scholarly journals Absence of focally enhanced gastritis in macaques with idiopathic colitis

2011 ◽  
Vol 17 (12) ◽  
pp. 2456-2461 ◽  
Author(s):  
Amnon Sonnenberg ◽  
Shelby D. Melton ◽  
Robert M. Genta ◽  
Anne D. Lewis
2021 ◽  
Vol 11 (2) ◽  
pp. 374-385
Author(s):  
Andrea Maia Pimentel ◽  
Luiz Antônio Rodrigues de Freitas ◽  
Rita de Cássia Reis Cruz ◽  
Isaac Neri de Novais Silva ◽  
Laíla Damasceno Andrade ◽  
...  

(1) The aim of the present study was to describe the endoscopic and histopathological findings in the esophagus, stomach, and duodenum in patients with Crohn’s disease. (2) Methods: This was a cross-sectional study that included patients receiving treatment from the inflammatory bowel disease outpatient clinic. Esophagogastroduodenoscopies with biopsies of the stomach and proximal duodenum were performed. Presence of Helicobacter pylori bacteria was assessed by Giemsa staining. (3) Results: We included 58 patients. Erosive esophagitis was identified in 25 patients (43.1%), gastritis was diagnosed in 32 patients (55.2%) and erosive duodenitis was found in eight (13.8%). The most frequent histopathological finding in the H. pylori-positive group was increased inflammatory activity in the gastric body and antrum, with a predominance of mononuclear and polymorphonuclear cells. In turn, the most frequent finding in the H. pylori-negative group was chronic inflammation with predominance of mononuclear cells. Focally enhanced gastritis was identified in four patients (6.9%), all of whom were negative for H. pylori. Granulomas were not observed. H. pylori infection was present in 19 patients (32.8%). (4) Conclusions: Nonspecific endoscopic and histological findings were frequent in patients with Crohn’s disease. Focally enhanced gastritis was uncommon and observed only in H. pylori-negative patients. The time from the diagnosis, patient age, and therapy in use may have influenced the nondetection of epithelioid granuloma.


2010 ◽  
Vol 71 (5) ◽  
pp. AB340
Author(s):  
Toshifumi Ashida ◽  
Fumika Orii ◽  
Atsuo Maemoto ◽  
Yasunobu Gohira ◽  
Hirohito Kawauchi ◽  
...  

2013 ◽  
Vol 37 (2) ◽  
pp. 295-299 ◽  
Author(s):  
Jonathan B. McHugh ◽  
Purva Gopal ◽  
Joel K. Greenson

2001 ◽  
Vol 33 ◽  
pp. A112
Author(s):  
P.E. Lxis ◽  
B. Gennank ◽  
C. Doglioni ◽  
E. Galliani ◽  
F. Costan

2013 ◽  
Vol 108 ◽  
pp. S431
Author(s):  
Hester Choi ◽  
Arvind Mathur ◽  
Heather Rojas ◽  
Christian Jackson

2002 ◽  
Vol 97 (6) ◽  
pp. 1415-1420 ◽  
Author(s):  
F. Sharif ◽  
M. McDermott ◽  
M. Dillon ◽  
B. Drumm ◽  
M. Rowland ◽  
...  

2009 ◽  
Vol 23 (2) ◽  
pp. 95-98 ◽  
Author(s):  
Polymnia Galiatsatos ◽  
Adrian Gologan ◽  
Esther Lamoureux

Autism spectrum disorder refers to syndromes of varying severity, typified by impaired social interactions, communicative delays and restricted, repetitive behaviours and interests. The prevalence of autism spectrum disorders has been on the rise, while the etiology remains unclear and most likely multifactorial. There have been several reports of a link between autism and chronic gastrointestinal symptoms. Endoscopy trials have demonstrated a higher prevalence of nonspecific colitis, lymphoid hyperplasia and focally enhanced gastritis compared with controls. Postulated mechanisms include aberrant immune responses to some dietary proteins, abnormal intestinal permeability and unfavourable gut microflora. Two autism spectrum disorder patients with chronic intestinal symptoms and abnormal endoscopic findings are described, followed by a review of this controversial topic.


2000 ◽  
Vol 118 (4) ◽  
pp. A1343
Author(s):  
Bastianello Germana' ◽  
Claudio Doglioni ◽  
Ermenegildo Galliani ◽  
Franco Costan

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