Prostaglandin E2 inhibits migration of colonic lamina propria fibroblasts

2010 ◽  
Vol 16 (9) ◽  
pp. 1505-1513 ◽  
Author(s):  
Florian Rieder ◽  
Martina Georgieva ◽  
Anja Schirbel ◽  
Monika Artinger ◽  
Anita Zügner ◽  
...  
Keyword(s):  
Prostaglandin E2 ◽  
Lamina Propria ◽  
Colonic Lamina Propria Fibroblasts

M1821 Prostaglandin E2 Inhibits Migration of Colonic Lamina Propria Fibroblasts

2010 ◽  
Vol 138 (5) ◽  
pp. S-426
Author(s):  
Florian Rieder ◽  
Martina Georgieva ◽  
Anja Schirbel ◽  
Monika Artinger ◽  
Anita Zuegner ◽  
...  
Keyword(s):  
Prostaglandin E2 ◽  
Lamina Propria ◽  
Colonic Lamina Propria Fibroblasts

Modulation of the Migration of Colonic Lamina Propria Fibroblasts by Elevation of Intracellular Cyclic AMP

2006 ◽  
Vol 12 ◽  
pp. S23
Author(s):  
F. Rieder ◽  
J. Brenmoehl ◽  
M. Artinger ◽  
A. Zügner ◽  
M. Georgieva ◽  
...  
Keyword(s):  
Cyclic Amp ◽  
Lamina Propria ◽  
Colonic Lamina Propria Fibroblasts

scholarly journals Inflammation modulates fibronectin isoform expression in colonic lamina propria fibroblasts (CLPF)

2008 ◽  
Vol 23 (10) ◽  
pp. 947-955 ◽  
Author(s):  
Julia Brenmoehl ◽  
Werner Falk ◽  
Michael Göke ◽  
Jürgen Schölmerich ◽  
Gerhard Rogler
Keyword(s):  
Lamina Propria ◽  
Isoform Expression ◽  
Colonic Lamina Propria Fibroblasts

Scanning Electron Microscopy of Human Jejunum in Whipple's Disease

1977 ◽  
Vol 35 ◽  
pp. 354-355
Author(s):  
John H. L. Watson ◽  
C. N. Sun
Keyword(s):  
Electron Microscopy ◽  
Scanning Electron Microscopy ◽  
Basement Membrane ◽  
Lamina Propria ◽  
Brush Border ◽  
Whipple’S Disease ◽  
Whipple's Disease ◽  
Biopsy Specimens ◽  
Brush Borders ◽  
Scanning Electron

That the etiology of Whipple's disease could be bacterial was first suggested from electron micrographs in 1960. Evidence for binary fission of the bacteria, their phagocytosis by histiocytes in the lamina propria, their occurrence between and within the cells of the epithelium and on the brush border of the lumen were reported later. Scanning electron microscopy has been applied by us in an attempt to confirm the earlier observations by the new technique and to describe the bacterium further. Both transmission and scanning electron microscopy have been used concurrently to study the same biopsy specimens, and transmission observations have been used to confirm those made by scanning.The locations of the brush borders, the columnar epithelial cells, the basement membrane and the lamina propria beneath it were each easily identified by scanning electron microscopy. The lamina propria was completely filled with the wiener-shaped bacteria, Fig. 1.

The protease phenotype and crystalline nature of the granules of intraepithelial mast cells in Trichinella spiralis-infected mice

1995 ◽  
Vol 53 ◽  
pp. 818-819
Author(s):  
D.S. Friend ◽  
N. Ghildyal ◽  
M.F. Gurish ◽  
K.F. Austen ◽  
R.L. Stevens
Keyword(s):  
Small Intestine ◽  
Mast Cells ◽  
Epithelial Cells ◽  
Lamina Propria ◽  
Trichinella Spiralis ◽  
Intestinal Epithelial ◽  
Carboxypeptidase A ◽  
C3h Mice ◽  
Granule Morphology ◽  
Crystalline Nature

Trichinella spiralis induces a profound mastocytosis and eosinophilia in the small intestine of the infected mouse. Mouse mast cells (MC) store in their granules various combinations of at least five chymotryptic chymases [designated mouse MC protease (mMCP) 1 to 5], two tryptic proteases designated mMCP-6 and mMCP-7 and an exopeptidase, carboxypeptidase A (mMC-CPA). Using antipeptide, protease -specific antibodies to these MC granule proteases, immunohistochemistry was done to determine the distribution, number and protease phenotype of the MCs in the small intestine and spleen 10 to >60 days after Trichinella infection of BALB/c and C3H mice. TEM was performed to evaluate the granule morphology of the MCs between intestinal epithelial cells and in the lamina propria (mucosal MCs) and in the submucosa, muscle and serosa of the intestine (submucosal MCs).As noted in the table below, the number of submucosal MCs remained constant throughout the study. In contrast, on day 14, the number of MCs in the mucosa increased ~25 fold. Increased numbers of MCs were observed between epithelial cells in the mucosal crypts, in the lamina propria and to a lesser extent, between epithelial cells of the intestinal villi.

Ultrastructure Research of the Gastritis from Returning Bile

1990 ◽  
Vol 48 (3) ◽  
pp. 202-203
Author(s):  
Shaopeng Hu ◽  
Jianhua Wang ◽  
Zhen Li ◽  
Huei Chen ◽  
Fei Cu ◽  
...  
Keyword(s):  
Electron Microscope ◽  
Gastric Mucosa ◽  
Scanning Electron Microscope ◽  
Lamina Propria ◽  
Transmission Electron Microscope ◽  
Ultrastructural Change ◽  
Common Disease ◽  
Epithelial Surface ◽  
Transmission Electron ◽  
Scanning Electron

Gastritis from returning bile is a common disease, but the reason for the disease is not clear. As the pathologic ultrastructure research progresses, it has drawn attention to the ultrastructural change of cells in gastric mucosa by clinical workers. We observed gastric mucosa tissues of 15 patients suffering from gastritis with a transmission electron microscope (TEM) and a scanning electron microscope (SEM). It is the first report in China that fungus exists in the lamina propria of gastric mucosa tissue. The result is as follows.The gastric mucosa tissues of 15 patients suffering from gastritis were acquired by stomachoscopy. Both TEM and SEM specimens were prepared by the usual methods. Under the TEM, the epithelial surface became higher and larger. Mitochondria of the cells were swollen and cristae were disrupted. There were vacuoles in the cells. The nucleus showed disorder, heterochromatin became darker, and nucleolae could be observed.

Activation of isolated heterophils from chicks stimulated in vivo with salmonella enteritidis-immune lymphokines

1996 ◽  
Vol 54 ◽  
pp. 760-761
Author(s):  
R. B. Moyes ◽  
R. E. Droleskey ◽  
M. H. Kogut ◽  
J. R. DeLoach
Keyword(s):  
Lamina Propria ◽  
Intestinal Mucosa ◽  
Salmonella Enteritidis ◽  
Intestinal Tract ◽  
Polymorphonuclear Cells ◽  
Subclinical Infection ◽  
Egg Products ◽  
Immune Lymphokines ◽  
Organ Invasion

Salmonella enteritidis (SE) is of great concern to the poultry industry due to the organism's ability to penetrate the intestinal mucosa of the laying hen and subsequently colonize the ovaries and yolk membrane. The resultant subclinical infection can lead to SE infection of raw eggs and egg products. Interference with the ability of the organism to invade has been linked to the activation and recruitment of inflammatory polymorphonuclear cells, heterophils, to the lamina propria of the intestinal tract.Recently it has been established that heterophil activation and increased resistance to SE organ invasion can be accomplished by the administration of SE-immune lymphokines (SE-ILK) obtained from supernatants of concanavalin-A stimulated SE immune T lymphocytes from SE hyperimmunized hens. Invasion of SE into the lamina propria provides a secondary signal for directing activated heterophils to the site of SE invasion.

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