Ethane Production and Release in Comet C/1995 O1 Hale–Bopp

Icarus ◽  
2001 ◽  
Vol 153 (1) ◽  
pp. 162-179 ◽  
Author(s):  
N Russo
Keyword(s):  
1986 ◽  
Vol 251 (5) ◽  
pp. F839-F843 ◽  
Author(s):  
M. S. Paller ◽  
R. P. Hebbel

After renal ischemia, oxygen free radicals are formed and produce tissue injury, in large part, through peroxidation of polyunsaturated fatty acids. We used an in vivo method to monitor lipid peroxidation after renal ischemia, the measurement of ethane in expired gas, to determine the time course of lipid peroxidation and the effect of several agents to limit lipid peroxidation after renal ischemia. In anesthetized rats there was no significant increase in ethane production during 60 min of renal ischemia. During the first 10 min of renal reperfusion, there was a prompt increase in ethane production from 2.9 +/- 1.3 to 6.3 +/- 1.9 pmol/min (P less than 0.05). Ethane production was significantly increased during the first 50 min of reperfusion and then rapidly tapered to base-line levels. Preischemic administration of allopurinol to prevent superoxide radical generation or the superoxide radical scavenger superoxide dismutase prevented the increase in ethane production during postischemic reperfusion. These studies confirm that there is increase lipid peroxidation following renal ischemia that can be prevented by agents which limit the formation or accumulation of oxygen free radicals. This in vivo method for measuring lipid peroxidation could also be employed to study the effects of ischemia on lipid peroxidation in other organs, as well as to monitor lipid peroxidation in other forms of injury.


1988 ◽  
Vol 153 (2) ◽  
pp. 535-539 ◽  
Author(s):  
Anthony L. Kiorpes ◽  
John W. Sword ◽  
William G. Hoekstra
Keyword(s):  

1987 ◽  
Vol 36 (2) ◽  
pp. 142-147 ◽  
Author(s):  
Gary Slater ◽  
Jae Kang ◽  
Gerald Cohen ◽  
Arnold Szporn ◽  
Arthur H. Aufses

1982 ◽  
Vol 37 (3-4) ◽  
pp. 205-209 ◽  
Author(s):  
Georg Schmetterer

Abstract The cyanobacterium Anacystis nidulans is bleached when subjected to both light and O2 to­ gether with suitable (pre)treatment o f the cells such as incubation at high (≧ 48 °C) or low (≦ 17 °C) temperatures, or in presence of metabolic inhibitors, or of substances forming com­ plexes with divalent cations. Concomitantly degradation o f the intracellular membranes is ob­ served (G. Schmetterer, G. A. Peschek, Biochem. Physiol. Pflanzen 176, 90-100 (1981)). The same three conditions cause formation of hydrocarbons, mostly ethane, a characteristic product of lipid peroxidation. Ethane production is unchanged and still light-sensitive even when no more pigments can be detected in the cells. In "white" cells light-dependent O2-uptake is also observed. The action spectrum of this process suggests that "completely" bleached cells retain very small amounts of residual chlorophyll, which must be unusually resistant to photooxidation.


FEBS Letters ◽  
1981 ◽  
Vol 126 (2) ◽  
pp. 241-244 ◽  
Author(s):  
Armun Müller ◽  
Peter Graf ◽  
Albrecht Wendel ◽  
Helmut Sies

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