scholarly journals Fat‐Shaped Microbiota Affects Lipid Metabolism, Liver Steatosis, and Intestinal Homeostasis in Mice Fed a Low‐Protein Diet

2020 ◽  
Vol 64 (15) ◽  
pp. 1900835 ◽  
Author(s):  
Marco Busnelli ◽  
Stefano Manzini ◽  
Amin Jablaoui ◽  
Aurélia Bruneau ◽  
Aïcha Kriaa ◽  
...  
Keyword(s):  
Lipid Metabolism ◽  
Liver Steatosis ◽  
Protein Diet ◽  
Low Protein Diet ◽  
Intestinal Homeostasis ◽  
Low Protein

Effect of different microbiota on lipid metabolism, liver steatosis and intestinal homeostasis in mice fed a low-protein diet

2017 ◽  
Vol 263 ◽  
pp. e6-e7 ◽  
Author(s):  
Marco Busnelli ◽  
Aurelia Bruneau ◽  
Stefano Manzini ◽  
Abdelhak Boukadiri ◽  
Catherine Philippe ◽  
...  
Keyword(s):  
Lipid Metabolism ◽  
Liver Steatosis ◽  
Protein Diet ◽  
Low Protein Diet ◽  
Intestinal Homeostasis ◽  
Low Protein

A low-protein diet eliminates the circadian rhythm of serum insulin and hepatic lipid metabolism in mice

2019 ◽  
Vol 63 ◽  
pp. 177-185
Author(s):  
Shin-Ichi Yokota ◽  
Kaai Nakamura ◽  
Midori Ando ◽  
Atsushi Haraguchi ◽  
Kanako Omori ◽  
...  
Keyword(s):  
Circadian Rhythm ◽  
Lipid Metabolism ◽  
Serum Insulin ◽  
Protein Diet ◽  
Low Protein Diet ◽  
Hepatic Lipid Metabolism ◽  
Hepatic Lipid ◽  
Low Protein

scholarly journals Alcoholic liver steatosis in mice is aggravated by low-protein diet and reversed by FXR agonist

2012 ◽  
Vol 6 (Suppl 3) ◽  
pp. P2
Author(s):  
Francislaine Lívero ◽  
Aline Stolf ◽  
Arturo Dreifuss ◽  
Amanda Bastos-Pereira ◽  
Raphaella Chicorski ◽  
...  
Keyword(s):  
Liver Steatosis ◽  
Protein Diet ◽  
Low Protein Diet ◽  
Low Protein

Hepatic Ultrastructure Changes Induced by Lithocholic Acid

1967 ◽  
Vol 25 ◽  
pp. 162-163 ◽  
Author(s):  
F. G. Zaki
Keyword(s):  
Endoplasmic Reticulum ◽  
Lithocholic Acid ◽  
Smooth Endoplasmic Reticulum ◽  
Protein Diet ◽  
Low Protein Diet ◽  
Electron Microscopic ◽  
Hydropic Degeneration ◽  
Hepatic Cells ◽  
Low Protein ◽  
Microscopic Studies

Addition of lithocholic acid (LCA), a naturally occurring bile acid in mammals, to a low protein diet fed to rats induced marked inflammatory reaction in the hepatic cells followed by hydropic degeneration and ductular cell proliferation. These changes were accompanied by dilatation and hyperplasia of the common bile duct and formation of “gallstones”. All these changes were reversible when LCA was withdrawn from the low protein diet except for the hardened gallstones which persisted.Electron microscopic studies revealed marked alterations in the hepatic cells. Early changes included disorganization, fragmentation of the rough endoplasmic reticulum and detachment of its ribosomes. Free ribosomes, either singly or arranged in small clusters were frequently seen in most of the hepatic cells. Vesiculation of the smooth endoplasmic reticulum was often encountered as early as one week after the administration of LCA (Fig. 1).

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