scholarly journals Camel Milk Is a Safer Choice than Goat Milk for Feeding Children with Cow Milk Allergy

ISRN Allergy ◽  
2011 ◽  
Vol 2011 ◽  
pp. 1-5 ◽  
Author(s):  
Mohammad Ehlayel ◽  
Abdulbari Bener ◽  
Khalid Abu Hazeima ◽  
Fatima Al-Mesaifri
Keyword(s):  
Atopic Dermatitis ◽  
Prospective Study ◽  
Goat Milk ◽  
Specific Ige ◽  
Cross Reactivity ◽  
Medical Examination ◽  
Camel Milk ◽  
Cow Milk ◽  
Milk Allergy ◽  
Cow Milk Allergy

Background. Various sources of mammalian milk have been tried in CMA. Objectives. To determine whether camel milk is safer than goat milk in CMA. Methods. Prospective study conducted at Hamad Medical Corporation between April 2007 and April 2010, on children with CMA. Each child had medical examination, CBC, total IgE, cow milk-specific IgE and SPT. CMA children were tested against fresh camel and goat milks. Results. Of 38 children (median age 21.5 months), 21 (55.3%) presented with urticaria, 17 (39.5%) atopic dermatitis, 10 (26.3%) anaphylaxis. WBC was 10,039±4,735 cells/μL, eosinophil 1,143±2,213 cells/μL, IgE 694±921 IU/mL, cow's milk-specific-IgE 23.5±35.6 KU/L. Only 7 children (18.4%) tested positive to camel milk and 24 (63.2%) to goat milk. 6 (15.8%) were positive to camel, goat, and cow milks. Patients with negative SPT tolerated well camel and goat milks. Conclusions. In CMA, SPT indicates low cross-reactivity between camel milk and cow milk, and camel milk is a safer alternative than goat milk.

Camel milk in children with cow milk allergy: Is it time for more investigations and less skepticism?

2015 ◽  
Vol 47 ◽  
pp. e265
Author(s):  
G. Monti ◽  
A. Bua ◽  
M. Rubino ◽  
A.M. Mahdi ◽  
G.A. Mazza ◽  
...  
Keyword(s):  
Camel Milk ◽  
Cow Milk ◽  
Milk Allergy ◽  
Cow Milk Allergy

scholarly journals Clinic al and immunologic al characteristic ofinfants with atopic dermatitis

2011 ◽  
Vol 8 (5) ◽  
pp. 31-36
Author(s):  
Natal'ya Aleksandrovna Ivanova ◽  
G A Kuz'mina ◽  
L T Kochish ◽  
N N Begaeva ◽  
N M Afanas'eva ◽  
...  
Keyword(s):  
Atopic Dermatitis ◽  
House Dust ◽  
Clinical Manifestations ◽  
Milk Proteins ◽  
Goat Milk ◽  
Specific Ige ◽  
Cow Milk ◽  
Enzyme Analysis ◽  
Group I ◽  
Chicken Eggs

Background. The research is focused on the particularities of formation and course of atopic dermatitis in infancy depending on the range of sensitization and class of revealed antibodies. Methods. The article presents the results of clinical and immunological observation of 116 children aged from 1 month to 3 years with the clinical-anamnestic signs of atopic dermatitis. The immunological observation consisted of the following stages: determination of concentration of the general IgE, quantitative testing of concentrations of the serum IgE and IgG4 antibodies to allergens of cow and goat milk, chicken eggs and wheat, house dust and through the immune-enzyme analysis. Results and discussion. According to the results of the immunological observation the patients were divided into 3 groups: I - IgE+IgG4 - positive, II - IgG4 - positive, III - absence of above-mentioned classes of antibodies. The patients of group I are characterized by the early sensitization to cow milk proteins, the manifestation of the disease up to 3 months appears on pure breast feeding. The detection of the specific IgE antibodies to allergens of chicken eggs is a sign of danger of the development of sensitization towards inhalation house dust allergens and of the formation of respiratory allergy. For the children of group II the later age of manifestation of skin process (in more than half of children after introduction of milk food) and easier course of disease are significant. Anamnesis of the disease and the clinical manifestations of one third of patients of group III in the absence of the evidence of IgE-sensitization coincides with that of the patients of group I (age of manifestation, severity of disease). In accordance with received results, we can suggest that in this group there are patients with the unidentified immunopathological mechanism of formation of the disease. Conclusion. The immunological observation of infants by the detection of the specific IgE and IgG4 antibodies to widespread food allergens allows to reveal the peculiarities of immune response which in their turn define the clinical-anamnestic signs of atopic dermatitis.

The Role of Cow Milk Allergy in Increasing the Severity of Atopic Dermatitis

2004 ◽  
Vol 33 (1) ◽  
pp. 69-79 ◽  
Author(s):  
Z. Pourpak ◽  
A. Farhoudi ◽  
Maryam Mahmoudi ◽  
M. Movahedi ◽  
M. Ghargozlou ◽  
...  
Keyword(s):  
Atopic Dermatitis ◽  
Cow Milk ◽  
Milk Allergy ◽  
Cow Milk Allergy

scholarly journals Oral Desensitisation in Cow Milk Allergy: Immunological Findings

2002 ◽  
Vol 15 (1) ◽  
pp. 53-58 ◽  
Author(s):  
G. Patriarca ◽  
A. Buonomo ◽  
C. Roncallo ◽  
M. Del Ninno ◽  
E. Pollastrini ◽  
...  
Keyword(s):  
Food Allergy ◽  
Specific Ige ◽  
Cow Milk ◽  
Milk Allergy ◽  
Eosinophilic Cationic Protein ◽  
Cationic Protein ◽  
Total Ige ◽  
Immunological Mechanisms ◽  
Cow Milk Allergy ◽  
Β Lactoglobulin

In the literature there are several reports dealing with the possibility of a desensitising treatment in food allergy, but there are very few studies concerning the immunological mechanisms of or al desensitisation. We studied the immunological modifications in four children who underwent oral desensitisation with cow milk. Four children with cow milk allergy underwent oral desensitisation according to a standardized protocol. Total IgE, eosinophilic cationic protein in serum, and specific IgE and IgG4 to α-lactalbumin, to β-lactoglobulin and to casein were determined at the beginning of the treatment and after 6, 12 and 18 months in the 4 children treated. All the 4 treated patients successfully completed the treatment. Specific IgE to casein showed a significant reduction (p<0.01), while specific IgG4 to α-lactalbumin (p<0.02), to β-lactoglobulin (p<0.01) and to casein (p<0.01) showed a significant increase. Total IgE, eosinophilic cationic protein, and specific IgE to a-lactalbumin and to β-Iactoglobulin did not show any significant modification. Control patients did not show any immunological modification and still had a positive double-blind, placebo-controlled food challenge. These results make us think that oral desensitisation in food allergy occurs with the same mechanisms of traditional desensitising treatments for respiratory and insect sting allergies.

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