The relative viability of male and female mouse embryos

1926 ◽  
Vol 37 (1) ◽  
pp. 127-140 ◽  
Author(s):  
E. C. Macdowell ◽  
E. M. Lord
2005 ◽  
Vol 38 (2) ◽  
pp. 245-256 ◽  
Author(s):  
L WILLEMS ◽  
A ZATTA ◽  
K HOLMGREN ◽  
K ASHTON ◽  
J HEADRICK

Author(s):  
Alexandra L. Cara ◽  
Emily L. Henson ◽  
Bethany G. Beekly ◽  
Carol F. Elias

2013 ◽  
Vol 190 (4S) ◽  
pp. 1610-1617 ◽  
Author(s):  
Bruce J. Schlomer ◽  
Max Feretti ◽  
Esequiel Rodriguez ◽  
Sarah Blaschko ◽  
Gerald Cunha ◽  
...  

2006 ◽  
Vol 16 (2) ◽  
pp. 166-172 ◽  
Author(s):  
Shin Kobayashi ◽  
Ayako Isotani ◽  
Nathan Mise ◽  
Masamichi Yamamoto ◽  
Yoshitaka Fujihara ◽  
...  

1993 ◽  
Vol 235 (2) ◽  
pp. 303-311 ◽  
Author(s):  
Yang Chai ◽  
Donna K. Klauser ◽  
Patricia A. Denny ◽  
Paul C. Denny

1995 ◽  
Vol 65 (3) ◽  
pp. 223-227 ◽  
Author(s):  
Jeanne M. Lebon ◽  
Patrick P. L. Tam ◽  
Judith Singer-Sam ◽  
Arthur D. Riggs ◽  
Seong-Seng Tan

SummaryX chromosome inactivation (XCI) has been assumed to be complete in all cells of female mouse embryos at about 6 d post coitum (dpc). However, a recent study on β-galactosidase expression of an X-linkedlacZtransgene suggests that XCI is probably not complete several days after this time in some lineages. To help resolve this issue, we analysed XCI in embryos which carry the T(X;16)16H (Searle's) translocation and are heterozygous at the X-linkedHprtandPgk-1genes. The quantitative RT-PCR single nucleotide primer extension (SNuPE) assay was used to measureHprtandPgk-1allele-specific transcripts in embryos 9·5 dpc. No transcripts from the normal X chromosome were found in any of the tissues tested, indicating that inactivation was complete for these endogenous genes.


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