Plasmon-Assisted Production of Reactive Oxygen Species by Single Gold Nanorods

Small ◽  
2015 ◽  
Vol 11 (35) ◽  
pp. 4475-4479 ◽  
Author(s):  
Timothée Labouret ◽  
Jean-Frédéric Audibert ◽  
Robert B. Pansu ◽  
Bruno Palpant
Keyword(s):  
Reactive Oxygen Species ◽  
Gold Nanorods ◽  
Reactive Oxygen ◽  
Oxygen Species

scholarly journals TiO2-Capped Gold Nanorods for Plasmon-Enhanced Production of Reactive Oxygen Species and Photothermal Delivery of Chemotherapeutic Agents

2018 ◽  
Vol 10 (33) ◽  
pp. 27965-27971 ◽  
Author(s):  
Liangcan He ◽  
Chenchen Mao ◽  
Michael Brasino ◽  
Albert Harguindey ◽  
Wounjhang Park ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Gold Nanorods ◽  
Reactive Oxygen ◽  
Chemotherapeutic Agents ◽  
Oxygen Species ◽  
Enhanced Production

Coating gold nanorods with silica prevents the generation of reactive oxygen species under laser light irradiation for safe biomedical applications

2022 ◽  
Author(s):  
Sarra Mitiche ◽  
Syrine Gueffrache ◽  
Sylvie Marguet ◽  
Jean-Frédéric Audibert ◽  
Robert Bernard Pansu ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Gold Nanoparticles ◽  
Photodynamic Therapy ◽  
Gold Nanorods ◽  
Biomedical Applications ◽  
Laser Light ◽  
Reactive Oxygen ◽  
Light Irradiation ◽  
Oxygen Species ◽  
Pulsed Light

Gold nanoparticles can produce reactive oxygen species (ROS) under the action of ultrashort pulsed light. While beneficial for photodynamic therapy, this phenomenon is prohibitive for other biomedical applications such as...

Mitochondria as a source of reactive oxygen species

2009 ◽  
pp. c3 ◽  
Author(s):  
Helena M. Cochemé ◽  
Michael P. Murphy
Keyword(s):  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Oxygen Species

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

Fas ligand expression in rat kupffer cells in response to lipopolysaccharide is mediated by reactive oxygen species

2001 ◽  
Vol 120 (5) ◽  
pp. A361-A361
Author(s):  
K UCHIKURA ◽  
T WADA ◽  
Z SUN ◽  
S HOSHINO ◽  
G BULKLEY ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Kupffer Cells ◽  
Fas Ligand ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Ligand Expression
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