Explanations accounting for transduction by bacteriophage λ in maltose negative bacteriophage λ resistant mutants of Escherichia coli K-12

1978 ◽  
Vol 159 (2) ◽  
pp. 143-149 ◽  
Author(s):  
C. Braun-Breton ◽  
M. Hofnung
Keyword(s):  
Escherichia Coli ◽  
Bacteriophage Λ ◽  
Resistant Mutants ◽  
K 12

scholarly journals Cell-Wall Lipopolysaccharides of Ampicillin-Resistant Mutants of Escherichia coli K-12

1976 ◽  
Vol 66 (2) ◽  
pp. 369-377 ◽  
Author(s):  
Peter PREHM ◽  
Stephan STIRM ◽  
Barbara JANN ◽  
Klaus JANN ◽  
Hans G. BOMAN
Keyword(s):  
Escherichia Coli ◽  
Cell Wall ◽  
Resistant Mutants ◽  
K 12

scholarly journals Isolation and characterization of norfloxacin-resistant mutants of Escherichia coli K-12.

1986 ◽  
Vol 30 (2) ◽  
pp. 248-253 ◽  
Author(s):  
K Hirai ◽  
H Aoyama ◽  
S Suzue ◽  
T Irikura ◽  
S Iyobe ◽  
...  
Keyword(s):  
Escherichia Coli ◽  
Isolation And Characterization ◽  
Resistant Mutants ◽  
K 12

scholarly journals Isolation and Characterization of Tributyltin Resistant Mutants of Escherichia coli

1984 ◽  
Vol 39 (3-4) ◽  
pp. 293-299 ◽  
Author(s):  
Akhand P. Singh ◽  
Kiran Singh
Keyword(s):  
Escherichia Coli ◽  
Class Ii ◽  
Class I ◽  
Isolation And Characterization ◽  
Membrane Bound ◽  
Resistant Mutants ◽  
K 12 ◽  
Spontaneous Mutants ◽  
Wild Type Parent

Two classes of tributyltin (TBT) resistant, spontaneous mutants of Escherichia coli K-12 were isolated, using a cytochrome containing (W 1485) and a cytochrome deficient (SASX76) strain. In contrast to the cytochrome sufficient strain, the cytochrome deficient strain was found to be fifty times more sensitive to TBT. The class I mutants, isolated from strain W 1485, also showed crossresistance to triphenyltin (TPT). As compared to its wild type parent, the TBT-resistant mutants exhibited mucoid colony type, aberrant cell morphology and reduced uptake of TPT. Based on these results, it was suggested that the resistance of class I mutants to TBT may be associated with above mentioned alterations. The class II TBT-resistant mutants were isolated from the cytochrome deficient strain, SASX76. In comparison to class I mutants, these class II mutants were found to have TBT-resistant membrane bound adenosine triphosphatase (ATPase) which may account for their resistance to TBT

scholarly journals Modulation of DNA Repair and Recombination by the Bacteriophage λ Orf Function in Escherichia coli K-12

2004 ◽  
Vol 186 (9) ◽  
pp. 2699-2707 ◽  
Author(s):  
Anthony R. Poteete
Keyword(s):  
Escherichia Coli ◽  
Dna Repair ◽  
Uv Radiation ◽  
Bacteriophage Λ ◽  
Wild Type ◽  
Uv Sensitivity ◽  
Mutant Phenotypes ◽  
K 12 ◽  
Related Plasmid

ABSTRACT The orf gene of bacteriophage λ, fused to a promoter, was placed in the galK locus of Escherichia coli K-12. Orf was found to suppress the recombination deficiency and sensitivity to UV radiation of mutants, in a Δ(recC ptr recB recD)::P tac gam bet exo pae cI ΔrecG background, lacking recF, recO, recR, ruvAB, and ruvC functions. It also suppressed defects of these mutants in establishing replication of a pSC101-related plasmid. Compared to orf, the recA803 allele had only small effects on recF, recO, and recR mutant phenotypes and no effect on a ruvAB mutant. In a fully wild-type background with respect to known recombination and repair functions, orf partially suppressed the UV sensitivity of ruvAB and ruvC mutants.

scholarly journals Glutamyl-γ-methyl ester acts as a methionine analogue inEscherichia coli: analogue resistant mutants map at themetJandmetKloci

1979 ◽  
Vol 33 (1) ◽  
pp. 49-55 ◽  
Author(s):  
Jan Kraus ◽  
Dieter Soll ◽  
K. Brooks Low
Keyword(s):  
Escherichia Coli ◽  
Amino Acid ◽  
Methyl Ester ◽  
Gene Order ◽  
Cross Resistance ◽  
Inescherichia Coli ◽  
Amino Acid Analogues ◽  
Resistant Mutants ◽  
K 12

SUMMARYEscherichia coliK-12 mutants resistant to glutamyl-γ-methyl ester were isolated. A mutation leading to resistance of up to 1·4 mg/ml of the methionine analogue maps at min 63 and is 13% cotransducible withserAindicating an alteration in themetKgene. Another mutation leading to resistance to 3 mg/ml of the analogue and cross-resistance to other amino acid analogues maps at min 87. This mutation, which has the phenotype of MetJ−, is shown to be situated between theglpKandmetBgenes and thus indicates a different gene order from the published one.

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