Effects of fluphenazine enanthate and fluphenazine decanoate on discriminated avoidance response and water drinking behavior in rats

1979 ◽  
Vol 65 (1) ◽  
pp. 1-6 ◽  
Author(s):  
Hisashi Kuribara ◽  
Sakutaro Tadokoro
Keyword(s):  
Avoidance Response ◽  
Drinking Behavior ◽  
Fluphenazine Decanoate ◽  
Water Drinking ◽  
Fluphenazine Enanthate

Clozapine Reduces Water-Drinking Behavior in Schizophrenic Patients With Polydipsia

1996 ◽  
Vol 16 (4) ◽  
pp. 329-332 ◽  
Author(s):  
Matthew A. Fuller ◽  
George Jurjus ◽  
Kong Kwon ◽  
P. Eric Konicki ◽  
George E. Jaskiw
Keyword(s):  
Drinking Behavior ◽  
Water Drinking ◽  
Schizophrenic Patients

Long-acting injectable antipsychotic agents

1979 ◽  
Vol 17 (11) ◽  
pp. 41-43
Keyword(s):  
Antipsychotic Agents ◽  
Fluphenazine Decanoate ◽  
Fluphenazine Enanthate ◽  
Long Acting ◽  
Flupenthixol Decanoate

Five long-acting injectable antipsychotic agents (neuroleptics) are available. They are fluphenazine enanthate (Moditen enanthate),1 fluphenazine decanoate (Modecate),2 flupenthixol decanoate (Depixol), cis-clopenthixol decanoate (Clopixol) and fluspirilene (Redeptin).3 Their therapeutic and unwanted effects are broadly similar to those of their oral forms. Nevertheless, injectable preparations have distinct advantages as well as problems.

Excessive water drinking behavior in autism

1999 ◽  
Vol 21 (2) ◽  
pp. 103-106 ◽  
Author(s):  
Katsuyuki Terai ◽  
Toshio Munesue ◽  
Michio Hiratani
Keyword(s):  
Drinking Behavior ◽  
Water Drinking ◽  
Excessive Water

Circulating angiotensin II and drinking behavior in rats

1990 ◽  
Vol 259 (3) ◽  
pp. R531-R538 ◽  
Author(s):  
C. M. Pawloski ◽  
G. D. Fink
Keyword(s):  
Angiotensin Ii ◽  
Water Intake ◽  
Dose Range ◽  
Drinking Behavior ◽  
Ang Ii ◽  
Sprague Dawley ◽  
Physiological Conditions ◽  
Water Drinking ◽  
Sprague Dawley Rats ◽  
Before And After

This study was designed to investigate the effects on water drinking of acute and chronic increases in circulating angiotensin II (ANG II) concentrations in rats. Experiments were conducted in male Sprague-Dawley rats chronically instrumented with femoral arterial and venous catheters and permanently housed in metal metabolism cages. ANG II was infused intravenously either acutely (30 min-2 h) or chronically (3 days) in a dose range of 10-60 ng/min. In no instance did such infusions cause a statistically significant increase in water intake. Other experiments examined the influence of ANG II (10 ng/min iv) on drinking elicited by infusion of hypertonic sodium chloride (1.5 M at 3.5 microliters/min). ANG II administration did not increase drinking to a hypertonic saline stimulus or lower the osmotic threshold for drinking. Nitroprusside (12 micrograms/min) was infused for 30 min to produce hypotension and drinking. Water intake associated with this stimulus was not changed by blocking ANG II formation with enalapril (2 mg/kg iv) or by concomitant infusion of ANG II (10 ng/min iv). Finally, plasma ANG II concentrations were measured before and after 1-h intravenous infusion of saline or ANG II to determine the levels of circulating ANG II produced by the infusion rates used here. It is concluded that the range of circulating ANG II concentrations found under most physiological conditions in rats does not directly stimulate drinking or participate importantly in osmotic or hypotension-induced drinking.

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