Distribution of isoforms of the myofibrillar proteins in myoid cells of thymus

1986 ◽  
Vol 7 (4) ◽  
pp. 351-360 ◽  
Author(s):  
G. K. Dhoot ◽  
I. Dransfield ◽  
R. J. A. Grand ◽  
S. V. Perry
Keyword(s):  
Myofibrillar Proteins ◽  
Myoid Cells

Binding of aldehyde flavour compounds to beef myofibrillar proteins and the effect of nonenzymatic glycation with glucose and glucosamine

LWT ◽  
2021 ◽  
pp. 111198
Author(s):  
Peipei Dou ◽  
Xianchao Feng ◽  
Xingguang Cheng ◽  
Qinhao Guan ◽  
Junlan Wang ◽  
...  
Keyword(s):  
Myofibrillar Proteins ◽  
Nonenzymatic Glycation ◽  
Flavour Compounds

scholarly journals TCF21+ mesenchymal cells contribute to testis somatic cell development, homeostasis, and regeneration in mice

2021 ◽  
Vol 12 (1) ◽  
Author(s):  
Yu-chi Shen ◽  
Adrienne Niederriter Shami ◽  
Lindsay Moritz ◽  
Hailey Larose ◽  
Gabriel L. Manske ◽  
...  
Keyword(s):  
Tissue Homeostasis ◽  
Testicular Development ◽  
Myoid Cells ◽  
Regenerative Capacity ◽  
Adult Testis ◽  
Mesenchymal Progenitors ◽  
And Function ◽  
Potential Reserve ◽  
Resident Fibroblast

AbstractTesticular development and function rely on interactions between somatic cells and the germline, but similar to other organs, regenerative capacity declines in aging and disease. Whether the adult testis maintains a reserve progenitor population remains uncertain. Here, we characterize a recently identified mouse testis interstitial population expressing the transcription factor Tcf21. We found that TCF21lin cells are bipotential somatic progenitors present in fetal testis and ovary, maintain adult testis homeostasis during aging, and act as potential reserve somatic progenitors following injury. In vitro, TCF21lin cells are multipotent mesenchymal progenitors which form multiple somatic lineages including Leydig and myoid cells. Additionally, TCF21+ cells resemble resident fibroblast populations reported in other organs having roles in tissue homeostasis, fibrosis, and regeneration. Our findings reveal that the testis, like other organs, maintains multipotent mesenchymal progenitors that can be potentially leveraged in development of future therapies for hypoandrogenism and/or infertility.

scholarly journals Ultrasonic structural modification of myofibrillar proteins from Coregonus peled improves emulsification properties

2021 ◽  
pp. 105659
Author(s):  
Xiaorong Deng ◽  
Yigang Ma ◽  
Yongdong Lei ◽  
Xinrong Zhu ◽  
Lianfu Zhang ◽  
...  
Keyword(s):  
Structural Modification ◽  
Myofibrillar Proteins

scholarly journals Novel Gene Regulation in Normal and Abnormal Spermatogenesis

Cells ◽  
2021 ◽  
Vol 10 (3) ◽  
pp. 666
Author(s):  
Li Du ◽  
Wei Chen ◽  
Zixin Cheng ◽  
Si Wu ◽  
Jian He ◽  
...  
Keyword(s):  
Gene Regulation ◽  
Leydig Cells ◽  
Molecular Mechanisms ◽  
New Technologies ◽  
Genetic Regulation ◽  
Myoid Cells ◽  
Epigenetic Factors ◽  
Future Directions ◽  
New Genes ◽  
Human Spermatogenesis

Spermatogenesis is a complex and dynamic process which is precisely controlledby genetic and epigenetic factors. With the development of new technologies (e.g., single-cell RNA sequencing), increasingly more regulatory genes related to spermatogenesis have been identified. In this review, we address the roles and mechanisms of novel genes in regulating the normal and abnormal spermatogenesis. Specifically, we discussed the functions and signaling pathways of key new genes in mediating the proliferation, differentiation, and apoptosis of rodent and human spermatogonial stem cells (SSCs), as well as in controlling the meiosis of spermatocytes and other germ cells. Additionally, we summarized the gene regulation in the abnormal testicular microenvironment or the niche by Sertoli cells, peritubular myoid cells, and Leydig cells. Finally, we pointed out the future directions for investigating the molecular mechanisms underlying human spermatogenesis. This review could offer novel insights into genetic regulation in the normal and abnormal spermatogenesis, and it provides new molecular targets for gene therapy of male infertility.

Sign in / Sign up

Export Citation Format

Share Document