scholarly journals Vieussens' arterial ring

2020 ◽  
Author(s):  
Balint Botz ◽  
Patrick Rock
Keyword(s):  
2007 ◽  
Vol 47 (1) ◽  
pp. 25-30 ◽  
Author(s):  
Pei-Han Xu ◽  
Yuan Long ◽  
Fang Dai ◽  
Zhong-Li Liu

Circulation ◽  
2009 ◽  
Vol 120 (15) ◽  
Author(s):  
Chi Yuen Chan ◽  
Pui Wai Lee ◽  
Yat-Yin Lam ◽  
Wilson Wai-Man Chan ◽  
Ka Tak Wong ◽  
...  

2019 ◽  
Vol 25 (2) ◽  
pp. 109-113 ◽  
Author(s):  
Nurullah Dogan ◽  
◽  
Aydin Dursun ◽  
Hakan Ozkan ◽  
◽  
...  

1992 ◽  
Vol 263 (5) ◽  
pp. H1437-H1446 ◽  
Author(s):  
J. P. Headrick ◽  
F. J. Northington ◽  
M. R. Hynes ◽  
G. P. Matherne ◽  
R. M. Berne

Responses to luminal and adventitial adenosine were compared in perfused rabbit central ear arteries. Perfused arteries precontracted with 0.5 microM norepinephrine relaxed dose dependently but asymmetrically to luminal and adventitial adenosine. Arteries were more responsive to luminal adenosine in the 0.1- to 1.0-microM range, but they were more responsive to adventitial adenosine at doses > 10 microM. Alternatively, 2-chloroadenosine, a metabolically stable and poorly transported analogue, was equipotent when applied luminally or adventitially. Endothelial damage reduced sensitivity and response asymmetry to luminal and adventitial adenosine. This was consistent with reduced responses to adenosine in luminally rubbed arterial ring segments. Transport inhibition (10 microM dipyridamole) enhanced arterial reactivity to luminal and adventitial adenosine and reduced response asymmetry but was without effect on responses to 2-chloroadenosine. A comparison of the inhibitory effectiveness of adventitial and luminal 8-phenyltheophylline revealed that adventitial antagonist was approximately threefold more effective in inhibiting responses to adventitial adenosine than luminal antagonist (P < 0.05). This "side-dependent" difference was reduced by prolonged antagonist incubation or endothelial removal. The data indicate that adenosine relaxes ear arteries by activation of smooth muscle [half-maximum effective concentration (ED50) approximately 11 microM] and endothelial (ED50 approximately 2 microM) receptors. Nevertheless, a sensitive endothelial-dependent response does not consistently enhance responses to luminal adenosine in perfused arteries. This appears to be attributable to relative differences in the smooth muscle and endothelium-dependent components of the dilator response and transvascular concentration gradients for luminally and adventitially applied adenosine. A transendothelial diffusion barrier also reduces the ability of luminally applied antagonists to inhibit responses to adventitial adenosine.


1999 ◽  
Vol 2 (5) ◽  
pp. 464-472 ◽  
Author(s):  
A.W. Bates ◽  
S.M. Dodd

The anatomical features of a pair of female conjoined twins of the cephalothoracopagus type are described: the anterior face was well formed but the posterior face was cyclopean with a single ear canal. Visceral abnormalities included two shared hearts linked by an anastomotic arterial ring. The smaller of the hearts was univentricular with atrial situs ambiguus and anomalous pulmonary venous return, and was associated with two trilobed lungs. This, and the presence of a single spleen, suggested asplenia syndrome in one twin. The brains, orientated with respect to the notochordal axes, were joined only at the frontal poles. The abnormalities described in previous reports of twins of this type are reviewed and compared. In addition to alterations in body pattern due to conjoining, endodermal developmental defects and abnormal laterality appear to have been involved in the morphogenesis of this case.


2009 ◽  
Vol 64 (11) ◽  
pp. 1129-1131 ◽  
Author(s):  
A.R. Owen ◽  
S.C. Moten ◽  
M.P. Molan

2021 ◽  
Vol 8 ◽  
Author(s):  
Jianwen Liang ◽  
Jieping Huang ◽  
Wanbing He ◽  
Guangzi Shi ◽  
Jie Chen ◽  
...  

Background: Vascular calcification (VC) is a landmark of aging, while β-hydroxybutyric acid (BHB) induced by calorie restriction has been identified as a promising factor to extend the lifespan. However, the effect of BHB on VC and the potential mechanism remain unknown.Methods: A total of 160 subjects with or without metabolic abnormalities (MAs) were assigned to four groups according to different calcification severities. The association between BHB, MAs, and VC was investigated via mediation analysis. Then, with high phosphate-induced calcification models, the effect of BHB on arterial ring calcification and osteogenic phenotypic differentiation of vascular smooth muscle cells (VSMCs) was investigated. Hereafter the expressions of autophagy biomarkers, autophagy flux, and effects of autophagy inhibitors on VC were detected.Results: Severe VC was observed in the elderly, accompanied with a higher proportion of hypertension, chronic kidney disease, and lower estimated glomerular filtration rate. The serum BHB level was an independent influencing factor of VC severities. With mediation analysis, BHB was determined as a significant mediator in the effects of MAs on VC, and the indirect effect of BHB accounted for 23% of the total effect. Furthermore, BHB directly inhibited arterial ring calcification and osteogenic phenotypic differentiation in VSMCs, accompanied with autophagy enhancement in VSMCs. In accordance, the inhibition of autophagy counteracted the protective effect of BHB on VC.Conclusion: The present study demonstrated that BHB mediated the effects of MAs on VC; then, it further elucidated that BHB could inhibit arterial and VSMC calcification via autophagy enhancement.


2018 ◽  
Vol 17 (3) ◽  
pp. 193-194
Author(s):  
Haruomi Yamaguchi ◽  
Akira Uchino ◽  
Naoko Saito ◽  
Ichiro Deguchi ◽  
Masaki Takao

2020 ◽  
Vol 101 (7-8) ◽  
pp. 499-500
Author(s):  
M. Cellina ◽  
F. Bombaci ◽  
D. Gibelli ◽  
P. Marta ◽  
G. Oliva
Keyword(s):  

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