The ultimobranchial gland and congenital thyroid abnormalities in man

1989 ◽  
Vol 159 (2) ◽  
pp. 135-141 ◽  
Author(s):  
E. D. Williams ◽  
C. E. Toyn ◽  
H. R. Harach
2021 ◽  
Vol 6 (4) ◽  
pp. S148-S149
Author(s):  
E. Sabath ◽  
L. Cárdenas-Rodríguez ◽  
M.L. Robles-Osorio

2013 ◽  
Vol 34 (3) ◽  
pp. 226-235 ◽  
Author(s):  
Bhavik N. Patel ◽  
Aya Kamaya ◽  
Terry S. Desser

Thyroid ◽  
1997 ◽  
Vol 7 (6) ◽  
pp. 847-852 ◽  
Author(s):  
YODPHAT KRAUSZ ◽  
MICHAEL WILK ◽  
FUAD SALIMAN ◽  
ROLAND CHISIN

2015 ◽  
Vol 2015 ◽  
pp. 1-4
Author(s):  
Ruohollah Seddigh ◽  
Somayeh Azarnik ◽  
Amir-Abbas Keshavarz-Akhlaghi

There are many reports that show different thyroid abnormalities in schizophrenia without clear establishment of their role in etiology and treatment outcome of schizophrenia. Among these reports, there are only a few that consider a role for thyroid hormones as augmenting agents in the treatment with antipsychotic drugs. This case report outlines symptom subsidence of a patient with clozapine refractory paranoid schizophrenia and normal thyroid function who added levothyroxine to clozapine and found that symptoms of psychosis returned once levothyroxine was discontinued. Although this observation needs to be confirmed in controlled clinical trials, we aimed to discuss possible hypothesized mechanisms underlying this observation.


1992 ◽  
Vol 26 (3) ◽  
pp. 173-178 ◽  
Author(s):  
Sing Lee ◽  
Chun Chung Chow ◽  
Yun Kwok Wing ◽  
Chi Chung Shek

2019 ◽  
Vol 117 ◽  
pp. 150
Author(s):  
Trude Seselie Jahr Vedal ◽  
Nils Eiel Steen ◽  
Kåre I. Birkeland ◽  
Ingrid Dieset ◽  
Elina J. Reponen ◽  
...  

2009 ◽  
Vol 29 (12) ◽  
pp. 963-968 ◽  
Author(s):  
Ajai K. Srivastav ◽  
Sarita Singh ◽  
Diwakar Mishra ◽  
S.K. Srivastav

1997 ◽  
Vol 43 (4) ◽  
pp. 3-6
Author(s):  
E. P. Kasatkina ◽  
D. E. Shilin ◽  
G. V. Ibragimova ◽  
M. I. Pykov ◽  
G. A. Ryumin ◽  
...  

The incidence of endemic goiter in the Orel district of Russia has been studied in order to assess the informative value of the new WHO classification for identification of iodine deficiencies (1994). A total of 2103 children living in three rural regions, two of which were polluted with low-dose radiation after the Chernobyl accident, have been examined in 1992-1996. The incidence of goiter and thyroid dysfunction in the newborns corresponded to grave endemia, and the median of blood thyroglobulin was moderately increased in the region with the highest pollution (5 to 15 Ci/km2 for 127Cs), characterized by a slight iodine deficiency. In a less contaminated region (1 to 5 Ci/km2) with a similar level of iodine deficiency, incidence of goiter, and median of blood thyroglobulin the incidence of thyroid dysfunction in the newborns was moderate. In a pure region, despite a more evident iodine deficit and a compatible increase of the median of blood thyroglobulin, the incidence of endemic goiter and thyroid dysfunction of the newborns was the minimal. Hence, the values of the analyzed iodine deficit indicators did not correlate with the actual iodine deficiency in all the regions examined. These differences suggest a possible contribution of strumogenic factors other than iodine deficit to the formation of goiter endemia in the examined district. The detection of these factors in the environment is needed for objective evaluation of the causes and severity of the endemic process and for the development of differentiated approaches to the prevention and treatment of thyroid abnormalities in the population exposed to radiation.


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