scholarly journals Mitochondrial DNA transfer through Exomes during Immune Synapsis primes Antiviral Innate Immune Response in Dendritic Cells

2019 ◽  
Author(s):  
Daniel Torralba
Keyword(s):  
Immune Response ◽  
Dendritic Cells ◽  
Mitochondrial Dna ◽  
Innate Immune Response ◽  
Innate Immune ◽  
Dna Transfer

scholarly journals Salmonella escapes adaptive immune response via SIRT2 mediated modulation of innate immune response in dendritic cells

2018 ◽  
Vol 14 (11) ◽  
pp. e1007437 ◽  
Author(s):  
Mayuri Gogoi ◽  
Kasturi Chandra ◽  
Mohsen Sarikhani ◽  
Ramya Ramani ◽  
Nagalingam Ravi Sundaresan ◽  
...  
Keyword(s):  
Immune Response ◽  
Dendritic Cells ◽  
Innate Immune Response ◽  
Adaptive Immune Response ◽  
Innate Immune ◽  
Adaptive Immune

scholarly journals ER stress and its regulator X-box-binding protein-1 enhance polyIC-induced innate immune response in dendritic cells

2011 ◽  
Vol 41 (4) ◽  
pp. 1086-1097 ◽  
Author(s):  
Fanlei Hu ◽  
Xiaofei Yu ◽  
Hongxia Wang ◽  
Daming Zuo ◽  
Chunqing Guo ◽  
...  
Keyword(s):  
Immune Response ◽  
Dendritic Cells ◽  
Er Stress ◽  
Innate Immune Response ◽  
Binding Protein ◽  
Innate Immune

scholarly journals Correction: Salmonella escapes adaptive immune response via SIRT2 mediated modulation of innate immune response in dendritic cells

2020 ◽  
Vol 16 (2) ◽  
pp. e1008345
Author(s):  
Mayuri Gogoi ◽  
Kasturi Chandra ◽  
Mohsen Sarikhani ◽  
Ramya Ramani ◽  
Nagalingam Ravi Sundaresan ◽  
...  
Keyword(s):  
Immune Response ◽  
Dendritic Cells ◽  
Innate Immune Response ◽  
Adaptive Immune Response ◽  
Innate Immune ◽  
Adaptive Immune

The Regulatory Role of Dendritic Cells in the Innate Immune Response

2014 ◽  
pp. 95-109 ◽  
Author(s):  
F. Granucci ◽  
S. Feau ◽  
I. Zanoni ◽  
G. Raimondi ◽  
N. Pavelka ◽  
...  
Keyword(s):  
Immune Response ◽  
Dendritic Cells ◽  
Innate Immune Response ◽  
Innate Immune ◽  
Regulatory Role

scholarly journals Innate Immune Response in Kidney Ischemia/Reperfusion Injury: Potential Target for Therapy

2017 ◽  
Vol 2017 ◽  
pp. 1-10 ◽  
Author(s):  
Aleksandra Kezić ◽  
Natasa Stajic ◽  
Friedrich Thaiss
Keyword(s):  
Immune Response ◽  
Dendritic Cells ◽  
Reperfusion Injury ◽  
Innate Immune Response ◽  
Ischemia Reperfusion Injury ◽  
Ischemia Reperfusion ◽  
Graft Function ◽  
Innate Immune ◽  
Injury Potential ◽  
Kidney Ischemia

Acute kidney injury caused by ischemia and subsequent reperfusion is associated with a high rate of mortality and morbidity. Ischemia/reperfusion injury in kidney transplantation causes delayed graft function and is associated with more frequent episodes of acute rejection and progression to chronic allograft nephropathy. Alloantigen-independent inflammation is an important process, participating in pathogenesis of injurious response, caused by ischemia and reperfusion. This innate immune response is characterized by the activity of classical cells belonging to the immune system, such as neutrophils, macrophages, dendritic cells, lymphocytes, and also tubular epithelial cells and endothelial cells. These immune cells not only participate in inflammation after ischemia exerting detrimental influence but also play a protective role in the healing response from ischemia/reperfusion injury. Delineating of complex mechanisms of their actions could be fruitful in future prevention and treatment of ischemia/reperfusion injury. Among numerous so far conducted experiments, observed immunomodulatory role of adenosine and adenosine receptor agonists in complex interactions of dendritic cells, natural killer T cells, and T regulatory cells is emphasized as promising in the treatment of kidney ischemia/reperfusion injury. Potential pharmacological approaches which decrease NF-κB activity and antagonize mechanisms downstream of activated Toll-like receptors are discussed.

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