scholarly journals The oxygen transport system of red blood cells during diabetic ketoacidosis and recovery

Diabetologia ◽  
1975 ◽  
Vol 11 (4) ◽  
pp. 255-260 ◽  
Author(s):  
J. Ditzel ◽  
E. Standl
Keyword(s):  
Red Blood Cells ◽  
Diabetic Ketoacidosis ◽  
Transport System ◽  
Oxygen Transport ◽  
Blood Cells ◽  
Oxygen Transport System

scholarly journals SEASONAL DYNAMICS OF ENDOCRINE REGULATION OF METABOLIC RATE, BLOOD OXYGEN TRANSPORT SYSTEM AND PHYSICAL PERFORMANCE OF MIDDLE OB REGION ATHLETES FROM CYCLIC WINTER SPORTS

2020 ◽  
Vol 20 (3) ◽  
pp. 41-50
Author(s):  
A Rutkovskiy ◽  
An Koynosov ◽  
A Gubina
Keyword(s):  
Red Blood Cells ◽  
Physical Performance ◽  
Transport System ◽  
Oxygen Transport ◽  
Blood Cells ◽  
Physical Work ◽  
Physical Work Capacity ◽  
Endocrine Regulation ◽  
Blood Oxygen ◽  
Oxygen Transport System

Aim. The paper aims to study the seasonal dynamics of endocrine regulation of metabolic rate, blood oxygen transport system and physical performance of Middle Ob region athletes from cyclic winter sports. Materials and methods. A comprehensive two-stage study of the endocrine system, blood oxygen transport system and physical performance was carried out among elite cross country skiers and biathletes aged from 15 to 18 years during the periods of the year with pronounced daylight variability. The research methods included enzyme-linked immunoassay (ELISA) with the measurements of total triiodothyronine, total thyroxine, thyroid-stimulating hormone, total testosterone and growth hormone; analysis of peripheral blood with the measurements of red blood cells, hemoglobin, hematocrit and red blood cell indices; cardiopulmonary exercise testing with ergospirometry and assessment of maximum oxygen consumption, physical work capacity, oxygen pulse and anaerobic threshold. Results. Statistical analysis revealed significant (p < 0.05) seasonal changes in the endocrine system, blood oxygen transport system and physical performance of Middle Ob region athletes. Namely, during the short daylight period there was a significant increase in total triiodothyronine and cortisol, a decrease in total testosterone, red blood cells, hemoglobin, hematocrit and the average volume of red blood cells, an increase in absolute and relative values of maximum oxygen consumption, oxygen pulse, physical work capacity and a decrease in anaerobic threshold. Conclusion. The results of the research characterize the features of athletes’ adaptation to specific climatic factors of the Northern region against the background of intense physical exertion.

scholarly journals Organization of blood oxygen transport system for cottoid fishes of Lake Baikal

2020 ◽  
pp. 826-827
Author(s):  
V. Yakhnenko ◽  
◽  
I. Klimenkov ◽  
N. Sudakov ◽  
S. Kirilchik ◽  
...  
Keyword(s):  
Transport System ◽  
Lake Baikal ◽  
Oxygen Transport ◽  
Blood Oxygen ◽  
Cottoid Fishes ◽  
Oxygen Transport System

scholarly journals Ouabain-Insensitive Sodium Movements in the Human Red Blood Cell

1971 ◽  
Vol 57 (3) ◽  
pp. 259-282 ◽  
Author(s):  
John R. Sachs
Keyword(s):  
Blood Cell ◽  
Red Blood Cells ◽  
Transport System ◽  
Rate Constant ◽  
Red Blood Cell ◽  
Blood Cells ◽  
Electrochemical Gradient ◽  
Sodium Influx ◽  
K Concentration

Red blood cells exposed to ouabain are capable of net Na outflux against an electrochemical gradient; the net outflux is inhibited by the diuretic, furosemide. In ouabain-treated cells, both the unidirectional Na outflux and the unidirectional Na influx are inhibited by furosemide. Furosemide also inhibits the ouabain-sensitive Na-Na exchange accomplished by the Na-K pump in K-free solutions. From the interaction of extracellular K, furosemide, and ouabain with the transport system, it seems possible that the ouabain-insensitive Na outflux is accomplished by the same mechanism that is responsible for the ouabain-sensitive Na-K exchange. The ouabain-insensitive Na outflux is increased by extracellular Na, and the influx increases as the intracellular Na increases. In fresh cells, high extracellular K concentrations decrease the ouabain-insensitive Na outflux and increase the ouabain-insensitive Na influx. When the rate constant for sodium outflux and the rate constant for sodium influx in ouabain-treated cells are plotted against the extracellular K concentration, the curves obtained are mirror images of each other. In starved cells, extracellular K increases the ouabain-insensitive Na outflux as does extracellular Na, and it has little effect on the Na influx.

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