsorption and renal excretion. Alterations in these mechanisms can lead to a reduction of inositols levels, exposing patient to several pathological conditions, such as Polycystic Ovary Syndrome (PCOS), hypothyroidism, hormonal and metabolic imbalances, like weight gain, hyperinsulinemia, dyslipidemia, and metabolic syndrome. Indeed, myo-Ins is involved in different physiological processes as a key player in signal pathways, including reproductive, hormonal, and metabolic modulation. Genetic mutations in genes codifying for proteins of myo-Ins synthesis and transport, competitive processes with structurally similar molecules, and the administration of specific drugs that cause a central depletion of myo-Ins as a therapeutic outcome, can lead to a reduction of inositols levels. A deeper knowledge of the main mechanisms involved in cellular inositols depletion may add new insights for developing tailored therapeutic approaches and shaping the dosages and the route of administration, with the aim to develop efficacious and safe approaches counteracting inositols depletion-induced pathological events.

Guanylin and Uroguanylin: Intestinal Peptide Hormones That Regulate Epithelial Transport

Physiology ◽  
1996 ◽  
Vol 11 (1) ◽  
pp. 17-24 ◽  
Author(s):  
LR Forte ◽  
FK Hamra
Keyword(s):  
Water Transport ◽  
Guanylate Cyclase ◽  
Epithelial Transport ◽  
Intestinal Bacteria ◽  
Peptide Hormones ◽  
Membrane Guanylate Cyclase ◽  
Water Homeostasis ◽  
And Control ◽  
Stimulation Of ◽  
Transport Intestinal

Guanylin and uroguanylin are peptides that activate a membrane guanylate cyclase and control salt and water transport. Intestinal bacteria secrete toxins that mimic the actions of these peptides, causing a secretory form of diarrhea. Stimulation of guanylin/uroguanylin receptors in the kidney increases salt excretion. Thus guanylin and uroguanylin may regulate salt and water homeostasis.

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