Cooling the vascular bed of an organ makes anoxia a less effective stimulus

1987 ◽  
Vol 104 (3) ◽  
pp. 1205-1208
Author(s):  
B. I. Tkachenko ◽  
A. A. Nurmatov ◽  
Yu. A. Kudryashov
Hypertension ◽  
1997 ◽  
Vol 30 (5) ◽  
pp. 1260-1266 ◽  
Author(s):  
Hunter C. Champion ◽  
Philip J. Kadowitz

Respiration ◽  
1962 ◽  
Vol 19 (5) ◽  
pp. 362-369
Author(s):  
Peter E. Pool ◽  
Keith H. Averill ◽  
John H.K. Vogel

1991 ◽  
Vol 113 (1) ◽  
pp. 27-29 ◽  
Author(s):  
E. Belardinelli ◽  
M. Ursino ◽  
G. Fabbri ◽  
A. Cevese ◽  
F. Schena

In the present paper pressure changes induced by sudden body acceleration are studied “in vivo” on the dog and compared to the results obtainable with a recently developed mathematical model. A dog was fixed to a movable table, which was accelerated by a compressed air piston for less than 1 s. Acceleration was varied by changing the air pressure in the piston. Pressure was measured during the experiment at different points along the vascular bed. However, only data obtained in the carotid artery and abdominal aorta are presented here. The results demonstrated that impulse body accelerations cause significant pressure peaks in the vessel examined (about + 25 mmHg in the carotid artery with body acceleration of g/2). Moreover, pressure changes are rapidly damped, with a time constant of about 0.1s. From the present results it may be concluded that, according to the prediction of the mathematical model, body accelerations such as those occurring in normal life can induce pressure changes well beyond the normal pressure value.


Sign in / Sign up

Export Citation Format

Share Document