Different interdigestive antroduodenal motility patterns in chronic antral gastritis with and withoutHelicobacter pylori infection

1993 ◽  
Vol 38 (12) ◽  
pp. 2255-2261 ◽  
Author(s):  
P. A. Testoni ◽  
F. Bagnolo ◽  
E. Masci ◽  
E. Colombo ◽  
A. Tittobello
Keyword(s):  
Antral Gastritis ◽  
Antroduodenal Motility

scholarly journals Manometric evaluation of the interdigestive antroduodenal motility in subjects with fasting bile reflux, with and without antral gastritis.

Gut ◽  
1989 ◽  
Vol 30 (4) ◽  
pp. 443-448 ◽  
Author(s):  
P A Testoni ◽  
L Fanti ◽  
F Bagnolo ◽  
S Passaretti ◽  
M Guslandi ◽  
...  
Keyword(s):  
Bile Reflux ◽  
Antral Gastritis ◽  
Antroduodenal Motility

scholarly journals Is Helicobacter pylori associated hypergastrinaemia due to the bacterium's urease activity or the antral gastritis?

Gut ◽  
1991 ◽  
Vol 32 (11) ◽  
pp. 1286-1290 ◽  
Author(s):  
R S Chittajallu ◽  
C A Dorrian ◽  
W D Neithercut ◽  
S Dahill ◽  
K E McColl
Keyword(s):  
Helicobacter Pylori ◽  
Urease Activity ◽  
Antral Gastritis

scholarly journals Frequency of Helicobacter pylori Infection in Uremic Hemodialyzed Patients with Antral Gastritis

Nephron ◽  
1996 ◽  
Vol 74 (3) ◽  
pp. 621-622 ◽  
Author(s):  
Wojciech Krawczyk ◽  
Ewa Görna ◽  
Janusz Suwata ◽  
Piotr Różyc ◽  
Leszek Pawłowski ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Antral Gastritis

Prolonged ambulatory recording of antroduodenal motility in slow-transit constipation

2000 ◽  
Vol 87 (2) ◽  
pp. 211-217 ◽  
Author(s):  
C. Penning ◽  
H. A. J. Gielkens ◽  
M. Hemelaar ◽  
J. B. V. M. Delemarre ◽  
W. A. Bemelman ◽  
...  
Keyword(s):  
Slow Transit Constipation ◽  
Ambulatory Recording ◽  
Antroduodenal Motility ◽  
Slow Transit ◽  
Transit Constipation

Antroduodenal motility and hormone secretion in chronic pancreatitis

1998 ◽  
Vol 10 (12) ◽  
pp. A39
Author(s):  
M. K. Vu ◽  
J. Vecht ◽  
E. H. Eddes ◽  
I. Biemond ◽  
C. B.H.W. Lamers ◽  
...  
Keyword(s):  
Chronic Pancreatitis ◽  
Hormone Secretion ◽  
Antroduodenal Motility

scholarly journals Verrucous antral gastritis in relation to Helicobacter pylori infection, nutrition, and gastric atrophy

2019 ◽  
Vol 8 (4) ◽  
pp. 293-298
Author(s):  
Naoko Tsuji ◽  
Yasuko Umehara ◽  
Mamoru Takenaka ◽  
Yasunori Minami ◽  
Tomohiro Watanabe ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Reflux Esophagitis ◽  
English Literature ◽  
Indigo Carmine ◽  
Gastric Atrophy ◽  
Helicobacter Pylori Infection ◽  
Antral Gastritis ◽  
Numerous Type ◽  
History Of ◽  
H Pylori

Abstract Background There have been few studies in the English literature regarding verrucous gastritis (VG). The present study investigated the clinical and endoscopic features of verrucous antral gastritis, especially focusing on Helicobacter pylori infection, nutrition, and gastric atrophy. Methods We performed a retrospective study of patients who underwent routine endoscopy with indigo carmine chromoendoscopy and a comparative study was conducted between VG-positive and VG-negative groups. VG was subdivided into classical and numerous types based on the number and distribution of verrucous lesions. Demographic, clinical, and endoscopic data including body mass index (BMI), serum albumin and cholesterol, gastric atrophy, reflux oesophagitis, Barrett’s oesophagus, and H. pylori status were collected. Univariate and multivariable analyses were performed to identify factors associated with VG. Results We analysed the data of 621 patients undergoing routine endoscopy and found that VG (n = 352) was significantly associated with increased BMI (1.12 [1.05–1.18], P < 0.01), reflux esophagitis (1.96 [1.10–3.28], P < 0.01), and H. pylori negativity with or without a history of eradication (9.94 [6.00–16.47] and 6.12 [3.51–10.68], P < 0.001, respectively). Numerous-type (n = 163) VG was associated with both closed- and open-type gastric atrophy (9.9 [4.04–21.37] and 8.10 [3.41–19.24], P < 0.001, respectively). There were no statistical differences between groups regarding age, sex, total cholesterol, albumin, and bile-colored gastric juice. Conclusions Verrucous antral gastritis was related to increased BMI, reflux esophagitis, and H. pylori negativity. Numerous-type verrucous lesions were associated with gastric atrophy. These indicate that VG may be a physiological phenomenon due to high gastric acidity, mechanical overload, and vulnerability of background mucosa.

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