Routine Screening for Callosal Dysgenesis in the Second Trimester Is Achievable With Intensive Training

2016 ◽  
Vol 35 (4) ◽  
pp. 717-722 ◽  
Author(s):  
Ilona Lavender ◽  
Peter R. Coombs ◽  
Keith Van Haltren ◽  
Alice J. Robinson
Keyword(s):  
Routine Screening ◽  
Second Trimester ◽  
Intensive Training ◽  
Callosal Dysgenesis

Breast Ca Deaths Higher Without Routine Screening

2009 ◽  
Vol 42 (19) ◽  
pp. 1-5
Author(s):  
SHERRY BOSCHERT
Keyword(s):  
Routine Screening

Abstract #809: Spontaneous Pituitary Apoplexy in the Second Trimester Associated with Sensory Loss

2015 ◽  
Vol 21 ◽  
pp. 152
Author(s):  
Rtika Abraham ◽  
Rachel Pollitzer ◽  
Murat Gokden ◽  
Peter Goulden
Keyword(s):  
Pituitary Apoplexy ◽  
Sensory Loss ◽  
Second Trimester

Supplementation with beta-hydroxy-beta-methylbutyrate impacts glucose homeostasis and increases liver size in trained mice

2020 ◽  
Vol 90 (1-2) ◽  
pp. 113-123
Author(s):  
Ines Schadock ◽  
Barbara G. Freitas ◽  
Irae L. Moreira ◽  
Joao A. Rincon ◽  
Marcio Nunes Correa ◽  
...  
Keyword(s):  
Strength Training ◽  
Muscle Mass ◽  
Glucose Homeostasis ◽  
Fasting Blood Glucose ◽  
Hepatic Metabolism ◽  
Mass Gain ◽  
Trained Group ◽  
Tissue Mass ◽  
Liver Size ◽  
Intensive Training

Abstract. β-hydroxy-β-methyl butyrate (HMB) is a bioactive metabolite derived from the amino acid leucine, usually applied for muscle mass increase during physical training, as well as for muscle mass maintenance in debilitating chronic diseases. The hypothesis of the present study is that HMB is a safe supplement for muscle mass gain by strength training. Based on this, the objective was to measure changes in body composition, glucose homeostasis and hepatic metabolism of HMB supplemented mice during strength training. Two of four groups of male mice (n = 6/group) underwent an 8-week training period session (climbing stairs) with or without HMB supplementation (190 mg/kgBW per day). We observed lower body mass gain (4.9 ± 0.43% versus 1.2 ± 0.43, p < 0.001) and increased liver mass (40.9 ± 0.9 mg/gBW versus 44.8 ± 1.3, p < 0.001) in the supplemented trained group compared with the non-supplemented groups. The supplemented trained group had an increase in relative adipose tissue mass (12.4 ± 0.63 mg/gBW versus 16.1 ± 0.88, P < 0.01) compared to the non-supplemented untrained group, and an increase in fasting blood glucose (111 ± 4.58 mg/dL versus 122 ± 3.70, P < 0.05) and insulin resistance (3.79 ± 0.19 % glucose decay/min versus 2.45 ± 0.28, P < 0.05) comparing with non-supplemented trained group. Adaptive heart hypertrophy was observed only in the non-supplemented trained group (4.82 ± 0.05 mg/gBW versus 5.12 ± 0.13, P < 0.05). There was a higher hepatic insulin-like growth factor-1 expression (P = 0.002) in supplemented untrained comparing with non-supplemented untrained group. Gene expression of gluconeogenesis regulatory factors was increased by training and reduced by HMB supplementation. These results confirm that HMB supplementation associated with intensive training protocol drives changes in glucose homeostasis and liver metabolism in mice.

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