scholarly journals The effect of soft tissue mobilization on pain, disability level and depressive symptoms in patients with chronic low back pain

2020 ◽  
Vol 11 (4) ◽  
Keyword(s):  
Low Back Pain ◽  
Back Pain ◽  
Depressive Symptoms ◽  
Soft Tissue ◽  
Chronic Low Back Pain ◽  
Low Back ◽  
Pain Disability ◽  
Soft Tissue Mobilization ◽  
Disability Level

Pain, Disability, and Psychological Functioning in Chronic Low Back Pain Subgroups

Neurosurgery ◽  
1993 ◽  
Vol 33 (3) ◽  
pp. 379-386 ◽  
Author(s):  
Jeffrey E. Cassisi ◽  
George W. Sypert ◽  
Luciana Laganá ◽  
Elizabeth M. Friedman ◽  
Michael E. Robinson
Keyword(s):  
Low Back Pain ◽  
Back Pain ◽  
Chronic Low Back Pain ◽  
Psychological Functioning ◽  
Low Back ◽  
Pain Disability

scholarly journals Prevalence and Associated Factors of Depressive Symptoms Among Patients With Chronic Low Back Pain: A Cross-Sectional Study

2022 ◽  
Vol 12 ◽  
Author(s):  
Yueming Hu ◽  
Zechuan Yang ◽  
Yong Li ◽  
Yong Xu ◽  
Mengge Tian ◽  
...  
Keyword(s):  
Low Back Pain ◽  
Back Pain ◽  
Depressive Symptoms ◽  
Chronic Low Back Pain ◽  
Associated Factors ◽  
Cross Sectional Study ◽  
Low Back ◽  
Sectional Study ◽  
Cross Sectional ◽  
Family Function

Background: Few studies have focused on depressive symptoms among patients with chronic low back pain in China. The aim of this cross-sectional study was to assess the prevalence and associated factors of depressive symptoms in patients with chronic low back pain.Methods: From May to August 2021, 1,172 patients with chronic low back pain were recruited in China. Depressive symptoms were assessed through the Patient Health Questionnaire. Associations of demographic characteristics, clinical characters and social-psychological factors with depressive symptoms were investigated among patients with chronic low back pain.Results: The prevalence of depressive symptoms was 25.00%. Logistic regression analysis found that duration of pain in 1-5 years (1-3 years: OR = 2.91, 95%CI: 1.65-5.14, 3-5 years: OR = 3.09, 95%CI: 1.55-6.15) and more severe pain (OR = 1.13, 95%CI: 1.10-1.17) were associated with higher risks of depressive symptoms. Better family function (good family function: OR = 0.25, 95%CI: 0.15-0.41, moderate family dysfunction: OR = 0.47, 95%CI: 0.29-0.77) and higher pain self-efficacy (OR = 0.94, 95%CI: 0.93-0.95) were associated with lower risks of depressive symptoms.Conclusion: Patients with chronic low back pain have a high prevalence of depressive symptoms in China. Duration of pain, pain severity, family function and pain self-efficacy were predictors of depressive symptoms among chronic low back pain patients in China. Early identification of the associated factors may be helpful for the timely management of depressive symptoms.

Circulating Adipokines in Predicting the Transition from Acute to Persistent Low Back Pain

Pain Medicine ◽  
2020 ◽  
Vol 21 (11) ◽  
pp. 2975-2985 ◽  
Author(s):  
David M Klyne ◽  
Paul W Hodges
Keyword(s):  
Low Back Pain ◽  
Back Pain ◽  
Depressive Symptoms ◽  
Repeated Measurements ◽  
Low Back ◽  
Reactive Protein ◽  
Acute Low Back Pain ◽  
Pain Disability ◽  
Inflammation Cytokines ◽  
Necrosis Factor

Abstract Objectives Cytokines such as tumor necrosis factor (TNF) contribute to the transition from acute to persistent pain. Despite increasing incidence of obesity and its linkage with chronic pain and inflammation, cytokines predominantly produced by adipose tissue (adipokines) have received little attention. Here we aimed to explore the longitudinal trajectory of adipokines from the onset of acute low back pain (LBP) and identify combinations of adipokines and/or other features that predict outcome. Methods Individuals with acute LBP (less than two weeks after onset) who had either recovered (no pain, N = 15) or remained unrecovered (no reduction/increase in pain, N = 13) at six months and 15 controls were retrospectively selected from a larger prospective cohort. Participants provided blood for the measurement of TNF, interleukin-6 (IL-6), resistin, visfatin, adiponectin, leptin, and C-reactive protein (CRP), and completed questionnaires related to pain/disability, depression, and sleep at baseline. LBP participants repeated measurements at six months. Results Compared with controls, acute LBP individuals had higher TNF and CRP but lower adiponectin. In LBP, unrecovered individuals had higher TNF at both time points, but lower CRP at baseline and leptin at six months. Although combined low CRP, high TNF, and depressive symptoms at baseline predicted poor recovery, the primary adipokines leptin, resistin, visfatin, and adiponectin did not. Conclusions Primary adipokines did not add to the prediction of poor LBP outcome that has been identified for the combination of low CRP, high TNF, and depressive symptoms in acute LBP. Whether adipokines play a role in LBP persistence in overweight/obese individuals requires investigation.

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