scholarly journals Cerebral Venous Infarct After Recovery From COVID-19 Pneumonia

Cureus ◽  
2021 ◽  
Author(s):  
Mohanned F Alfahhad ◽  
Saeed S Alghamdi ◽  
Osama A Alzahrani ◽  
Saleh K Aldakhil ◽  
Abdulaziz A Algarni ◽  
...  
Keyword(s):  
Venous Infarct

scholarly journals Cryptogenic Isolated Cortical Venous Infarct: Three Case Reports

2010 ◽  
Author(s):  
Ertugrul Uzar ◽  
Atilla Ilhan ◽  
Banu Cakir ◽  
Alevtina Ersoy ◽  
Ulkuhan Duzgun ◽  
...  
Keyword(s):  
Case Reports ◽  
Venous Infarct

scholarly journals Correction to: A case of verbal and emotional prosody processing dissociation after a right temporal venous infarct

2020 ◽  
Vol 41 (3) ◽  
pp. 737-737
Author(s):  
Julie Bourgeois–Vionnet ◽  
Annie Moulin ◽  
Marc Hermier ◽  
Agathe Pralus ◽  
Barbara Tillmann ◽  
...  
Keyword(s):  
Emotional Prosody ◽  
Venous Infarct

scholarly journals Prevalence of subarachnoid haemorrhage among patients with cranial venous sinus thrombosis in the presence and absence of venous infarcts

2018 ◽  
Vol 31 (5) ◽  
pp. 496-503 ◽  
Author(s):  
Muhammad Azeemuddin ◽  
Muhammad Awais ◽  
Fatima Mubarak ◽  
Abdul Rehman ◽  
Noor Ul-Ain Baloch
Keyword(s):  
Subarachnoid Haemorrhage ◽  
Sinus Thrombosis ◽  
Venous Sinus ◽  
P Value ◽  
Chi Square ◽  
Venous Sinus Thrombosis ◽  
Venous Infarct ◽  
Fluid Attenuated Inversion Recovery ◽  
Chi Square Test ◽  
Venous Infarcts

Introduction In patients with cranial venous sinus thrombosis, the occurrence of subarachnoid haemorrhage in association with haemorrhagic venous infarcts is a well described phenomenon. However, the presence of subarachnoid haemorrhage in patients with cranial venous sinus thrombosis in the absence of a haemorrhagic venous infarct is exceedingly rare. Methods We retrospectively reviewed charts and scans of all patients who had cranial venous sinus thrombosis confirmed by magnetic resonance venography at our hospital between September 2004 and May 2015. The presence of subarachnoid haemorrhage was ascertained on fluid-attenuated inversion recovery, susceptibility-weighted imaging and/or unenhanced computed tomography scans by a single experienced neuroradiologist. Statistical analysis was performed using the Statistical Package for Social Sciences version 20. Differences in the proportion of haemorrhagic venous infarcts among patients with subarachnoid haemorrhage versus those without subarachnoid haemorrhage were compared using the chi-square test. A P value of less than 0.05 was considered significant. Results A total of 138 patients who had cranial venous sinus thrombosis were included in the study. Seventy-three (52.9%) were women and the median age of subjects was 35 (interquartile range 22–47) years. Venous infarcts and haemorrhagic venous infarcts were noted in 20/138 (14.5%) and 62/138 (44.9%) cases, respectively. Subarachnoid haemorrhage was present in 15/138 (10.9%) cases and, in three cases, subarachnoid haemorrhage occurred in the absence of a venous infarct. Haemorrhagic venous infarcts were more prevalent ( P = 0.021) among patients with subarachnoid haemorrhage (11/15) than in those without subarachnoid haemorrhage (51/123). Conclusion In patients with cranial venous sinus thrombosis, subarachnoid haemorrhage can occur even in the absence of a haemorrhagic venous infarct. The recognition of cranial venous sinus thrombosis as the underlying cause of subarachnoid haemorrhage is important to avoid misdiagnosis and inappropriate management.

Effect of age on cerebral venous circulation disturbances in the rat

2000 ◽  
Vol 93 (2) ◽  
pp. 298-304 ◽  
Author(s):  
Hiroyuki Otsuka ◽  
Hiroyuki Nakase ◽  
Kiyoshi Nagata ◽  
Katsuhiro Ueda ◽  
Oliver Kempski ◽  
...  
Keyword(s):  
Venous Occlusion ◽  
Aged Rats ◽  
Venous Flow ◽  
Content Type ◽  
Young Rats ◽  
Venous Circulation ◽  
Age Related ◽  
Venous Infarct ◽  
Group A ◽  
Fine Print

Object. Mild cerebral venous circulation disturbances (CVCDs) in aged patients are frequently known to cause unexpectedly severe postoperative complications in neurosurgical practice. The object of the present study was to determine whether there are age-related differences involved in vulnerability to CVCDs.Methods. Thirty-eight male Wistar rats were used. A single cortical vein with a 100-µm diameter was occluded photochemically by using rose bengal dye and fiberoptic illumination in young (Group Y, 19 animals aged 10–14 weeks) and aged (Group A, seven animals aged 80–100 weeks) rats. Five young and seven aged animals served as sham-operated controls. Regional cerebral blood flow (rCBF) was determined from local CBF, which was measured at 25 (5 × 5) identical locations, with the occluded vein located central to the scanning field, by using a laser Doppler scanning technique every 15 minutes for 90 minutes after venous occlusion. The cerebral venous flow pattern was examined using fluorescence angiography until 90 minutes after occlusion. Histological specimens were examined 24 hours after occlusion. In Group Y, rCBF did not change significantly after venous occlusion. However, in Group A, rCBF decreased rapidly beginning 15 minutes after occlusion. Significant intergroup differences were observed 30, 60, and 90 minutes after occlusion. Venous flow arrest, which resulted in venous infarct, was observed on angiography 90 minutes after occlusion in two (10.5%) of 19 young and six (85.7%) of seven aged rats. The venous thrombus in Group A rats was significantly larger than that in Group Y rats 90 minutes after occlusion. Venous infarction was seen in all aged rats (100%) and in six young rats (31.6%); the infarct size, expressed as a percentage of the size of the ipsilateral hemisphere, was significantly larger in aged rats than in young rats.Conclusions. This study demonstrated an age-related increase in the rate and size of venous infarct following vein occlusion, suggesting that the greater vulnerability to CVCDs in the aged brain might be attributed to early and extensive hypoperfusion of circumscribed brain areas drained by the occluded vein. The larger thrombus formation in aged animals indicates that a shift in the thrombogenetic/thrombolytic equilibrium is responsible for the observed effect.

Role of local thrombolysis in cerebral hemorrhagic venous infarct

2014 ◽  
Vol 62 (5) ◽  
pp. 521 ◽  
Author(s):  
ShaileshkumarS Garge ◽  
VirtiD Shah ◽  
Nirmal Surya ◽  
SatishS Khadilkar ◽  
PranavD Modi ◽  
...  
Keyword(s):  
Local Thrombolysis ◽  
Venous Infarct

Sticky Platelet Syndrome: A Cause of Neurovascular Thrombosis and Thrombo-Embolism

1997 ◽  
Vol 3 (2) ◽  
pp. 145-154 ◽  
Author(s):  
E. Berg-Dammer ◽  
H. Henkes ◽  
H. Trobisch ◽  
D. Kühne
Keyword(s):  
Parietal Lobe ◽  
Sinus Thrombosis ◽  
Cerebral Arteries ◽  
Positive Family History ◽  
Adenosine Diphosphate ◽  
Venous Sinus ◽  
Left Vertebral Artery ◽  
Venous Sinus Thrombosis ◽  
Venous Infarct ◽  
Basilar Tip Aneurysm

Increased platelet aggregation induced by adenosine diphosphate and epinephrine and enhanced platelet activation response to surface contact are the key features of the “sticky platelet syndrome”, in which the phenotype is transmitted with an autosomal dominant pattern. Two thirds of the patients with this syndrome have a positive family history of thrombo-embolic disease, which may be triggered by stress. We have seen two patients suffering from intracranial arterial and venous sinus thrombosis due to sticky platelet syndrome. Hyperaggregability of the platelets after stimulation with adenosine diphosphate and epinephrine was found in both. Case 1: A 43 year old man with a superior sagittal and right transverse venous sinus thrombosis developed subdural hygromas and a venous infarct of the parietal lobe. After burr hole evacuation of the hygromas, aspirin and phenoprocumone were prescribed. The patient made a good neurological recovery. Case 2: A 52 year old woman experienced spontaneous occlusion of a right MCA branch. The work-up revealed bilateral MCA and a basilar tip aneurysm. During the attempt to treat the basilar tip aneurysm by endovascular means, a thrombus formed within the left vertebral artery and was dislodged to both posterior cerebral arteries (PCAs). Local intra-arterial fibrinolysis yielded good recanalization of both PCAs. No new neurological deficit occurred. Continuous prophylaxis with aspirin was started. In sticky platelet syndrome stress and adrenaline release can trigger the thrombosis of intracranial arterial or venous vessels in otherwise healthy subjects. This is a potential cause of thromboembolic complications during endovascular procedures. Low-dose aspirin normalizes platelet hyperaggregability.

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