scholarly journals Myocardial oedema

2020 ◽  
Author(s):  
Joachim Feger
Keyword(s):  
Myocardial Oedema

The Role of Na+–H+Exchange Occurring During Hypoxia in the Genesis of Reoxygenation-induced Myocardial Oedema

1997 ◽  
Vol 29 (4) ◽  
pp. 1167-1175 ◽  
Author(s):  
Javier Inserte ◽  
David Garcia-Dorado ◽  
Marisol Ruiz-Meana ◽  
Julia Solares ◽  
J. Soler
Keyword(s):  
Myocardial Oedema

scholarly journals Acute Ventricular Wall Thickening: Sepsis, Thrombotic Microangiopathy, or Myocarditis?

2015 ◽  
Vol 2015 ◽  
pp. 1-4
Author(s):  
Nicolas De Schryver ◽  
Delphine Hoton ◽  
Diego Castanares-Zapatero ◽  
Philippe Hantson
Keyword(s):  
Thrombotic Microangiopathy ◽  
Ischemia Reperfusion Injury ◽  
Ischemia Reperfusion ◽  
Interventricular Septum ◽  
Left Ventricular ◽  
Left Ventricular Ejection ◽  
Wall Thickening ◽  
Ventricular Wall ◽  
Myocardial Oedema ◽  
Ventricular Ejection Fraction

Background. Acute myocardial oedema has been documented in experimental models of ischemia-reperfusion injury or sepsis and is usually investigated by magnetic resonance imaging.Purpose. We describe a case of acute ventricular wall thickening documented by echocardiography in a patient developing sepsis and thrombotic microangiopathy.Case Description. A 40-year-old woman, with a history of mixed connective tissue disease, was admitted with laryngeal oedema and fever. She developedStreptococcus pneumoniaesepticaemia and subsequent laboratory abnormalities were consistent with a thrombotic microangiopathy. Echocardiography revealed an impressive diffuse thickening of the whole myocardium (interventricular septum 18 mm; posterior wall 16 mm) with diffuse hypokinesia and markedly reduced left ventricular ejection fraction (31%). There was also a moderate pericardial effusion. Echocardiography was normal two months before. The patient died from acute heart failure. Macroscopic and microscopic examination of the heart suggested that the ventricular wall thickening was induced by oedematous changes, together with an excess of inflammatory cells.Conclusion. Acute ventricular wall thickening that corresponded to myocardial oedema as a first hypothesis was observed at echocardiography during the course of septicaemia complicated by thrombotic microangiopathy.

scholarly journals The natural time course of myocardial oedema in the 12 months post ST-elevation MI in patients treated with primary angioplasty

2012 ◽  
Vol 14 (S1) ◽  
Author(s):  
Thomas R Burchell ◽  
Mark Westwood ◽  
Steffen E Petersen ◽  
Saidi A Mohiddin ◽  
Ceri Davies
Keyword(s):  
Time Course ◽  
Primary Angioplasty ◽  
Myocardial Oedema ◽  
Natural Time ◽  
St Elevation

Feature-tracking myocardial strain analysis in acute myocarditis: diagnostic value and association with myocardial oedema

2017 ◽  
Vol 27 (11) ◽  
pp. 4661-4671 ◽  
Author(s):  
Julian A. Luetkens ◽  
Ulrike Schlesinger-Irsch ◽  
Daniel L. Kuetting ◽  
Darius Dabir ◽  
Rami Homsi ◽  
...  
Keyword(s):  
Feature Tracking ◽  
Acute Myocarditis ◽  
Myocardial Strain ◽  
Strain Analysis ◽  
Diagnostic Value ◽  
Myocardial Oedema ◽  
Myocardial Strain Analysis

scholarly journals Myocardial oedema and ventricular function after cardioplegia with added mannitol

1991 ◽  
Vol 38 (1) ◽  
pp. 7-14 ◽  
Author(s):  
Ryokichi Goto ◽  
Howard Tearle ◽  
David J. Steward ◽  
Phillip G. Ashmore
Keyword(s):  
Ventricular Function ◽  
Myocardial Oedema

scholarly journals Multimodality cardiac evaluation in children and young adults with multisystem inflammation associated with COVID-19

2020 ◽  
Author(s):  
Paraskevi Theocharis ◽  
James Wong ◽  
Kuberan Pushparajah ◽  
Sujeev K Mathur ◽  
John M Simpson ◽  
...  
Keyword(s):  
Coronary Artery ◽  
Multimodality Imaging ◽  
Cardiac Computed Tomography ◽  
Three Dimensional ◽  
Cardiovascular Effects ◽  
Tissue Doppler ◽  
Valvular Regurgitation ◽  
Close Monitoring ◽  
Myocardial Oedema ◽  
Coronary Artery Dilatation

Abstract Aims Following the peak of the UK COVID-19 epidemic, a new multisystem inflammatory condition with significant cardiovascular effects emerged in young people. We utilized multimodality imaging to provide a detailed sequential description of the cardiac involvement. Methods and Results Twenty consecutive patients (mean age 10.6 ± 3.8 years) presenting to our institution underwent serial echocardiographic evaluation on admission (median day 5 of illness), the day coinciding with worst cardiac function (median day 7), and the day of discharge (median day 15). We performed cardiac computed tomography (CT) to assess coronary anatomy (median day 15) and cardiac magnetic resonance imaging (CMR) to assess dysfunction (median day 20). On admission, almost all patients displayed abnormal strain and tissue Doppler indices. Three-dimensional (3D) echocardiographic ejection fraction (EF) was <55% in half of the patients. Valvular regurgitation (75%) and small pericardial effusions (10%) were detected. Serial echocardiography demonstrated that the mean 3D EF deteriorated (54.7 ± 8.3% vs. 46.4 ± 8.6%, P = 0.017) before improving at discharge (P = 0.008). Left main coronary artery (LMCA) dimensions were significantly larger at discharge than at admission (Z score –0.11 ± 0.87 vs. 0.78 ± 1.23, P = 0.007). CT showed uniform coronary artery dilatation commonly affecting the LMCA (9/12). CMR detected abnormal strain in all patients with global dysfunction (EF <55%) in 35%, myocardial oedema in 50%, and subendocardial infarct in 5% (1/20) patients. Conclusions Pancarditis with cardiac dysfunction is common and associated with myocardial oedema. Patients require close monitoring due to coronary artery dilatation and the risk of thrombotic myocardial infarction.

Myocardial oedema detected by T2-mapping: a key marker of recent ischaemia after resuscitated sudden cardiac death

2019 ◽  
Vol 20 (11) ◽  
pp. 1319-1319
Author(s):  
Georgios Tzimas ◽  
David C Rotzinger ◽  
Olivier Muller ◽  
Pierre Monney
Keyword(s):  
Sudden Cardiac Death ◽  
Cardiac Death ◽  
T2 Mapping ◽  
Myocardial Oedema

R2 prime (R2′) magnetic resonance imaging for post-myocardial infarction intramyocardial haemorrhage quantification

2019 ◽  
Vol 21 (9) ◽  
pp. 1031-1038 ◽  
Author(s):  
Xavier Rossello ◽  
Pedro Lopez-Ayala ◽  
Rodrigo Fernández-Jiménez ◽  
Eduardo Oliver ◽  
Carlos Galán-Arriola ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Magnetic Resonance ◽  
T2 Mapping ◽  
Post Myocardial Infarction ◽  
Myocardial Oedema ◽  
Accurate Identification ◽  
Tissue Samples ◽  
Linear Regressions ◽  
Histological Validation

Abstract Aims To assess whether R2* is more accurate than T2* for the detection of intramyocardial haemorrhage (IMH) and to evaluate whether T2′ (or R2′) is less affected by oedema than T2* (R2*), and thus more suitable for the accurate identification of post-myocardial infarction (MI) IMH. Methods and results Reperfused anterior MI was performed in 20 pigs, which were sacrificed at 120 min, 24 h, 4 days, and 7 days. At each time point, cardiac magnetic resonance (CMR) T2- and T2*-mapping scans were recorded, and myocardial tissue samples were collected to quantify IMH and myocardial water content. After normalization by the number of red blood cells in remote tissue, histological IMH increased 5.2-fold, 10.7-fold, and 4.1-fold at Days 1, 4, and 7, respectively. The presence of IMH was correlated more strongly with R2* (r = 0.69; P = 0.013) than with T2* (r = −0.50; P = 0.085). The correlation with IMH was even stronger for R2′ (r = 0.72; P = 0.008). For myocardial oedema, the correlation was stronger for R2* (r = −0.63; P = 0.029) than for R2′ (r = −0.50; P = 0.100). Multivariate linear regressions confirmed that R2* values were significantly explained by both IMH and oedema, whereas R2′ values were mostly explained by histological IMH (P = 0.024) and were little influenced by myocardial oedema (P = 0.262). Conclusion Using CMR mapping with histological validation in a pig model of reperfused MI, R2′more accurately detected IMH and was less influenced by oedema than R2* (and T2*). Further studies are needed to elucidate whether R2′ is also better suited for the characterization of post-MI IMH in the clinical setting.

Sign in / Sign up

Export Citation Format

Share Document