scholarly journals Potential of rosmarinic acid to ameliorate toxic effects of diethyl methoxy thio‑phosphoryl thio‑succinate on albino wistar rats’ lung, mast cell infiltration inhibitory pathway

2021 ◽  
Author(s):  
Ahmed S. Ahmed ◽  
Marwa M. Mona ◽  
Mona A. Abdel‑Kareem ◽  
Rasha A. Elsisy
Keyword(s):  
Mast Cell ◽  
Rosmarinic Acid ◽  
Wistar Rats ◽  
Toxic Effects ◽  
Cell Infiltration ◽  
Inhibitory Pathway ◽  
Lung Mast Cell

Prevention of toxic effects of orally administered tartrazine by crocin in Wistar rats

2021 ◽  
pp. 1-15
Author(s):  
Eyup Altinoz ◽  
Mehmet Erman Erdemli ◽  
Mehmet Gül ◽  
Zeynep Erdemli ◽  
Semir Gül ◽  
...  
Keyword(s):  
Wistar Rats ◽  
Toxic Effects

scholarly journals Toxic effects of Cr(VI) in liver after administration in the drinking water to Wistar rats

2006 ◽  
Vol 164 ◽  
pp. S202
Author(s):  
A. Rafael ◽  
A. Almeida ◽  
I. Parreira ◽  
P. Santos ◽  
M. Carvalho ◽  
...  
Keyword(s):  
Drinking Water ◽  
Wistar Rats ◽  
Toxic Effects

GENERALIZED MAST CELL DISEASE AND URTICARIA PIGMENTOSA

PEDIATRICS ◽  
1957 ◽  
Vol 19 (6) ◽  
pp. 1023-1032
Author(s):  
Thomas L. Rider ◽  
Arthur A. Stein ◽  
John W. Abbuhl
Keyword(s):  
Mast Cell ◽  
Fibrous Tissue ◽  
Cell Activity ◽  
Gastrointestinal Symptoms ◽  
Urticaria Pigmentosa ◽  
Cell Infiltration ◽  
Cell Disease ◽  
Tissue Proliferation ◽  
Bone Changes ◽  
Mast Cell Disease

The case which is presented and review of the literature indicate that urticaria pigmentosa may be accompanied by mast cell infiltration of many tissues and viscera. No definite conclusions may be drawn regarding etiology, incidence, or prognosis of this disorder. The evidence indicates that both local and generalized symptoms occur which are principally related to the pathophysiologic changes resulting from mast cell activity, i.e., fibrous tissue proliferation, hyperemia and edema. In the case reported herein there was no histologic evidence of fibrous tissue increase but it is postulated that the hepatosplenomegaly and the bone changes in roentgenograms may be in part due to such changes. The dermatographism, skin flushing, salivary gland swelling and gastrointestinal symptoms are probably due to the physiologic action of mast cell products, i.e., histamine and serotonin. The diagnosis of generalized mast cell disease can be made in a patient who presents a chronic maculopapular skin rash, dermatographism, hepatosplenomegaly and mast cell infiltration of the bone marrow. Demonstration of mast cell infiltration in the skin and other tissues is confirmatory but not necessary.

Lung mast cell density and distribution in chronically hypoxic animals

1977 ◽  
Vol 42 (2) ◽  
pp. 174-178 ◽  
Author(s):  
A. Tucker ◽  
I. F. McMurtry ◽  
A. F. Alexander ◽  
J. T. Reeves ◽  
R. F. Grover
Keyword(s):  
Cell Proliferation ◽  
Pulmonary Hypertension ◽  
Mast Cell ◽  
Mast Cells ◽  
Cell Density ◽  
Mammalian Species ◽  
Cell Hyperplasia ◽  
Mast Cell Density ◽  
Lung Mast Cell ◽  
Mast Cell Hyperplasia

Changes in the density and distribution of pulmonary mast cells were determined in six mammalian species exposed to hypobaric hypoxia (PB = 435 Torr) for 19–48 days. Control animals were studied at 1,600 m (PB = 635 Torr). Total lung mast cell hyperplasia was observed only in calves exposed to high altitude. Pigs, rats, and sheep exhibited small, but insignificant, increases in mast cell density. Perivascular mast cell proliferation adjacent to vessels of 30–500 mum in diameter was seen in both calves and pigs. Bronchial, alveolar septal, and systemic tissue (tongue) mast cell hyperplasia was not observed in any of the species. Three indices of pulmonary hypertension (right ventricular hypertrophy, medial thickness of pulmonary arteries, and pulmonary arterial pressure) correlated with perivascular mast cell density. The findings indicate that perivascular mast cell proliferation may relate more to the morphological pulmonary vascular changes and to pulmonary hypertension than to hypoxia, leading to the speculation that mast cells increase in number in response to the hypertension, rather than to mediate and maintain the hypertension.

scholarly journals Gastric corticotropin-releasing factor influences mast cell infiltration in a rat model of functional dyspepsia

PLoS ONE ◽  
2018 ◽  
Vol 13 (9) ◽  
pp. e0203704 ◽  
Author(s):  
Shin-ichiro Hagiwara ◽  
Esha Kaushal ◽  
Sreenivasan Paruthiyil ◽  
Pankaj J. Pasricha ◽  
Burcu Hasdemir ◽  
...  
Keyword(s):  
Mast Cell ◽  
Functional Dyspepsia ◽  
Rat Model ◽  
Corticotropin Releasing Factor ◽  
Cell Infiltration

scholarly journals The human lung mast cell.

1984 ◽  
Vol 55 ◽  
pp. 259-269 ◽  
Author(s):  
S I Wasserman
Keyword(s):  
Mast Cell ◽  
Human Lung ◽  
Lung Mast Cell
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