scholarly journals Periodontal Inflammation as Risk Factor for Pancreatic Diseases

10.5772/25711 ◽  
2011 ◽  
Author(s):  
Jelena Milasin ◽  
Natasa Nikolic ◽  
Dejan Stefanovic ◽  
Jelena Sopta ◽  
Ana Pucar ◽  
...  
Keyword(s):  
Risk Factor ◽  
Pancreatic Diseases ◽  
Periodontal Inflammation

scholarly journals An Evaluation of the Academic Stress’ Affect on Periodontal Tissues

2019 ◽  
Vol 23 (2) ◽  
pp. 68-72
Author(s):  
Hulya Sariaslan Goleli ◽  
Murat Akkaya ◽  
Melike Camgoz
Keyword(s):  
Risk Factor ◽  
Significant Risk ◽  
Academic Stress ◽  
Dental Students ◽  
Gingival Index ◽  
Pocket Depth ◽  
Periodontal Tissues ◽  
Periodontal Inflammation ◽  
Final Exams ◽  
Crevicular Fluid

Summary Background/Aim: It has been discussed over the years whether emotional stress might be a risk factor for periodontal diseases. The correlation between periodontal disease and stress can still not be explained. Our aim was to evaluate the effects of academic stress on gingival tissues in a prospective design. Material and Methods: The study population consisted of randomly selected 40 dental students. Clinical examinations of plaque (PI), gingival (GI) and sulcus bleeding (SBI) indices, probing pocket depth (PPD) and gingival crevicular fluid flow rate were performed along with State-Trait Anxiety Inventory at three different periods of the school year. Measurements were recorded one month before the finals (1st period), during the final exams (2nd period), and two months after the final exams (3rd period). Results: The changes in mean values of all parameters except plaque and pocket depth between the final and control terms were statistically significant. There was a significant correlation between gingival index and stress, plaque, pocket depth, and sulcus bleeding indices at 1st period. There was a significant correlation between gingival index and crevicular fluid at 2nd period. There was a significant correlation between gingival index and plaque, and sulcus bleeding indices at 3rd period. Conclusions: The present results support the hypothesis that academic stress is a significant risk factor for gingival and periodontal inflammation.

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

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