Structural changes of the central nervous system in swayback (enzootic Ataxia) of lambs

1969 ◽  
Vol 12 (4) ◽  
pp. 307-313 ◽  
Author(s):  
P. A. Cancilla ◽  
R. M. Barlow
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Structural Changes ◽  
The Central Nervous System

The Pathology of Milkiness, Thickening, and Opacity of the Pia-arachnoid in the Insane

1895 ◽  
Vol 41 (175) ◽  
pp. 622-635
Author(s):  
W. F. Robertson
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Structural Changes ◽  
Brain Cortex ◽  
Mental Disease ◽  
Waste Products ◽  
The Central Nervous System ◽  
The Subject ◽  
The Brain

There is at the present time great need of more complete and definite knowledge as to the pathology of the very marked structural changes that so commonly affect the pia-arachnoid in the insane. The subject is one of much importance to all of us as medical psychologists, for not only is the condition in question one of the most conspicuous lesions associated with mental disease, but it implicates a structure of primary importance in the economy of the central nervous system. It is by way of vessels that course through this membrane that nutriment is conveyed to the brain cortex, and the waste products resulting from metabolism in the cerebral tissues are mainly conveyed away in the fluid that circulates in its lymph spaces. Therefore it is evident that these morbid changes may very seriously interfere with the functions both of nutrition and excretion in the brain.

scholarly journals Experience-Associated Structural Events, Subependymal Cellular Proliferative Activity, and Functional Recovery After Injury to the Central Nervous System

2000 ◽  
Vol 20 (11) ◽  
pp. 1513-1528 ◽  
Author(s):  
Timothy Schallert ◽  
J. Leigh Leasure ◽  
Bryan Kolb
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Functional Recovery ◽  
Proliferative Activity ◽  
Structural Changes ◽  
Direct Interaction ◽  
Recovery Process ◽  
Intervention Strategies ◽  
Brain Areas ◽  
The Central Nervous System

Considerable structural plasticity is possible in the damaged neocortex and connected brain areas, and the potential for significant functional recovery remains even during the chronic phases of the recovery process. In this article, the authors review the literature on use-dependent morphologic events, focusing on the direct interaction of behavioral experience and structural changes associated with plasticity and degeneration. Experience-associated neural changes have the potential to either hinder or enhance functional recovery; therefore, issues concerning the nature, timing, and intensity of behavior-based intervention strategies are addressed.

What are “normal movements” in atypical populations?

1996 ◽  
Vol 19 (1) ◽  
pp. 55-68 ◽  
Author(s):  
Mark L. Latash ◽  
J. Greg Anson
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Structural Changes ◽  
Motor Patterns ◽  
Control Structures ◽  
Current State ◽  
Primary Disorder ◽  
Motor Problems ◽  
The Central Nervous System ◽  
Control Patterns

AbstractRedundancy of the motor control system is an important feature that gives the central control structures options for solving everyday motor problems. The choice of particular control patterns is based on priorities (coordinative rules) that are presently unknown. Motor patterns observed in unimpaired young adults reflect these priorities. We hypothesize that under certain atypical conditions, which may include disorders in perception of the environment and in decision making, structural or biochemical changes within the central nervous system (CNS), and/or structural changes of the effectors, the central nervous system may reconsider its priorities. A new set of priorities will reflect the current state of the system and may lead to different patterns of voluntary movement. Under such conditions, changed motor patterns should be considered not pathological but rather adaptive to a primary disorder and may even be viewed as optimal for a given state of the system of movement production. Therapeutic approaches should not be directed toward restoring the motor patterns to as close to “normal” as possible but rather toward resolving the original underlying problem. We illustrate this approach using, as examples, movements in amputees, in patients with Parkinson's disease, in patients with dystonia, and in persons with Down syndrome.

Sign in / Sign up

Export Citation Format

Share Document