Delayed Neurologic Sequelae Caused By Carbon Monoxide Poisoning in a Kidney Transplant Recipient

2019 ◽  
Vol 17 (6) ◽  
pp. 849-851
Author(s):  
Sotaro Tsuda ◽  
Junji Uchida ◽  
Tomoaki Iwai ◽  
Chiaki Morooka ◽  
Kazuya Kabei ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Transplant Recipient ◽  
Kidney Transplant ◽  
Kidney Transplant Recipient ◽  
Carbon Monoxide Poisoning ◽  
Neurologic Sequelae ◽  
Delayed Neurologic Sequelae

scholarly journals Development of delayed neurologic sequelae in acute carbon monoxide poisoning cases caused by briquette-based kotatsu

Medicine ◽  
2021 ◽  
Vol 100 (16) ◽  
pp. e25009
Author(s):  
Makoto Onodera ◽  
Yasuhiko Tsukada ◽  
Tsuyoshi Suzuki ◽  
Kotaro Sorimachi ◽  
Kenichi Ebihara ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Carbon Monoxide Poisoning ◽  
Acute Carbon Monoxide Poisoning ◽  
Neurologic Sequelae ◽  
Delayed Neurologic Sequelae

scholarly journals Effect of ethanol in carbon monoxide poisoning and delayed neurologic sequelae: A prospective observational study

PLoS ONE ◽  
2021 ◽  
Vol 16 (1) ◽  
pp. e0245265
Author(s):  
Sungwoo Choi ◽  
Sangsoo Han ◽  
Sangun Nah ◽  
Young Hwan Lee ◽  
Young Soon Cho ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Observational Study ◽  
Prospective Observational Study ◽  
Neuroprotective Effect ◽  
Carbon Monoxide Poisoning ◽  
Co Poisoning ◽  
Ethanol Level ◽  
Neurologic Sequelae ◽  
Gcs Score ◽  
Delayed Neurologic Sequelae

Objectives Carbon monoxide (CO) is one of the most common poisoning substances, which causes mortality and morbidity worldwide. Delayed neurologic sequelae (DNS) have been reported to occur from several days to months after exposure to CO. Thus, there is a need for prevention, recognition, and treatment of DNS. Patients with CO poisoning as a component of intentional suicide often also consume ethanol, but there is debate regarding its role in DNS. We explored whether ethanol has a neuroprotective effect in CO poisoning. Methods This prospective observational study included patients who visited the emergency department from August 2016 to August 2019 due to CO poisoning. After treatment of acute CO poisoning, patients were interviewed by telephone to ascertain whether DNS had occurred within 2 weeks, 1 month, and 3 months from the time of CO exposure. Results During the study period, 171 patients were enrolled. 28 patients (16.37%) developed DNS. The initial Glasgow Coma Scale (GCS) scores were 15 (10.5–15) for the non-DNS group and 10 (7–15) for the DNS group (p = 0.002). The ethanol levels were 11.01 ± 17.58 mg/dL and 1.49 ± 2.63 mg/dL for each group (p < 0.001). In multivariate logistic regression analysis, the GCS score had an odds ratio of 0.770 (p < 0.001) and the ethanol level had 0.882 (p < 0.030) for onset of DNS. Conclusions Higher ethanol level and higher initial GCS score were associated with lower incidence of DNS. Ethanol could have a neuroprotective effect on the occurrence of DNS in CO poisoning patients.

scholarly journals Technetium-99m HM-PAO SPECT in patients with delayed neurologic sequelae after carbon monoxide poisoning

1992 ◽  
Vol 7 (1) ◽  
pp. 11 ◽  
Author(s):  
Il Sang Choi ◽  
Myung Sik Lee ◽  
Young Jin Lee ◽  
Jin Ho Kim ◽  
Sung Soo Lee ◽  
...  
Keyword(s):  
Carbon Monoxide ◽  
Carbon Monoxide Poisoning ◽  
Technetium 99M ◽  
Neurologic Sequelae ◽  
Delayed Neurologic Sequelae
Sign in / Sign up

Export Citation Format

Share Document