Genetic studies of erythromycin resistant mutants of Escherichia coli

1974 ◽  
Vol 135 (3) ◽  
pp. 257-268 ◽  
Author(s):  
D. Pardo ◽  
R. Rosset
1973 ◽  
Vol 127 (2) ◽  
pp. 175-189 ◽  
Author(s):  
H. G. Wittmann ◽  
G. Stöffler ◽  
D. Apirion ◽  
L. Rosen ◽  
K. Tanaka ◽  
...  

1972 ◽  
Vol 110 (1) ◽  
pp. 186-195 ◽  
Author(s):  
Simon Silver ◽  
Paula Johnseine ◽  
Eleanor Whitney ◽  
Dan Clark

1969 ◽  
Vol 105 (3) ◽  
pp. 219-224 ◽  
Author(s):  
Satoshi Dekio ◽  
Renkichi Takata

1976 ◽  
Vol 66 (2) ◽  
pp. 369-377 ◽  
Author(s):  
Peter PREHM ◽  
Stephan STIRM ◽  
Barbara JANN ◽  
Klaus JANN ◽  
Hans G. BOMAN

2012 ◽  
Vol 78 (9) ◽  
pp. 3488-3491 ◽  
Author(s):  
Jinman Liu ◽  
Zhoujie Xie ◽  
Justin Merritt ◽  
Fengxia Qi

ABSTRACTWe have constructed the firstEscherichia coli-Veillonellashuttle vector based on an endogenous plasmid (pVJL1) isolated from a clinicalVeillonellastrain. A highly transformableVeillonellastrain was also identified. Both the shuttle vector and the transformable strain should be valuable tools for futureVeillonellagenetic studies.


2001 ◽  
Vol 45 (9) ◽  
pp. 2432-2435 ◽  
Author(s):  
Peter Margolis ◽  
Corinne Hackbarth ◽  
Sara Lopez ◽  
Mita Maniar ◽  
Wen Wang ◽  
...  

ABSTRACT Resistance to peptide deformylase inhibitors in Escherichia coli or Staphylococcus aureus is due to inactivation of transformylase activity. Knockout experiments in Streptococcus pneumoniae R6x indicate that the transformylase (fmt) and deformylase (defB) genes are essential and that adef paralog (defA) is not. Actinonin-resistant mutants of S. pneumoniae ATCC 49619 harbor mutations indefB but not in fmt. Reintroduction of the mutated defB gene into wild-type S. pneumoniaeR6x recreates the resistance phenotype. The altered enzyme displays decreased sensitivity to actinonin.


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