Oral ferrous citrate or ferrous sulfate use during predialysis may reduce serum phosphate level at dialysis initiation

2019 ◽  
Vol 92 (4) ◽  
pp. 180-189
Author(s):  
Shigehisa Koide ◽  
Daijo Inaguma ◽  
Eri Koshi-Ito ◽  
Kazuo Takahashi ◽  
Hiroki Hayashi ◽  
...  
1996 ◽  
Vol 25 (4) ◽  
pp. 182-187 ◽  
Author(s):  
Y. Masatomi ◽  
Y. Nakagawa ◽  
Y. Kanamoto ◽  
S. Sobue ◽  
T. Ooshima

Resuscitation ◽  
2018 ◽  
Vol 128 ◽  
pp. 56-62 ◽  
Author(s):  
Yong Hun Jung ◽  
Byung Kook Lee ◽  
Kyung Woon Jeung ◽  
Chun Song Youn ◽  
Dong Hun Lee ◽  
...  

2019 ◽  
Vol 38 (1) ◽  
pp. 1-6
Author(s):  
Seiji Fukumoto ◽  
Yuichi Takashi ◽  
Maria K. Tsoumpra ◽  
Shun Sawatsubashi ◽  
Toshio Matsumoto

Circulation ◽  
2005 ◽  
Vol 112 (17) ◽  
pp. 2627-2633 ◽  
Author(s):  
Marcello Tonelli ◽  
Frank Sacks ◽  
Marc Pfeffer ◽  
Zhiwei Gao ◽  
Gary Curhan

Burns ◽  
2010 ◽  
Vol 36 (7) ◽  
pp. 1112-1115 ◽  
Author(s):  
Shahriar Loghmani ◽  
Mohammad Reza Maracy ◽  
Reza Kheirmand

2017 ◽  
Vol 2017 ◽  
pp. 1-6 ◽  
Author(s):  
Kiyonori Ito ◽  
Susumu Ookawara ◽  
Tomohisa Okochi ◽  
Yuichiro Ueda ◽  
Masaya Kofuji ◽  
...  

Background. Near-infrared spectroscopy revealed that the regional saturation of oxygen (rSO2) in cerebral tissue is lower in hemodialysis (HD) patients than in healthy subjects. However, no study has examined the changes in cerebral oxygenation by aortic arch calcification (AAC) progression in HD patients. Methods. A total of 104 HD patients were divided into four groups by AAC grade determined using chest radiography: 23 patients at grade 0, 24 at grade 1, 30 at grade 2, and 27 at grade 3. Differences in clinical parameters, including cerebral rSO2, among AAC grades were investigated and atherosclerotic parameters affecting cerebral rSO2 values were identified. Results. Cerebral rSO2 significantly decreased as AAC progressed (AAC grade 3 versus grade 0, p<0.01 versus grade 1, p<0.05). Multivariate logistic regression analysis was performed using parameters with p values < 0.20 in univariate analysis between cerebral rSO2 values less than the mean and atherosclerotic parameters. AAC grades 2 and 3, serum phosphate level, and history of smoking were factors associated with the cerebral rSO2 decrease. Conclusions. Cerebral rSO2 significantly decreased as AAC progressed and was independently associated with higher AAC grade, serum phosphate level, and history of smoking.


2021 ◽  
Vol 9 ◽  
Author(s):  
Peong Gang Park ◽  
Seon Hee Lim ◽  
HyunKyung Lee ◽  
Yo Han Ahn ◽  
Hae Il Cheong ◽  
...  

Background: X-linked hypophosphatemia (XLH) is the most frequent form of hypophosphatemic rickets and is caused by mutations in the PHEX gene. We analyzed genotype-phenotype correlations in XLH patients with proven PHEX mutations.Methods:PHEX mutations were detected in 55 out of 81 patients who clinically presented with hypophosphatemic rickets. The patients were grouped into nontruncating (n = 9) and truncating (n = 46) mutation groups; their initial presentation as well as long-term clinical findings were evaluated according to these groups.Results: Initial findings, including presenting symptoms, onset age, height standard deviation scores (SDS), and laboratory tests, including serum phosphate level and tubular resorption of phosphate, were not significantly different between the two groups (onset age: nontruncating mutation group, 2.0 years, truncating mutation group, 2.2 years; height SDS: nontruncating mutation group, −1.9, truncating mutation group, −1.7; serum phosphate: nontruncating mutation group, 2.5 mg/dL, truncating mutation group, 2.6 mg/dL). However, at their last follow-up, the serum phosphate level was significantly lower in patients with truncating mutations (nontruncating mutation group: 3.2 mg/dl, truncating mutation group: 2.3 mg/dl; P = 0.006). Additionally, 62.5% of patients with truncating mutations developed nephrocalcinosis at their last follow-up, while none of the patients with nontruncating mutations developed nephrocalcinosis (P = 0.015). Orthopedic surgery due to bony deformations was performed significantly more often in patients with truncating mutations (52.3 vs. 10.0%, P = 0.019).Conclusion: Although considerable inconsistency exists regarding the correlation of truncating mutations and their disease phenotype in several other studies, we cautiously suggest that there would be genotype-phenotype correlation in some aspects of disease manifestation after long-term follow-up. This information can be used when consulting patients with confirmed XLH regarding their disease prognosis.


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