scholarly journals Bilateral internal carotid artery and vertebral artery dissections with retinal artery occlusion after a roller coaster ride - case report and a review

2011 ◽  
Vol 17 (1) ◽  
pp. 75-78 ◽  
Author(s):  
Yonca ÖZKAN ARAT
Keyword(s):  
Case Report ◽  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Vertebral Artery ◽  
Internal Carotid ◽  
Retinal Artery Occlusion ◽  
Artery Occlusion ◽  
Roller Coaster ◽  
Retinal Artery

Central retinal artery occlusion in association with an aneurysm of the internal carotid artery

2001 ◽  
Vol 132 (2) ◽  
pp. 270-271 ◽  
Author(s):  
Christos Haritoglou ◽  
Stefanie Müller-Schunk ◽  
Christian Weber ◽  
Ulrich Hoffmann ◽  
Michael W Ulbig
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Internal Carotid ◽  
Central Retinal Artery Occlusion ◽  
Retinal Artery Occlusion ◽  
Artery Occlusion ◽  
Central Retinal Artery ◽  
Retinal Artery

scholarly journals Monocular central retinal artery occlusion caused by bilateral internal carotid artery hypoplasia complicated with patent foramen ovale: a case report and review of literature

2021 ◽  
Vol 26 (1) ◽  
Author(s):  
Lanbing Zhu ◽  
Na Xu ◽  
Yan Li
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Patent Foramen Ovale ◽  
Internal Carotid ◽  
Central Retinal Artery Occlusion ◽  
Retinal Artery Occlusion ◽  
Artery Occlusion ◽  
Central Retinal Artery ◽  
Foramen Ovale ◽  
Retinal Artery

Abstract Background Central retinal artery occlusion (CRAO) is an emergent ophthalmic disease which is commonly caused by atherosclerosis, thromboembolism, and arteriospasm. Here, we report a case of CRAO which is caused by extreme rare bilateral internal carotid artery (ICA) hypoplasia complicated with patent foramen ovale (PFO). The cardiogenic emboli blocked central retinal artery through unclosed foramen ovale and specific blood flow pathway. Case presentation This report describes a case of a 46-year-old woman sudden onset with amaurosis fugax for about 20 min and persistent visual impairment of left eye. Fundus fluorescein angiography shows the arm-retinal circulation time of left eye is 25 s, indicating that the occlusion occurs in the pathway from aortic arch to ophthalmic artery. The MRA and CTA examinations reveal the bilateral ICA hypoplasia and variation of Wills circle. Furthermore, transesophageal echocardiography (TEE) confirms the PFO and cardiogenic embolic event. Conclusions This work presents a CRAO case caused by rare congenital hypoplasia of ICA complicated with PFO, reminding us every single cause of vascular disease should be investigated carefully and the TOAST typing of cerebrovascular disease can be of great reference to the ocular vascular disease.

Analysis of emboli and blood flow in the ophthalmic artery to understand retinal artery occlusion

2014 ◽  
Vol 59 (6) ◽  
Author(s):  
Tim A.S. Kaufmann ◽  
Christoph Leisser ◽  
Jeannie Gemsa ◽  
Ulrich Steinseifer
Keyword(s):  
Blood Flow ◽  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Ophthalmic Artery ◽  
Internal Carotid ◽  
Retinal Artery Occlusion ◽  
Artery Occlusion ◽  
Patient Specific ◽  
Retinal Artery ◽  
The Brain

AbstractRetinal artery occlusion (RAO) is a common ocular vascular occlusive disorder that may lead to partial or complete retinal ischemia with sudden visual deterio ration and visual field defects. Although RAO has been investigated since 1859, the main mechanism is still not fully understood. While hypoperfusion of the ophthalmic artery (OA) due to severe stenosis of the internal carotid artery might lead to RAO, emboli are assumed to be the main reason. Intra-arterial thrombolysis is not a sufficient treatment for RAO, and current research is mainly focused on risk factors. In this study, a computational fluid dynamic model is presented to analyse flow conditions and clot behaviour at the junction of the internal carotid artery and OA based on a realistic geometry from a RAO patient. Clot diameters varied between 5 and 200 μm, and the probability of clots reaching the OA or being washed into the brain was analysed. Results show sufficient blood flow and perfusion pressure at the end of OA. The probability that clots from the main blood flow will to be washed into the brain is 7.32±1.08%. A wall shear stress hotspot is observed at the curvature proximal to the internal carotid artery/OA junction. Clots released from this hotspot have a higher probability of causing RAO. The occurrence of such patient-specific pathophysiologies will have to be considered in the future.

Tirofiban as Treatment for Acute Retinal Artery Occlusion following Internal Carotid Artery Flow Diverter Implantation

2016 ◽  
Vol 26 (4) ◽  
pp. e74-e76
Author(s):  
Luisa Pierro ◽  
Carlo La Spina ◽  
Federico Corvi ◽  
Maria V. Cicinelli ◽  
Francesco Scomazzoni ◽  
...  
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Internal Carotid ◽  
Retinal Artery Occlusion ◽  
Flow Diverter ◽  
Artery Occlusion ◽  
Retinal Artery ◽  
Artery Flow
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