scholarly journals Elbow synovial fold syndrome

2021 ◽  
Author(s):  
Balint Botz ◽  
Henry Knipe
Keyword(s):  
1999 ◽  
Vol 70 (1) ◽  
pp. 85-86 ◽  
Author(s):  
Kiyoshi Sakai ◽  
Masahiko Kanamori ◽  
Satoru Kitano
Keyword(s):  

2014 ◽  
Vol 38 (4) ◽  
pp. 503-506 ◽  
Author(s):  
Mimi Kim ◽  
Seunghun Lee ◽  
Bong Gun Lee ◽  
Doo-Jin Paik ◽  
Jiyoon Bae

2021 ◽  
Vol 29 (2) ◽  
pp. 72-75
Author(s):  
GILBERTO LUIS CAMANHO ◽  
RICCARDO GOMES GOBBI ◽  
MARTA HALASZ DE ANDRADE

ABSTRACT Objective: The synovial fold is an intra-articular structure found in more than 50% of the knees, which can cause symptoms similar to meniscal injuries. These symptoms are mostly related to hypertrophy of the synovial fold resulting from inadequate physical activity. Conservative treatment with readjustment of sports activity and muscle rebalancing solves most cases. Rare cases require surgical treatment, which is indicated due to the persistence of instability, blockage and pain. We present our experience in the treatment of this pathology. Methods: 58 patients (70 knees), with 62 knees treated conservatively and 8 treated surgically exclusively for the pathological synovial fold. Results: Description of the series and treatment results are reported. Conclusion: The non-surgical treatment of the pathological synovial fold of the knee provided good results within 60 days of rehabilitation program in almost 90% of the patients. Arthroscopic resection of the synovial fold is a surgery that has a longer and laborious rehabilitation period, despite good results in most cases. Level of Evidence IV, Case series.


2000 ◽  
Vol 5 (5) ◽  
pp. 475-480 ◽  
Author(s):  
Satoshi Inami ◽  
Koji Kaneoka ◽  
Koichiro Hayashi ◽  
Naoyuki Ochiai

2019 ◽  
Vol 4 (4) ◽  
pp. 2473011419S0026
Author(s):  
Jae Bum Kwon ◽  
Sungjun Kim ◽  
Seung-pyo Hong ◽  
Jin Woo Lee ◽  
Seung Hwan Han

Category: Ankle, Arthroscopy Introduction/Purpose: During ankle arthroscopic surgery, chronic syndesmotic instability is a common combined finding of ankle instability and induces the development of a hypertrophic synovial fold. This pathologic lesion protrudes into the ankle joint cavity in various tissue types, such and hypertrophied synovium, fat or fibrosis. However, the effect of this impingement tissue on the ankle joint and the associated clinical significance have not been well-evaluated. This study aimed to identify the histological type of the syndesmotic impingement tissue and analyze its effect on the lateral talar dome cartilage, in addition to the clinical correlation with the tissue type. Methods: A total of 62 patients (64 ankles) with chronic ankle instability and syndesmotic impingement who underwent arthroscopic surgical management from January 2017 to June 2018 were enrolled in this study. During arthroscopic surgery, we visually classified the hypertrophic syndesmotic tissue and performed biopsy. We also evaluated the prevalence of cartilage lesions at the lateral talar dome with its location. The association between the duration of symptoms and tissue diagnosis was also evaluated. Results: Based on the pathologic results, 4 samples with fat degeneration, 37 samples with fibrosis, 7 samples with synovial hypertrophy, 12 samples showing mixed type hypertrophy, and 4 samples with other type of hypertrophy were identified. Among the 64 cases, articular cartilage lesions at the lateral talar dome were present in 15 cases. In 10 of 15 cases, the pathologic results revealed the presence of fibrous tissue, while 5 cases of other type including synovial hypertrophy, were identified. In 13 cases, the cartilage lesions were located in the middle third of the lateral talar dome. The patients experienced ankle joint instability for 35.5 ± 33.7 months. Longer duration (>24 months) of instability was associated with a higher rate (81.8%) of fibrotic change in the syndesmosis. Conclusion: Repetitive syndesmotic impingement can cause hypertrophic synovial changes; the hypertrophic synovial tissue may evolve into fibrotic impingement tissue as the duration of symptom persistence increases. If the hypertrophic tissues are fibrotic, they may be harder in terms of physical properties and may cause the development of cartilage lesions at the talar dome due to the repetitive load. Considering our results, prophylactic debridement may be effective to prevent the development of cartilage lesions at the talar dome, when hypertrophic tissues are observed during arthroscopic surgeries.


Rheumatology ◽  
1987 ◽  
Vol 26 (2) ◽  
pp. 93-98 ◽  
Author(s):  
L. G. F. GILES ◽  
J. R. TAYLOR

1992 ◽  
Vol 5 (2) ◽  
pp. 227-231 ◽  
Author(s):  
Han Chang ◽  
Ernest M. Found ◽  
Charles R. Clark ◽  
Vijay K. Goel ◽  
Chang S. Gang
Keyword(s):  

1998 ◽  
Vol 7 (4) ◽  
pp. 427-429 ◽  
Author(s):  
Masao Akagi ◽  
Takashi Nakamura
Keyword(s):  

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