A study of the antiwear action mechanism of alkoxy aluminium in lubricating oil

1992 ◽  
Vol 4 (4) ◽  
pp. 255-262
Author(s):  
J. Dong ◽  
G. Chen ◽  
F. Luo
1996 ◽  
Vol 118 (3) ◽  
pp. 676-680 ◽  
Author(s):  
Ren-Gen Xiong ◽  
Hong Wang ◽  
Jing-Lin Zuo ◽  
Cai-Ming Liu ◽  
Xiao-Zeng You ◽  
...  

The copper(II) complex with alkylphosphonic acid monoalkyl ester (CuAMP) was synthesized, and its structure shown to be laminar on the basis of fast atom bombardment mass spectroscopy (FABMS). The extreme-pressure, antiwear and antiseizure performance of CuAMP was assessed using a four-ball machine. The results showed that CuAMP exhibits better extreme-pressure and antiwear properties than tricresyl phosphate. Based on the results of surface analysis, the antiwear action mechanism of CuAMP is discussed.


Wear ◽  
1997 ◽  
Vol 210 (1-2) ◽  
pp. 83-87 ◽  
Author(s):  
Yong Wan ◽  
Wenqin Yao ◽  
Xiaoyan Ye ◽  
Lili Cao ◽  
Guangqiu Shen ◽  
...  

2005 ◽  
Vol 48 (2) ◽  
pp. 171-175 ◽  
Author(s):  
Hu Jianqiang ◽  
Wei Xianyong ◽  
Yao Junbing ◽  
Xie Feng ◽  
Zhu Huanqin ◽  
...  

1979 ◽  
Vol 41 (03) ◽  
pp. 475-490 ◽  
Author(s):  
Chaoho Ouyang ◽  
Che-Ming Teng

SummaryThe minimal concentration of the platelet aggregation principle (Platelet Aggregoserpen- tin, PAS) necessary to induce platelet aggregation was 10 ng/ml, about one-hundredth of that of the crude venom. PAS induced the release of platelet factors 3 and 4 from platelets, but the released platelet factor 3 was easily inactivated by the anti-phospholipid effect of PAS. Pretreatment of platelets with neuraminidase potentiated PAS-induced platelet aggregation. PAS-induced platelet aggregation was independent on released ADP; it could occur in the ADP-removing systems, such as apyrase or a combination of phosphoenolpyruvate and pyruvate kinase. However, PAS-induced platelet aggregation could be inhibited by adenine nucleotides and adenosine.PAS-induced platelet aggregation was inhibited by some anti-inflammatory agents, antimalarial drugs, local anesthetics, antihistamine and smooth muscle relaxants. After deaggregation of PAS-treated platelets, thrombin and sodium arachidonate could further induce platelet aggregation, but ADP and second dose of PAS could not. It is concluded that PAS-induced platelet aggregation is due to prostaglandin synthesis. Recent literatures on the mechanism of platelet aggregation were surveyed and the actions of PAS were discussed.


Author(s):  
Claudio Santana ◽  
Jose Eduardo Mautone Barros ◽  
Juan Carlos Horta Gutiérrez ◽  
Helder Alves de Almeida Junior ◽  
jorgimara braga

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