Highly-sensitive automatic potentiometer with a high input resistance

1964 ◽  
Vol 7 (1) ◽  
pp. 52-56
Author(s):  
Yu. P. Volkov ◽  
S. G. Rabinovich
Keyword(s):  
Input Resistance ◽  
High Input ◽  
Automatic Potentiometer ◽  
High Input Resistance ◽  
Highly Sensitive

Ih channels prevent overexcitability of early developmental CA1 neurons showing high input resistance in rats

2013 ◽  
Vol 91 ◽  
pp. 14-20 ◽  
Author(s):  
Yoon-Sil Yang ◽  
Moon-Seok Kang ◽  
Seon-Hee Kim ◽  
Se-Jae Kim ◽  
Su-Yong Eun ◽  
...  
Keyword(s):  
Input Resistance ◽  
High Input ◽  
Ca1 Neurons ◽  
High Input Resistance ◽  
Early Developmental

scholarly journals Uniquely excitable neurons enable precise and persistent information transmission through the retrosplenial cortex

2019 ◽  
Author(s):  
Ellen K.W. Brennan ◽  
Shyam Kumar Sudhakar ◽  
Izabela Jedrasiak-Cape ◽  
Omar J. Ahmed
Keyword(s):  
Input Resistance ◽  
Retrosplenial Cortex ◽  
Inhibitory Neurons ◽  
Head Direction ◽  
Cell Type ◽  
Intrinsic Properties ◽  
High Input ◽  
Biophysical Models ◽  
High Input Resistance ◽  
Fast Spiking

ABSTRACTThe retrosplenial cortex (RSC) is essential for both memory and navigation, but the neural codes underlying these functions remain largely unknown. Here, we show that the most prominent cell type in layers 2/3 (L2/3) of the granular RSC is a uniquely excitable, small pyramidal cell. These cells have a low rheobase (LR), high input resistance, lack of spike-frequency adaptation, and spike widths intermediate to those of neighboring fast-spiking (FS) inhibitory neurons and regular-spiking (RS) excitatory neurons. LR cells are excitatory but rarely synapse onto neighboring neurons. Instead, L2/3 of RSC is an inhibition-dominated network with dense connectivity between FS cells and from FS to LR neurons. Biophysical models of LR but not RS cells precisely and continuously encode sustained input from afferent postsubicular head-direction cells. Thus, the unique intrinsic properties of LR neurons can support both the precision and persistence necessary to encode information over multiple timescales in the RSC.

Direct reading dc electro‐optic voltmeter with high input resistance

1977 ◽  
Vol 48 (7) ◽  
pp. 790-792 ◽  
Author(s):  
K. Geisen ◽  
W. Käppler ◽  
H. Kühn
Keyword(s):  
Input Resistance ◽  
High Input ◽  
Direct Reading ◽  
Electro Optic ◽  
High Input Resistance

scholarly journals A PHOTOMIXER DRIVEN TERAHERTZ DIPOLE ANTENNA WITH HIGH INPUT RESISTANCE AND GAIN

2015 ◽  
Vol 44 ◽  
pp. 13-20 ◽  
Author(s):  
Wenfei Yin ◽  
Kenneth Kennedy ◽  
Jayanta Sarma ◽  
Richard A. Hogg ◽  
Salam Khamas
Keyword(s):  
Input Resistance ◽  
Dipole Antenna ◽  
High Input ◽  
High Input Resistance

High input resistance transistor amplifiers

1963 ◽  
Vol 40 (5) ◽  
pp. 239-243 ◽  
Author(s):  
A G Bell ◽  
B H Venning
Keyword(s):  
Input Resistance ◽  
High Input ◽  
High Input Resistance

The effect of axotomy on posttetanic potentiation of group Ia synapses in the cat

1986 ◽  
Vol 56 (4) ◽  
pp. 1174-1184 ◽  
Author(s):  
B. Gustafsson ◽  
M. J. Pinter ◽  
H. Wigstrom
Keyword(s):  
Electrical Properties ◽  
High Resistance ◽  
Previous Analysis ◽  
Normal Population ◽  
Input Resistance ◽  
Posttetanic Potentiation ◽  
High Input ◽  
Synaptic Release ◽  
High Input Resistance ◽  
Passive Electrical Properties

Posttetanic potentiation (PTP) of composite Ia excitatory postsynaptic potentials (EPSPs) has been studied in normal cat alpha-motoneurons and in motoneurons axotomized 2-3 wk earlier by ventral root section. The maximal amount of PTP of EPSP amplitude (expressed relative to unpotentiated amplitude) was considerably less in the axotomized population compared with the normal population. The decrease in PTP provoked by axotomy occurs in association with a postaxotomy increase of input resistance, the net effect being that PTP in axotomized cells was much the same as that observed by others in normal motoneurons possessing similarly high input resistance. In agreement with previous results, EPSP peak amplitudes were decreased after axotomy. This decrease seemed to be largely related to an absence of the largest EPSPs, since otherwise the EPSP distributions of normal and axotomized motoneurons showed considerable overlap. It is suggested that the observed decrease in PTP after axotomy is related to a change in synaptic release properties and not secondary to changes in the electrical properties of motoneurons. A previous analysis has suggested that axotomy causes an alteration of the distribution of passive electrical properties among motoneurons such that axotomized cells resemble normal high-resistance motoneurons. The present results suggest that axotomy may affect the distribution of Ia synaptic release properties in a similar manner, since PTP in axotomized motoneurons resembles that observed in normal high-resistance motoneurons.

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