Influence of the vertebral nerve on the cochlear circulation

1973 ◽  
Vol 75 (6) ◽  
pp. 610-612
Author(s):  
V. A. Romanov ◽  
L. G. Miller ◽  
M. D. Gaevyi
Keyword(s):  
Vertebral Nerve

The vertebral nerve revisited

2007 ◽  
Vol 20 (6) ◽  
pp. 644-647 ◽  
Author(s):  
R. Shane Tubbs ◽  
Marios Loukas ◽  
Allison C. Remy ◽  
Mohammadali M. Shoja ◽  
E. George Salter ◽  
...  
Keyword(s):  
Vertebral Nerve

Vertebral Nerve and Plexus

1957 ◽  
Vol 74 (3) ◽  
pp. 430 ◽  
Author(s):  
HENRY H. HOFFMAN
Keyword(s):  
Vertebral Nerve

The Anatomy and Physiology of the Vertebral Nerve in Relation to Cervical Migraine

Cephalalgia ◽  
1981 ◽  
Vol 1 (1) ◽  
pp. 11-24 ◽  
Author(s):  
Nikolai Bogduk ◽  
Geoffrey A. Lambert ◽  
John W. Duckworth
Keyword(s):  
Vertebral Artery ◽  
Sympathetic Trunk ◽  
Pathogenetic Mechanism ◽  
Anatomy And Physiology ◽  
Cervical Sympathetic Trunk ◽  
Rami Communicantes ◽  
Cervical Sympathetic ◽  
Vertebral Nerve ◽  
Stimulation Of ◽  
Carotid Flow

The anatomy of the vertebral nerve was investigated in humans and in monkeys. The effect of stimulation of the vertebral nerve and the cervical sympathetic trunk in the monkey was studied. The vertebral nerves in man and monkey represent a series of deep grey rami communicantes which form intersegmental neural arcades around the vertebral artery between C7 and C3. Above C3 the vertebral artery is accompanied by direct branches from the C1–3 ventral rami. Electrical stimulation of either the vertebral nerve or the cervical sympathetic trunk had a minimal effect on vertebral blood flow. In contrast, sympathetic stimulation had pronounced effects on carotid flow and resistance. Anatomically and physiologically there are no grounds to support the hypothesis that irritation of the “vertebral nerve” is the pathogenetic mechanism of cervical migraine.

Role of the sinu-vertebral nerve in low back pain and anatomical basis of therapeutic implications

2002 ◽  
Vol 24 (6) ◽  
pp. 366-370 ◽  
Author(s):  
S. Raoul ◽  
A. Faure ◽  
R. Robert ◽  
J.-M. Rogez ◽  
O. Hamel ◽  
...  
Keyword(s):  
Low Back Pain ◽  
Back Pain ◽  
Low Back ◽  
Therapeutic Implications ◽  
Anatomical Basis ◽  
Vertebral Nerve

scholarly journals Spinal segments communicating resting sympathetic activity to postganglionic nerves of the stellate ganglion

1998 ◽  
Vol 275 (2) ◽  
pp. R400-R409 ◽  
Author(s):  
Bernat Kocsis ◽  
Katalin Gyimesi-Pelczer
Keyword(s):  
Spinal Cord ◽  
Sympathetic Activity ◽  
Stellate Ganglion ◽  
Rhythmic Activity ◽  
Spinal Segment ◽  
Preganglionic Neurons ◽  
Spinal Segments ◽  
Sympathetic Reflexes ◽  
Adjacent Segments ◽  
Vertebral Nerve

It has been shown earlier using sympathetic reflexes and anatomic techniques that preganglionic neurons controlling different effectors occupy wide and overlapping ranges of adjacent segments in the spinal cord (cardiac: T1–T7, vertebral: T2–T8). Because, however, the majority of preganglionic neurons are silent at resting states, the present study was designed to estimate the segmental map of subsets of these neurons including only those active at rest using simultaneous recordings from the inferior cardiac and vertebral nerves, under chloralose-urethan or urethan anesthesia. In 22 cats, thoracic white rami T1–T8 were cut in a sequential manner. Three-minute-long data segments were recorded between sectionings and analyzed in the frequency domain using the fast Fourier transform. We found that cardiac and vertebral active maps involved segments T3–T5 and T4–T8, respectively. In individual experiments, however, most of the power of rhythmic activity originated from only one or two segments and the dominant segments for the two nerves never overlapped. Moreover, the separation between dominant segments generating cardiac and vertebral nerve discharges was wider and the distribution of tonically active preganglionic neurons projecting to each nerve was narrower under urethan than chloralose-urethan anesthesia. We conclude that the proportion of active to quiescent preganglionic neurons regulating cardiac and vertebral nerve discharges varies from spinal segment to segment and that active neurons projecting to these nerves are nonoverlapping.

Dorsomedial medulla is more susceptible than rostral ventrolateral medulla to hypoxic insult in cats

2001 ◽  
Vol 90 (1) ◽  
pp. 248-260 ◽  
Author(s):  
Ling-Zong Hong ◽  
Jon-Son Kuo ◽  
Mao-Hsiung Yen ◽  
Chok-Yung Chai
Keyword(s):  
Arterial Pressure ◽  
Sympathetic Nerve ◽  
Sympathetic Nerve Activity ◽  
Systemic Arterial Pressure ◽  
Rostral Ventrolateral Medulla ◽  
Ventrolateral Medulla ◽  
Nerve Activity ◽  
Pressor Responses ◽  
Norepinephrine Depletion ◽  
Vertebral Nerve

We investigated the responses of systemic arterial pressure and vertebral sympathetic nerve activity to glutamate microinjections (0.1 M, 70 nl) in the dorsomedial (DM) and the rostral ventrolateral medulla (RVLM) before hypoxia and after reoxygenation (posthypoxia) after various degrees of hypoxia in anesthetized cats. Hypoxia was produced by ventilating 5% O2 and 95% N2 for different durations (hypoxia I-III). In intact cats, the glutamate-induced systemic arterial pressure and vertebral nerve activity responses of the DM were depressed after all degrees of hypoxia. Posthypoxic depression in the RVLM, however, was not observed until hypoxia II and III. Precollicular decerebration prevented depression in the RVLM, but, for the DM, it was effective only for hypoxia I. Baro- and chemoreceptor denervation abolished all posthypoxic depression in both the DM and the RVLM. Pressor responses to tyramine (100–400 μg/kg iv) remained unchanged after all degrees of hypoxia. These results suggest that the DM is more susceptible to hypoxia than the RVLM. The peripheral baro- and chemoreceptors and the suprapontine structures apparently play an important role in posthypoxic depression. Moreover, the depression is not due to the postganglionic norepinephrine depletion.

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