scholarly journals Concomitant Acute Rheumatic Fever and Acute Post Streptococcal Glomerulonephritis

Cureus ◽  
2021 ◽  
Author(s):  
Somshukla Ghosh ◽  
Kelli King-Morris ◽  
Joshua Shultz
Keyword(s):  
Rheumatic Fever ◽  
Acute Rheumatic Fever ◽  
Post Streptococcal Glomerulonephritis

A COMPARISON OF ATRIO-VENTRICULAR CONDUCTION IN NORMAL CHILDREN AND IN PATIENTS WITH RHEUMATIC FEVER, GLOMERULONEPHRITIS, AND ACUTE FEBRILE ILLNESSES

PEDIATRICS ◽  
1964 ◽  
Vol 33 (3) ◽  
pp. 334-340
Author(s):  
M. Mirowski ◽  
Beryl J. Rosenstein ◽  
Milton Markowitz
Keyword(s):  
Rheumatic Fever ◽  
Acute Rheumatic Fever ◽  
Clinical Evidence ◽  
Acute Glomerulonephritis ◽  
Normal Children ◽  
Quantitative Measurements ◽  
Myocardial Involvement ◽  
The Mean ◽  
Post Streptococcal Glomerulonephritis ◽  
Ventricular Conduction

1. Atrio-ventricular conduction was studied in 50 normal children, 25 children with acute febrile illnesses, 50 patients with an initial episode of active rheumatic carditis, 46 patients with acute rheumatic fever without clinical evidence of carditis, and 39 children with acute post-streptococcal glomerulonephritis. 2. Quantitative measurements of A-V conduction were obtained by calculating the P-R index, the ratio between the recorded P-R interval (numerator) and the upper limit of normal for the age and heart rate as given in standard tables (denominator). 3. The mean P-R indices were similar in patients with acute rheumatic fever without clinical evidence of myocardial involvement and in children with overt carditis, and were significantly higher than in normal children, children with acute febrile illnesses of patients with acute glomerulonephritis. 4. The frequency with which high P-R indices were found in patients with acute rheumatic fever suggests that disturbances in A-V conduction are even more common than has been previously recognized from conventional P-R interval determinations. 5. The data suggest also that A-V conduction abnormalities in acute rheumatic fever occur independent of other signs of myocardial involvement and are not related to the ultimate prognosis.

Acute Rheumatic Fever: A population-based study in Wallis, a South Pacific Island

2015 ◽  
Vol 181 ◽  
pp. 30-31 ◽  
Author(s):  
Thomas Fauchier ◽  
Muriel Tafflet ◽  
Graziella Filitoga ◽  
Laurent Morisse ◽  
Eloi Marijon ◽  
...  
Keyword(s):  
Rheumatic Fever ◽  
Acute Rheumatic Fever ◽  
South Pacific ◽  
Population Based ◽  
Pacific Island ◽  
Population Based Study ◽  
South Pacific Island

Mean platelet volume in acute rheumatic fever

Platelets ◽  
2012 ◽  
Vol 24 (5) ◽  
pp. 378-382 ◽  
Author(s):  
Ahmet Sert ◽  
Ebru Aypar ◽  
Dursun Odabas
Keyword(s):  
Rheumatic Fever ◽  
Mean Platelet Volume ◽  
Acute Rheumatic Fever ◽  
Platelet Volume

scholarly journals D8/17 and CD19 Expression on Lymphocytes of Patients with Acute Rheumatic Fever and Tourette's Disorder

2004 ◽  
Vol 11 (2) ◽  
pp. 330-336 ◽  
Author(s):  
Julie L. Weisz ◽  
William M. McMahon ◽  
Jill C. Moore ◽  
Nancy H. Augustine ◽  
John F. Bohnsack ◽  
...  
Keyword(s):  
B Cells ◽  
Rheumatic Fever ◽  
Acute Rheumatic Fever ◽  
Neuropsychiatric Disorders ◽  
Streptococcal Pharyngitis ◽  
Tourette’S Disorder ◽  
Compulsive Disorder ◽  
Tourette's Disorder ◽  
Group A ◽  
Normal Controls

ABSTRACT D8/17, an alloantigen found on B lymphocytes, has been reported to be elevated in patients susceptible to rheumatic fever and may be associated with autoimmune types of neuropsychiatric disorders. The pediatric-autoimmune-neuropsychiatric-disorders-associated-with-streptococci model is a putative model of pathogenesis for a group of children whose symptoms of obsessive-compulsive disorder and Tourette's disorder (TD) are abrupt and may be triggered by an infection with group A streptococci. As a test of this model, we have examined D8/17 levels on the B cells of patients with TD and acute rheumatic fever (ARF) along with those on the B cells of normal controls by flow cytometry. We have utilized several different preparations of D8/17 antibody along with a variety of secondary antibodies but have been unable to show an association with an elevated percentage of D8/17-positive, CD19-positive B cells in either ARF or TD. We did find, however, that the percentages of CD19-positive B cells in ARF and TD patients were significantly elevated compared to those in normal controls. Group A streptococcal pharyngitis patients also had an elevated percentage of CD19 B cells, however. These studies failed to confirm the utility of determining the percentage of B cells expressing the D8/17 alloantigen in ARF patients or our sample of TD patients. In contrast, the percentage of CD19-positive B cells was significantly elevated in ARF and TD patients, as well as group A streptococcal pharyngitis patients, suggesting a role for inflammation and/or autoimmunity in the pathogenesis of these disorders.

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