scholarly journals Coronary sinus diameter by echocardiography to differentiate atrioventricular nodal reentrant tachycardia from atrioventricular reentrant tachycardia

2014 ◽  
Vol 21 (3) ◽  
pp. 273-278 ◽  
Author(s):  
Babu Ezhumalai ◽  
Santhosh Satheesh ◽  
Ajith Anantha ◽  
Gobu Pakkirisamy ◽  
Jayaraman Balachander ◽  
...  
Keyword(s):  
Coronary Sinus ◽  
Atrioventricular Nodal Reentrant Tachycardia ◽  
Reentrant Tachycardia ◽  
Atrioventricular Reentrant Tachycardia

Supraventricular Tachycardia

2020 ◽  
Author(s):  
Laurence M. Epstein ◽  
Saurabh Kumar
Keyword(s):  
Atrial Flutter ◽  
Supraventricular Tachycardia ◽  
Cardiac Tissue ◽  
Accessory Pathway ◽  
Maze Procedure ◽  
Atrioventricular Nodal Reentrant Tachycardia ◽  
Reentrant Tachycardia ◽  
Inappropriate Sinus Tachycardia ◽  
Atrioventricular Reentrant Tachycardia ◽  
Supraventricular Tachycardias

Supraventricular tachycardias (SVTs) comprise a group of usually benign arrhythmias that originate from cardiac tissue at or above the His bundle. SVTs include inappropriate sinus tachycardia, atrial tachycardias (ATs), atrial flutter (AFL), junctional tachycardia, atrioventricular nodal reentrant tachycardia (AVNRT), and forms of accessory pathway–mediated reentrant tachycardias (atrioventricular reentrant tachycardia [AVRT]). Although mostly benign, symptoms can be debilitating, in the form of palpitations, shortness of breath, chest discomfort, dizziness, and/or syncope; rarely, SVTs can result in cardiomyopathy due to incessant arrhythmia. This review covers the epidemiology, diagnosis, management, and classification of SVTs.  This review contains 14 figures, 17 tables, and 61 references. Keywords: Supraventricular tachycardia, cardioversion, arrhythmia, atrial flutter, atrial fibrillation, Wolff-Parkinson-White syndrome, MAZE procedure, catheter ablation

Introduction

ESC CardioMed ◽  
2018 ◽  
pp. 2049-2050
Author(s):  
Carina Blomström-Lundqvist
Keyword(s):  
Atrial Fibrillation ◽  
Catheter Ablation ◽  
Atrial Tachycardia ◽  
Atrioventricular Node ◽  
Supraventricular Arrhythmia ◽  
Atrioventricular Nodal Reentrant Tachycardia ◽  
Reentrant Tachycardia ◽  
Atrioventricular Reentrant Tachycardia ◽  
Dc Cardioversion ◽  
Premature Beats

Supraventricular arrhythmias encompass atrial premature beats, supraventricular tachycardias (SVTs), and atrial fibrillation. SVT is used to describe tachycardias in which the mechanism involves tissue from the His bundle or above, thus including atrial tachycardias, atrioventricular nodal reentrant tachycardia, and atrioventricular reentrant tachycardia due to accessory pathways. Atrial fibrillation is not included among the SVTs and is described elsewhere. The term tachycardia refers to atrial and/or ventricular rates greater than 100 beats per minute at rest. Atrial premature beats, the most common supraventricular arrhythmia, can be seen in Holter recordings in the majority of healthy individuals, and increase in frequency with age and presence of structural heart disease. Paroxysmal SVTs that can be terminated by vagal manoeuvres are usually reentrant tachycardias involving the atrioventricular node, such as atrioventricular nodal reentrant tachycardia or atrioventricular reentrant tachycardia. Symptoms may result in a poor quality of life. Rarely, patients with the Wolff–Parkinson–White syndrome develop atrial fibrillation that may degenerate into ventricular fibrillation in case the anterograde refractory period of the accessory pathway is very short and permanent forms of SVTs result in tachycardiomyopathy with left ventricular dysfunction. Paroxysmal SVT can be terminated by vagal manoeuvres, adenosine, overdrive pacing, and DC cardioversion. Atrial flutter, the most common atrial tachycardia, is a macro-reentrant atrial tachycardia that can be terminated by drugs, overdrive atrial pacing, and DC cardioversion. Most SVTs can be successfully treated by catheter ablation facilitated by modern electroanatomical mapping systems. Long-term antiarrhythmic drug therapy may be required for patients who are not suitable for or cured by catheter ablation.

scholarly journals A Case of Atrioventricular Nodal Reentrant Tachycardia With Atresia of the Coronary Sinus Ostium

2005 ◽  
Vol 46 (5) ◽  
pp. 899-902 ◽  
Author(s):  
Yuji Okuyama ◽  
Takafumi Oka ◽  
Hiroya Mizuno ◽  
Taku Sakai ◽  
Atsushi Hirayama ◽  
...  
Keyword(s):  
Coronary Sinus ◽  
Atrioventricular Nodal Reentrant Tachycardia ◽  
Reentrant Tachycardia ◽  
Coronary Sinus Ostium ◽  
Sinus Ostium

Supraventricular Tachycardia

2012 ◽  
Author(s):  
Laurence M. Epstein
Keyword(s):  
Atrial Flutter ◽  
Considerable Proportion ◽  
Atrioventricular Nodal Reentrant Tachycardia ◽  
Minimal Risk ◽  
Reentrant Tachycardia ◽  
Av Node ◽  
Inappropriate Sinus Tachycardia ◽  
Atrioventricular Reentrant Tachycardia ◽  
Adults And Children ◽  
Antiarrhythmic Medications

Supraventricular tachycardias (SVTs) comprise a group of arrhythmias for which the atria and/or atrioventricular (AV) node are integral to sustaining the rhythm. These arrhythmias typically have a benign natural history but account for a considerable proportion of patients presenting with symptoms including palpitations, shortness of breath, chest discomfort, dizziness, and, on occasion, syncope. They affect a broad range of patients, from young, otherwise healthy adults and children to elderly patients with multiple comorbidities. Although medical management with AV nodal blocking medications or antiarrhythmic medications is a reasonable first-line approach, catheter ablation is a definitive, most often curable option that has minimal risk and offers the chance of avoiding long-term medications. This chapter covers the epidemiology, diagnosis, and management of SVTs, which include atrioventricular nodal reentrant tachycardia (AVNRT), atrioventricular reentrant tachycardia (AVRT), atrial tachycardia (AT), and others (atrial flutter, inappropriate sinus tachycardia, and junctional tachycardia). Atrial fibrillation, which is more prevalent than all other SVTs combined, are discussed elsewhere. Figures describe the differential diagnosis of tachycardia with narrow and wide QRS complexes, the relationship between the response to intravenous adenosine and the cause of tachycardia, the mechanism of tachycardia induction in patients with dual AV node conduction pathways, and the management of atrial flutter. Electrocardiograms illustrate features of various forms of SVT. This review contains 11 highly rendered figures (included 6 twelve-lead ECGs), 1 table, and 69 references.

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