scholarly journals Repetitive Negative Thinking in Social Anxiety Disorder 1: Anticipatory Processing

2017 ◽  
Vol a4 (3) ◽  
pp. 244-262 ◽  
Author(s):  
Rachel A. Sluis ◽  
Mark J. Boschen ◽  
David L. Neumann ◽  
Karen Murphy
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Experimental Studies ◽  
Poor Performance ◽  
Future Research ◽  
Repetitive Negative Thinking ◽  
Socially Anxious ◽  
Negative Thinking ◽  
Anticipatory Processing

Cognitive models of social anxiety disorder (SAD) emphasize anticipatory processing as a prominent maintaining factor that occurs before social-evaluative events. Anticipatory processing occurs when a socially anxious individual is expecting a social event and can be described as a mode of repetitive negative thinking dominated by past failures, negative images of oneself, predictions of poor performance and rejection. The present review examined the literature on anticipatory processing in social anxiety in an effort to highlight important findings pertaining to this construct. Correlational and experimental studies have investigated the relationship between anticipatory processing and the behavioural, physiological, cognitive and affective outcomes for socially anxious individuals. Studies investigating the characteristics, causes, and consequences of anticipatory processing according to models of social anxiety were included for review. The majority of study designs include those investigating anticipatory processing prior to social-evaluative threat. Directions for future research are discussed and an overview of a framework for explaining anticipatory processing biases in social anxiety is presented.

scholarly journals Repetitive Negative Thinking in Social Anxiety Disorder 2: Post-Event Processing

2016 ◽  
Vol a4 (3) ◽  
pp. 263-289 ◽  
Author(s):  
Rachel A. Sluis ◽  
Mark J. Boschen ◽  
David L. Neumann ◽  
Karen Murphy
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Experimental Studies ◽  
Response To Treatment ◽  
Empirical Literature ◽  
Future Research ◽  
Event Processing ◽  
Repetitive Negative Thinking ◽  
Negative Thinking

Cognitive models of social anxiety disorder (SAD) emphasize post-event processing as a prominent maintaining factor that occurs after social-evaluative events. Post-event processing involves repetitive negative thinking revolved around perceived social failure. The present review concentrates on the relevant and available empirical literature on post-event processing in social anxiety which centres on Clarke and Wells (1995) theoretical framework. Correlational and experimental studies have investigated the relationship between post-event processing and the behavioural, physiological, cognitive and affective outcomes for socially anxious individuals. The majority of study designs include those investigating post-event processing in response to social-evaluative threat, and in response to treatment. Limitations of the existing literature are discussed and suggestions for future research examining the underlying cognitive functions of post-event processing are proposed.

Social anxiety disorder in children: investigating the relative contribution of automatic thoughts, repetitive negative thinking and metacognitions

2020 ◽  
pp. 1-13
Author(s):  
Barbara Hoff Esbjørn ◽  
Anette Falch ◽  
Monika Anna Walczak ◽  
Nicoline Normann ◽  
Sonja Breinholst
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Anxiety Symptoms ◽  
Automatic Thoughts ◽  
Social Threat ◽  
Repetitive Negative Thinking ◽  
Negative Thinking ◽  
Relative Contribution ◽  
Negative Automatic Thoughts

Abstract Background: Social anxiety disorder (SAD) is common in youths. However, our understanding of SAD in children is inferior to that of SAD in adolescents or adults, and it is unclear if known adult SAD maintenance mechanisms may also operate in children with SAD. Aim: The paper sets out to investigate the specificity of positive automatic thoughts, social threat negative automatic thoughts, repetitive negative thinking, positive and negative metacognitions in predicting SAD symptoms and diagnoses in clinically anxious children. Method: We enrolled 122 clinically anxious children aged 7–13 years; of these, 33 had an SAD diagnosis. Results: SAD symptoms correlated positively with social threat negative automatic thoughts, repetitive negative thinking, and negative metacognitions, and negatively with positive automatic thoughts. Linear regression indicated that, of these variables, only social threat negative automatic thoughts predicted social anxiety symptoms. Logistic regression indicated that social threat negative automatic thoughts, a higher number of diagnoses and negative metacognitive beliefs specifically predicted the presence of SAD diagnosis. Conclusions: Our findings suggest that content-specific social threat negative automatic thoughts was the only variable that specifically distinguished both higher levels of social anxiety symptoms and diagnoses.

Measurement Invariance of Disorder-Specific and Transdiagnostic Measures of Repetitive Negative Thinking

Assessment ◽  
2021 ◽  
pp. 107319112110286
Author(s):  
Sarah Shihata ◽  
Andrew R. Johnson ◽  
David M. Erceg-Hurn ◽  
Peter M. McEvoy
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Depressive Disorder ◽  
Measurement Invariance ◽  
Generalized Anxiety Disorder ◽  
Social Anxiety Disorder ◽  
Generalized Anxiety ◽  
Response Scale ◽  
Repetitive Negative Thinking ◽  
Negative Thinking

Background: Repetitive negative thinking is conceptualized to be a transdiagnostic process linked to the development and maintenance of psychopathology. Prior research distinguishes between disorder-specific exemplars (worry, rumination) and transdiagnostic measures of repetitive negative thinking with differences across disorders reported. However, establishing the measurement invariance of these measures is necessary to support meaningful comparisons across clinical groups. Method: Bayesian structural equation modelling was used to assess the approximate invariance of the Ruminative Response Scale, Penn State Worry Questionnaire, and the Repetitive Thinking Questionnaire across individuals with a principal diagnosis of either depressive disorder, social anxiety disorder, or generalized anxiety disorder. Results: All scales demonstrated approximate measurement invariance across the three disorder groups. The depressive disorder group reported a higher level of rumination than the generalized anxiety disorder group (Δµ = 0.25, 95% Credibility Interval [0.06, 0.45]), with no difference between the generalized anxiety disorder and social anxiety disorder groups. The depressive disorder and generalized anxiety disorder groups did not differ in their levels of trait repetitive negative thinking, but the social anxiety disorder group was markedly lower than the generalized anxiety disorder group (Δµ = −0.21 [−0.37, −0.05]). Similarly, levels of worry did not differ between the generalized anxiety disorder and depressive disorder group but were lower in the social anxiety disorder group than the generalized anxiety disorder group (Δµ = −0.23 [−0.41, −0.06]). Conclusions: The Ruminative Response Scale, Penn State Worry Questionnaire, and Repetitive Thinking Questionnaire are measuring trait repetitive negative thinking in a consistent manner across individuals with a principal diagnosis of depressive disorder, social anxiety disorder, or generalized anxiety disorder. This supports their use in transdiagnostic contexts and indicates that it is appropriate to directly compare the scores on these measures between diagnostic groups.

scholarly journals Coping with negative mental images in social anxiety disorder: Investigating the potential benefits of image morphing

2019 ◽  
Vol 10 (1) ◽  
pp. 204380871881375 ◽  
Author(s):  
Vanja Vidovic ◽  
Mia Romano ◽  
David A. Moscovitch
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Positive Affect ◽  
Social Anxiety Disorder ◽  
Social Stress ◽  
Daily Practice ◽  
Future Research ◽  
Image Morphing ◽  
Healthy Control ◽  
Potential Benefits

Negative mental imagery contributes to symptom maintenance in social anxiety disorder (SAD). Here, we investigated the effects of image morphing, a brief mental strategy designed to facilitate access to positive images. Participants with SAD and healthy control (HC) participants were randomly assigned to receive either image morphing or supportive counseling. Although initial training and 1-week daily practice were successful in equipping morphing participants across groups with the required skill, those assigned to morphing failed to demonstrate differential improvements in positive affect, negative affect, or self-perception relative to control participants during a subsequent social stress task. Ancillary analyses revealed that the number of positive details contained in retrieved or morphed images prior to the task significantly predicted the level of positive affect reported after the task, but this effect was observed only for HC participants. We discuss the need for future research to refine innovative imagery-based psychotherapeutic strategies for social anxiety.

Integrating Neurobiology and Psychopathology into Evidence-Based Treatment of Social Anxiety Disorder

CNS Spectrums ◽  
2005 ◽  
Vol 10 (S13) ◽  
pp. 1-2 ◽  
Author(s):  
Michael R. Liebowitz ◽  
Philip T. Ninan ◽  
Franklin R. Schneier ◽  
Carlos Blanco ◽  
David L. Ginsberg ◽  
...  
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Selective Serotonin Reuptake Inhibitors ◽  
Treatment Strategies ◽  
Psychosocial Treatment ◽  
Development Theory ◽  
Future Research ◽  
Life Issues ◽  
Neuroreceptor Imaging

AbstractSocial anxiety disorder (SAD) is a common, chronic psychiatric disorder characterized by a persistent fear of social or performance situations in which embarrassment can occur. This disorder typically appears during the mid-adolescent years and is unremitting throughout life if not properly treated. SAD presents as two subtypes: the more common and debilitating generalized form, and the nongeneralized form, which consists predominantly of performance anxiety. The majority of patients with SAD have comorbid mental disorders, including mood, anxiety, and substance abuse. No single development theory has been proposed to account for the origins of SAD, although current understanding of the etiology of SAD posits an interaction between psychological and biological factors. Risk factors include environmental and parenting influences and dysfunctional cognitive and conditioning events in early childhood. The neurobiology of SAD appears to involve neurochemical dysfunction, as evidenced by studies of neuroreceptor imaging, neuroendocrine function, and profiles of response to specific medications. Clinical trials have demonstrated that benzodiazepines and antidepressants are effective in the treatment of SAD. The selective serotonin reuptake inhibitors are emerging as the first-line treatment for SAD, based on their proven safety, tolerability, and efficacy. Goals for ongoing future research include development of approaches to achieve remission, to convert nonresponders and partial responders to full responders, and to prevent relapse and maintain long-term efficacy.This monograph explores the epidemiology, clinical presentation, and differential diagnosis of SAD, with a focus on neural circuitry of social relationships and neurochemical dysfunction. The prevalence, rates of recognition and treatment, patterns of comorbidity, quality-of-life issues, and natural history of SAD are discussed as well as pharmacologic and psychosocial treatment strategies for SAD.

scholarly journals Neuroendocrine models of social anxiety disorder

2015 ◽  
Vol 17 (3) ◽  
pp. 287-293 ◽  
Keyword(s):  
Anxiety Disorder ◽  
Social Anxiety ◽  
Social Anxiety Disorder ◽  
Developmental Plasticity ◽  
Peptide Hormones ◽  
Social Avoidance ◽  
Socially Anxious ◽  
Submissive Behavior ◽  
Early Trauma ◽  
Developmental Windows

Social anxiety disorder (SAD) is a highly prevalent and disabling disorder with key behavioral traits of social fearfulness, social avoidance, and submissiveness. Here we argue that hormonal systems play a key role in mediating social anxiety, and so may be important in SAD. Hormonal alterations, often established early in development through the interaction between biological and psychological factors (eg, genetic predisposition x early trauma), predispose to socially fearful, avoidant, and submissive behavior. However, whereas gene variants and histories of trauma persist, hormonal systems can be remodeled over the course of life. Hormones play a key role during the periods of all sensitive developmental windows (ie, prenatal, neonatal, puberty, aging), and are capable of opening up new developmental windows in adulthood. Indeed, the developmental plasticity of our social brain, and thus of social behavior in adulthood, critically depends on steroid hormones such as testosterone and peptide hormones such as oxytocin. These steroid and peptide hormones in interaction with social experiences may have potential for reprogramming the socially anxious brain. Certainly, single administrations of oxytocin and testosterone in humans reduce socially fearful, avoidant, and submissive behavior. Such work may ultimately lead to new approaches to the treatment of SAD.

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