An Evaluation Of The Precision Of QIAGEN Quantiplex HYres Kit As a Screening Tool On Low Level Touch DNA Evidence

2016 ◽  
Vol 3 (2) ◽  
pp. 61 ◽  
Author(s):  
Stephen IP ◽  
Kamming LAI
2012 ◽  
Vol 6 (5) ◽  
pp. 548-558 ◽  
Author(s):  
E. Hanson ◽  
C. Haas ◽  
R. Jucker ◽  
J. Ballantyne
Keyword(s):  

2012 ◽  
Vol 6 (5) ◽  
pp. e141 ◽  
Author(s):  
E. Hanson ◽  
C. Haas ◽  
R. Jucker ◽  
J. Ballantyne
Keyword(s):  

2009 ◽  
Vol 18 (4) ◽  
pp. 129-133 ◽  
Author(s):  
Kelly Poskus

Abstract The bedside swallow screen has become an essential part of the evaluation of a patient after stroke in the hospital setting. Implementing this type of tool should be simple. However, reinforcement and monitoring of the tool presents a challenge. Verifying the consistency and reliability of nurses performing the bedside swallow screen can be a difficult task. This article will document the journey of implementing and maintaining a reliable and valid nursing bedside swallow screen.


2006 ◽  
Vol 76 (1) ◽  
pp. 28-33 ◽  
Author(s):  
Yukari Egashira ◽  
Shin Nagaki ◽  
Hiroo Sanada

We investigated the change of tryptophan-niacin metabolism in rats with puromycin aminonucleoside PAN-induced nephrosis, the mechanisms responsible for their change of urinary excretion of nicotinamide and its metabolites, and the role of the kidney in tryptophan-niacin conversion. PAN-treated rats were intraperitoneally injected once with a 1.0% (w/v) solution of PAN at a dose of 100 mg/kg body weight. The collection of 24-hour urine was conducted 8 days after PAN injection. Daily urinary excretion of nicotinamide and its metabolites, liver and blood NAD, and key enzyme activities of tryptophan-niacin metabolism were determined. In PAN-treated rats, the sum of urinary excretion of nicotinamide and its metabolites was significantly lower compared with controls. The kidneyα-amino-β-carboxymuconate-ε-semialdehyde decarboxylase (ACMSD) activity in the PAN-treated group was significantly decreased by 50%, compared with the control group. Although kidney ACMSD activity was reduced, the conversion of tryptophan to niacin tended to be lower in the PAN-treated rats. A decrease in urinary excretion of niacin and the conversion of tryptophan to niacin in nephrotic rats may contribute to a low level of blood tryptophan. The role of kidney ACMSD activity may be minimal concerning tryptophan-niacin conversion under this experimental condition.


1983 ◽  
Vol 28 (1) ◽  
pp. 79-79
Author(s):  
Claire B. Ernhart

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