scholarly journals Induction of Heme oxygenase 1 protects hepatocytes from Isoniazid – Rifampicin induced cell death: an in vitro study

2016 ◽  
Vol 37 (1) ◽  
pp. 27-36 ◽  
Author(s):  
Debasree Bishnu
Keyword(s):  
Cell Death ◽  
Heme Oxygenase ◽  
In Vitro Study ◽  
Vitro Study ◽  
Heme Oxygenase 1

scholarly journals Iron Contents in Rice Food Derived from the Iron Pot, and In Vitro Study Regarding Heme Oxygenase-1 Activity

2016 ◽  
Vol 23 (2) ◽  
pp. 77-82
Author(s):  
Keun Young Kim ◽  
Young Min An ◽  
Tae Wan Kim ◽  
Yong Hun Jun ◽  
Ji Eun Lee ◽  
...  
Keyword(s):  
Heme Oxygenase ◽  
In Vitro Study ◽  
Vitro Study ◽  
Heme Oxygenase 1

scholarly journals An in vitro study of the cytotoxicity of TTF·TCNQ nanoparticles to mammalian cells

2020 ◽  
Vol 1 (6) ◽  
pp. 1963-1970
Author(s):  
Hongjie Chen ◽  
Géraldine Albérola ◽  
Dominique de Caro ◽  
Christophe Faulmann ◽  
Muriel Golzio ◽  
...  
Keyword(s):  
Cell Death ◽  
Mammalian Cells ◽  
In Vitro Study ◽  
Vitro Study ◽  
Biomedical Devices ◽  
Induce Cell Death

Soluble functionalized TTF·TCNQ nanoparticles do not induce cell death at concentrations up to 50 μg mL−1, a promising feature for biomedical devices.

scholarly journals Carbon Monoxide Protects against Liver Failure through Nitric Oxide–induced Heme Oxygenase 1

2003 ◽  
Vol 198 (11) ◽  
pp. 1707-1716 ◽  
Author(s):  
Brian S. Zuckerbraun ◽  
Timothy R. Billiar ◽  
Sherrie L. Otterbein ◽  
Peter K.M. Kim ◽  
Fang Liu ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Carbon Monoxide ◽  
Cell Death ◽  
Protective Effect ◽  
Heme Oxygenase ◽  
Heme Oxygenase 1 ◽  
No Production ◽  
Protective Effects ◽  
In Vivo Models

Carbon monoxide (CO) and nitric oxide (NO) each have mechanistically unique roles in various inflammatory disorders. Although it is known that CO can induce production of NO and that NO can induce expression of the cytoprotective enzyme heme oxygenase 1 (HO-1), there is no information whether the protective effect of CO ever requires NO production or whether either gas must induce expression of HO-1 to exert its functional effects. Using in vitro and in vivo models of tumor necrosis factor α–induced hepatocyte cell death in mice, we find that activation of nuclear factor κB and increased expression of inducible NO are required for the protective effects of CO, whereas the protective effects of NO require up-regulation of HO-1 expression. When protection from cell death is initiated by CO, NO production and HO-1 activity are each required for the protective effect showing for the first time an essential synergy between these two molecules in tandem providing potent cytoprotection.

scholarly journals SP044ROLE OF CELL DEATH IN NEPHROCALCINOSIS: AN IN VITRO STUDY OF HK-2 CELLS

2018 ◽  
Vol 33 (suppl_1) ◽  
pp. i361-i361
Author(s):  
Giovanna Priante ◽  
Lisa Gianesello ◽  
Monica Ceol ◽  
Claudio Furlan ◽  
Dorella Del Prete ◽  
...  
Keyword(s):  
Cell Death ◽  
In Vitro Study ◽  
Vitro Study

scholarly journals Photodynamic Therapy in Pythium insidiosum – An In Vitro Study of the Correlation of Sensitizer Localization and Cell Death

PLoS ONE ◽  
2014 ◽  
Vol 9 (1) ◽  
pp. e85431 ◽  
Author(s):  
Layla Pires ◽  
Sandra de Moraes Gimenes Bosco ◽  
Maurício S. Baptista ◽  
Cristina Kurachi
Keyword(s):  
Cell Death ◽  
Photodynamic Therapy ◽  
In Vitro Study ◽  
Vitro Study ◽  
Pythium Insidiosum

scholarly journals 18-kDa Translocator Protein Ligands Protect H9C2 Cardiomyocytes from Cigarette Smoke-induced Cell Death: In Vitro Study

In Vivo ◽  
2020 ◽  
Vol 34 (2) ◽  
pp. 549-556
Author(s):  
RAFAEL NAGLER ◽  
NIDAL ZEINEH ◽  
MAYA AZRAD ◽  
NASRA YASSIN ◽  
ABRAHAM WEIZMAN ◽  
...  
Keyword(s):  
Cell Death ◽  
Cigarette Smoke ◽  
In Vitro Study ◽  
Vitro Study ◽  
Translocator Protein ◽  
Protein Ligands ◽  
H9c2 Cardiomyocytes
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