scholarly journals Frequency of Helicobacter pylori Infection in Uremic Hemodialyzed Patients with Antral Gastritis

Nephron ◽  
1996 ◽  
Vol 74 (3) ◽  
pp. 621-622 ◽  
Author(s):  
Wojciech Krawczyk ◽  
Ewa Görna ◽  
Janusz Suwata ◽  
Piotr Różyc ◽  
Leszek Pawłowski ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Antral Gastritis

scholarly journals Is Helicobacter pylori associated hypergastrinaemia due to the bacterium's urease activity or the antral gastritis?

Gut ◽  
1991 ◽  
Vol 32 (11) ◽  
pp. 1286-1290 ◽  
Author(s):  
R S Chittajallu ◽  
C A Dorrian ◽  
W D Neithercut ◽  
S Dahill ◽  
K E McColl
Keyword(s):  
Helicobacter Pylori ◽  
Urease Activity ◽  
Antral Gastritis

scholarly journals Verrucous antral gastritis in relation to Helicobacter pylori infection, nutrition, and gastric atrophy

2019 ◽  
Vol 8 (4) ◽  
pp. 293-298
Author(s):  
Naoko Tsuji ◽  
Yasuko Umehara ◽  
Mamoru Takenaka ◽  
Yasunori Minami ◽  
Tomohiro Watanabe ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Reflux Esophagitis ◽  
English Literature ◽  
Indigo Carmine ◽  
Gastric Atrophy ◽  
Helicobacter Pylori Infection ◽  
Antral Gastritis ◽  
Numerous Type ◽  
History Of ◽  
H Pylori

Abstract Background There have been few studies in the English literature regarding verrucous gastritis (VG). The present study investigated the clinical and endoscopic features of verrucous antral gastritis, especially focusing on Helicobacter pylori infection, nutrition, and gastric atrophy. Methods We performed a retrospective study of patients who underwent routine endoscopy with indigo carmine chromoendoscopy and a comparative study was conducted between VG-positive and VG-negative groups. VG was subdivided into classical and numerous types based on the number and distribution of verrucous lesions. Demographic, clinical, and endoscopic data including body mass index (BMI), serum albumin and cholesterol, gastric atrophy, reflux oesophagitis, Barrett’s oesophagus, and H. pylori status were collected. Univariate and multivariable analyses were performed to identify factors associated with VG. Results We analysed the data of 621 patients undergoing routine endoscopy and found that VG (n = 352) was significantly associated with increased BMI (1.12 [1.05–1.18], P < 0.01), reflux esophagitis (1.96 [1.10–3.28], P < 0.01), and H. pylori negativity with or without a history of eradication (9.94 [6.00–16.47] and 6.12 [3.51–10.68], P < 0.001, respectively). Numerous-type (n = 163) VG was associated with both closed- and open-type gastric atrophy (9.9 [4.04–21.37] and 8.10 [3.41–19.24], P < 0.001, respectively). There were no statistical differences between groups regarding age, sex, total cholesterol, albumin, and bile-colored gastric juice. Conclusions Verrucous antral gastritis was related to increased BMI, reflux esophagitis, and H. pylori negativity. Numerous-type verrucous lesions were associated with gastric atrophy. These indicate that VG may be a physiological phenomenon due to high gastric acidity, mechanical overload, and vulnerability of background mucosa.

scholarly journals Spectrotypic analysis of antibodies to Helicobacter pylori in patients with antral gastritis and duodenal ulcer.

1995 ◽  
Vol 48 (12) ◽  
pp. 1117-1121 ◽  
Author(s):  
R Biselli ◽  
D Vaira ◽  
R Nisini ◽  
M Miglioli ◽  
L Menegatti ◽  
...  
Keyword(s):  
Duodenal Ulcer ◽  
Helicobacter Pylori ◽  
Antral Gastritis

Antral gastritis, Helicobacter pylori, and gastric mucosal sensitivity

1992 ◽  
Vol 102 (3) ◽  
pp. 1088
Author(s):  
Eapen Thomas ◽  
James B. Farnum ◽  
Matthew Rohrbach ◽  
A.T. Mohan ◽  
K.R. Palaniswamy
Keyword(s):  
Helicobacter Pylori ◽  
Antral Gastritis

Superoxide Dismutase Activity in Helicobacter pylori—Positive Antral Gastritis in Children

1996 ◽  
Vol 23 (5) ◽  
pp. 609-613 ◽  
Author(s):  
Efrat Broide ◽  
Elieser Klinowski ◽  
Rina Varsano ◽  
Jerachmiel Eshchar ◽  
Mehrdad Herbert ◽  
...  
Keyword(s):  
Superoxide Dismutase ◽  
Helicobacter Pylori ◽  
Superoxide Dismutase Activity ◽  
Antral Gastritis

scholarly journals Atrophic gastric changes in both Helicobacter felis and Helicobacter pylori infected mice are host dependent and separate from antral gastritis.

Gut ◽  
1996 ◽  
Vol 39 (5) ◽  
pp. 639-648 ◽  
Author(s):  
T Sakagami ◽  
M Dixon ◽  
J O'Rourke ◽  
R Howlett ◽  
F Alderuccio ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Helicobacter Felis ◽  
Antral Gastritis

scholarly journals Helicobacter Pylori Associated Antral Gastritis in Peptic Ulcer Disease Patients and Normal Healthy Population of Kashmir, India

1998 ◽  
Vol 4 (3) ◽  
pp. 135-139 ◽  
Author(s):  
Gh. Jeelani Romshoo ◽  
G. M. Malik ◽  
M. Youssuf Bhat ◽  
Ab. Rashid rather ◽  
Javaid Ahmad Basu ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Peptic Ulcer ◽  
Gastric Ulcer ◽  
Peptic Ulcer Disease ◽  
Healthy Volunteers ◽  
Chronic Gastritis ◽  
Healthy Population ◽  
Ulcer Disease ◽  
Antral Gastritis ◽  
H Pylori

Aim: To study the association of Helicobacter pylori infection with chronic antral gastritis in peptic ulcer disease patients and healthy population of Kashmir.Methods: 50 peptic ulcer patients (duodenal ulcer = 46, gastric ulcer = 2 and combined duodenal and gastric ulcer = 2) and 30 asymptomatic healthy volunteers were included in this study. Peptic ulcer was diagnosed on endoscopic examination. 4–6 punch biopsies were taken from gastric antrum in all the individuals and in case of gastric ulcer an additional biopsy was taken from the edge of the ulcer to exclude its malignant nature. Helicobacter pylori (H. pylori) organism was diagnosed using three different test methods, viz. Histology (using Giemsa Stain), Microbiology (Gram Stain) and Biochemistry (using one minute Endoscopy Room Test). Histological diagnosis of H. pylori was taken as the “gold standard” for the presence of H. pylori organism. Histological diagnosis of gastritis was made using Hematoxylin and Eosin Stain and the gastritis was classified as active chronic gastritis and superficial chronic gastritis.Results: Out of 30 peptic ulcer disease patients with associated antral gastritis, 27 (90%) were positive for H. pylori on histological examination (13 superficial chronic gastritis and 14 active chronic gastritis) whereas out of 8 healthy volunteers with histological evidence of chronic antral gastritis, H. pylori was observed in 7 individuals (87.50%) (4 active chronic gastritis and 3 superficial chronic gastritis).Conclusion: A highly significant association between H. pylori infection with chronic antral gastritis both in peptic ulcer disease patients and healthy volunteers of Kashmir was found in this study. Association between H. pylori infection and chronic gastritis was 90% in peptic ulcer group and 87.50% in healthy population (P<0.005).

Helicobacter pylori and Chronic Antral Gastritis in Elderly Patients

1994 ◽  
Vol 23 (4) ◽  
pp. 277-279 ◽  
Author(s):  
I. A. GILLANDERS ◽  
P. J. W. SCOTT ◽  
G. D. SMITH
Keyword(s):  
Helicobacter Pylori ◽  
Elderly Patients ◽  
Antral Gastritis

scholarly journals 1210Understanding the effect of Helicobacter pylori infection on gastroesophageal reflux disease and Barrett’s oesophagus

2021 ◽  
Vol 50 (Supplement_1) ◽  
Author(s):  
Miss Sabrina Wang ◽  
Ghazaleh Dashti ◽  
Allison Hodge ◽  
Bradley Kendall ◽  
Suzanne Dixon-Suen ◽  
...  
Keyword(s):  
Gastric Cancer ◽  
Helicobacter Pylori ◽  
Gastroesophageal Reflux Disease ◽  
Gastroesophageal Reflux ◽  
Atrophic Gastritis ◽  
Reflux Disease ◽  
Barrett’S Oesophagus ◽  
Case Control Studies ◽  
Barrett's Oesophagus ◽  
Antral Gastritis

Abstract Background Helicobacter pylori (H.pylori) infection causes atrophic gastritis and gastric cancer. Ironically, decreased gastric acid production in those with atrophic gastritis might reduce harmful gastroesophageal refluxate, thereby reducing gastroesophageal reflux disease (GERD) and Barrett’s oesophagus (BE) risk. Methods In two nested case-control studies with 425 GERD and 169 BE cases, we compared sex-specific GERD and BE risk in H.pylori seronegative participants with seropositive participants. Where seronegativity was associated with increased BE risk, we quantified the effect mediated by GERD using a Monte Carlo simulation-based g-computation approach to estimate interventional effects. Moreover, we classified participants into gastritis types using serum pepsinogen-I and gastrin-17 data. Results For men, H.pylori seronegativity was associated with 1.69-fold (CI:1.03-2.75) and 2.14-fold (CI:1.18-3.88) higher odds for GERD and BE respectively. Five (33%) out of the 15 per 1000 excess BE risk from being seronegative was mediated by GERD. No association was observed for women. Among those seropositive, the proportion with atrophic antral gastritis was higher for men than for women (68% vs 56%; p = 0.015). Conclusions H.pylori seronegativity was associated with increased GERD and BE risk for men but not women, which could be partly explained by the higher proportion of H.pylori-associated atrophic antral gastritis in men. Evidence of GERD mediating seronegativity’s effect on BE supports this explanation. Key messages Whilst H.pylori infection might reduce GERD and BE risk, this is potentially a by-product of atrophic gastritis, a risk factor for gastric cancer. Treating GERD could partly reduce the excess BE risk for seronegative individuals.

scholarly journals Immunization against Natural Helicobacter pylori Infection in Nonhuman Primates

1998 ◽  
Vol 66 (9) ◽  
pp. 4340-4346 ◽  
Author(s):  
Andre Dubois ◽  
Cynthia K. Lee ◽  
Nancy Fiala ◽  
Harry Kleanthous ◽  
Patrick T. Mehlman ◽  
...  
Keyword(s):  
Escherichia Coli ◽  
Helicobacter Pylori ◽  
Side Effects ◽  
Nonhuman Primates ◽  
Safety And Efficacy ◽  
Antral Gastritis ◽  
Breeding Groups ◽  
H Pylori ◽  
To Receive ◽  
Monkey Colony

ABSTRACT Helicobacter pylori infection is widespread in some breeding groups of a rhesus monkey colony (71% H. pyloripositive by 1 year), and the rate of seroconversion is also high. As a result, these groups can be used to test the safety and efficacy of an anti-H. pylori vaccine. Nine-month-old female animals were randomized to receive either 8 mg of recombinant urease (rUre) plus 25 μg of Escherichia coli heat-labile enterotoxin (LT) (n = 26) or placebo plus LT (n = 29), given four times at 1-week intervals followed by a booster 1 month later. Ten months after the start of the immunization, the animals were subjected to endoscopy and biopsy samples were obtained. H. pylori negativity was defined as no H. pylori growth by culture and no H. pylori observed at histology. By this criterion, 2 (7%) of 29 animals receiving placebo and 8 (31%) of 26 immunized animals were H. pylori negative (P < 0.035). In addition, antral gastritis score was significantly less in H. pylori-negative immunized monkeys than in H. pylori-positive animals, whether they were given rUre plus LT or placebo plus LT (P < 0.02 orP < 0.01, respectively). Interestingly, antral gastritis was also significantly less in H. pylori-positive animals given rUre plus LT than in H. pylori-positive animals given placebo plus LT (P < 0.02). However, quantitative cultures did not demonstrate significant differences between the two latter groups. It is concluded that oral administration of rUre vaccine plus LT significantly protects nonhuman primates against H. pylori infection while not causing undesirable side effects.

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