scholarly journals Optimization of tractography of the optic radiations

2012 ◽  
Vol 35 (2) ◽  
pp. 683-697 ◽  
Author(s):  
Christopher F.A. Benjamin ◽  
Jolene M. Singh ◽  
Sanjay P. Prabhu ◽  
Simon K. Warfield
Keyword(s):  
Optic Radiations

Magnetic resonance diffusion tensor imaging (MRDTI) of the optic nerve and optic radiations at 3T in children with neurofibromatosis type I (NF-1)

2011 ◽  
Vol 42 (2) ◽  
pp. 168-174 ◽  
Author(s):  
Christopher G. Filippi ◽  
Aaron Bos ◽  
Joshua P. Nickerson ◽  
Michael B. Salmela ◽  
Chris J. Koski ◽  
...  
Keyword(s):  
Diffusion Tensor Imaging ◽  
Optic Nerve ◽  
Magnetic Resonance ◽  
Diffusion Tensor ◽  
Neurofibromatosis Type ◽  
Type I ◽  
Neurofibromatosis Type I ◽  
Optic Radiations

scholarly journals Diffusion tensor imaging of the optic radiations after optic neuritis

2011 ◽  
Vol 33 (9) ◽  
pp. 2047-2061 ◽  
Author(s):  
Scott Kolbe ◽  
Clare Bajraszewski ◽  
Caron Chapman ◽  
Tan Nguyen ◽  
Peter Mitchell ◽  
...  
Keyword(s):  
Diffusion Tensor Imaging ◽  
Optic Neuritis ◽  
Diffusion Tensor ◽  
Optic Radiations

Distribution of visual callosal projection neurons in hamsters subjected to transection of the optic radiations on the day of birth

1987 ◽  
Vol 32 (2) ◽  
pp. 217-232 ◽  
Author(s):  
Robert W. Rhoades ◽  
Stephen E. Fish ◽  
Richard D. Mooney ◽  
Nicolas L. Chiaia
Keyword(s):  
Projection Neurons ◽  
Callosal Projection ◽  
Optic Radiations

Studies on the Optic Radiations

1962 ◽  
Vol 19 (6) ◽  
pp. 522-528 ◽  
Author(s):  
Lyle A. French
Keyword(s):  
Optic Radiations

Validation of an automated tractography method for the optic radiations as a biomarker of visual acuity in neurofibromatosis-associated optic pathway glioma

2018 ◽  
Vol 299 ◽  
pp. 308-316 ◽  
Author(s):  
Peter de Blank ◽  
Michael J. Fisher ◽  
Haley Gittleman ◽  
Jill S. Barnholtz-Sloan ◽  
Chaitra Badve ◽  
...  
Keyword(s):  
Visual Acuity ◽  
Optic Pathway Glioma ◽  
Optic Pathway ◽  
Optic Radiations

Acute exacerbation of multiple sclerosis presenting with facial metamorphopsia and palinopsia

2012 ◽  
Vol 19 (3) ◽  
pp. 369-371 ◽  
Author(s):  
Deepti Anbarasan ◽  
Jonathan Howard
Keyword(s):  
Multiple Sclerosis ◽  
Visual Pathway ◽  
Brain Lesions ◽  
Ophthalmological Examination ◽  
Occipital Region ◽  
Relapsing Remitting ◽  
History Of ◽  
Association Area ◽  
Relapsing Remitting Multiple Sclerosis ◽  
Optic Radiations

We discuss the case of a patient with a known history of relapsing–remitting multiple sclerosis (MS) who presented with the isolated complaint of altered visual perception in the absence of abnormalities on ophthalmological examination. To the best of the authors’ knowledge, this is the first documented case of both facial metamorphopsia and palinopsia occurring as the symptoms of demyelinating brain lesions consistent with an acute MS exacerbation. These symptoms appear to be related to active demyelination that either involved the optic radiations in the visual pathway or the visual association area in the temporo-occipital region of the left hemisphere.

scholarly journals The Influence of “Blind” Distractors on Eye Movement Trajectories in Visual Hemifield Defects

2008 ◽  
Vol 20 (11) ◽  
pp. 2025-2036 ◽  
Author(s):  
Stefan Van der Stigchel ◽  
Wieske van Zoest ◽  
Jan Theeuwes ◽  
Jason J. S. Barton
Keyword(s):  
Eye Movements ◽  
Visual Information ◽  
Striate Cortex ◽  
Optic Chiasm ◽  
Cerebral Lesions ◽  
Movement Trajectories ◽  
Visual Hemifield ◽  
Irrelevant Distractors ◽  
Retinotectal Projections ◽  
Optic Radiations

There is evidence that some visual information in blind regions may still be processed in patients with hemifield defects after cerebral lesions (“blindsight”). We tested the hypothesis that, in the absence of retinogeniculostriate processing, residual retinotectal processing may still be detected as modifications of saccades to seen targets by irrelevant distractors in the blind hemifield. Six patients were presented with distractors in the blind and intact portions of their visual field and participants were instructed to make eye movements to targets in the intact field. Eye movements were recorded to determine if blind-field distractors caused deviation in saccadic trajectories. No deviation was found in one patient with an optic chiasm lesion, which affect both retinotectal and retinogeniculostriate pathways. In five patients with lesions of the optic radiations or the striate cortex, the results were mixed, with two of the five patients showing significant deviations of saccadic trajectory away from the “blind” distractor. In a second experiment, two of the five patients were tested with the target and the distractor more closely aligned. Both patients showed a “global effect,” in that saccades deviated toward the distractor, but the effect was stronger in the patient who also showed significant trajectory deviation in the first experiment. Although our study confirms that distractor effects on saccadic trajectory can occur in patients with damage to the retinogeniculostriate visual pathway but preserved retinotectal projections, there remain questions regarding what additional factors are required for these effects to manifest themselves in a given patient.

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